adolescent dev - growth: puberty Flashcards

1
Q

aspects of adolescent that makes it a critical period

A
  • changes in body composition
  • changes in diet, Pa
  • sedentary behaviours
  • sleep
  • sports and specialized motor dev
  • emerging identity and role
  • mental health
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2
Q

what is proven to affect diet in adulthood

A

having family meals

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3
Q

what happens physiologically with <9 hours

A

decrease leptin levels
increased gherkin levels
increase BMI, increase appetite, increase food intake

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4
Q

Stages in specialized movement abilities

A
  • Transition (7-9): mvt exploration
  • Application (11-13): children choose what to pursue
  • Lifelong utilization (14+): refinement
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5
Q

Factors affecting puberty

A
  • genetic
  • biological
  • stress
  • SES
  • Nutrition/diet
  • % body fat
  • chronic illness
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6
Q

Puberty onset

A

19th century: 16-17
20th/21st: 11-13 y/o

GirlsL age of menarche (1st menstrual cycle)
Boys: environ 2 years after girls
- obesity associated with later puberty

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7
Q

3 phases of puberty GIRLS

A

stage 1: pre-puberty, no sign
Stage 2: in puberty, breast enlargement, pubic or axila hair
STAGE 3: completing: period with signs of pubertal dev

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8
Q

3 phases of puberty BOYS

A

STAGE1: high voice
STAGE2: deepening of voice, early pubic or axillary hair, enlargement of testes or penis
STAGE3: voice fully broken, facial hair, adult size penis w/ pubic hair and axillary hair

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9
Q

Tanner stages puberty girls
Breast

A

1: elevation of papilla
2: elevation breast/papilla, areola diameter enlarged, age:9.8
3: further enlargement w/ separation breast/areola, age:11.2
4: secondary mound of areola and papilla above breast, age: 12.1
5: recession of areola to contour of breast, age 14.6

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10
Q

Tanner stages puberty girls
pubic hair

A

1: no hair
2: sparse, long, pigmented hair along labia majora, age 10.5
3: dark, course, curled spread overmons, age 11.4
4: adult hair type, abundant limited to mons, age 12
5: adult type spread in quantity and distribution, age 13.7

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11
Q

who is in charge of the hormonal control of puberty

A
  • hypothalamic
  • pituitary
  • gonadal axis
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12
Q

what does the hypothalamus releases

A

gonadotropin releasing hormone
- follicle stimulating hormone
- luteinizing hormone

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13
Q

What is the role of FSH/LH

A

activate the gonads
- rel estrogen/progesterone and testosterone

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14
Q

Hormonal changes in puberty

A

Increase: GH, liver lipolysis, FFA
Decreased: insulin sensitivity
Females: estrogen increases fat deposition (peripherally)
Males: testosterone increases FFM

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15
Q

delayed onset of puberty

A

the absence of secondary sexual characteristics by age 13 in girls or 16 in boys

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16
Q

causes of delayed onset of puberty

A
  • constitutional growth delay (inherited, temporary, delay in growth)
  • Underlying medical condition
    (diabetes, cystic fibrosis, kidney disease)
  • Not considered a medical concern
  • severely delayed/absent puberty can be treated with HRT
17
Q

Precocious puberty

A

appearance of secondary sexual characteristics before the age of 8 in girls 9 in boys

18
Q

types of precocious puberty

A
  • Central precocious puberty
    (normal pattern but too soon, could be tumor, hypothyroidism)
  • Peripheral precocious puberty
    (less common, congenital-> idiopathic, issue with testicls, ovaries, adrenal glands)
  • Isosexual
    (consistent w/ genetic and gonadal sex of the child)
  • Contra-sexual
    ( feminization or virilisation)
19
Q

Precocious risk factors

A
  • genetics: family hx of PP
  • females > males
  • racial background: more African American
  • obesity
  • exposure to sex hormones
  • medical condition: abnormal production of androgens
  • radiation therapy to CNS
  • exposure to blue light
20
Q

Precocious puberty complications

A
  • Short height
    (tall vs pears, but bones mature sooner resulting in shorter stature)
  • Social and emotional issues
    ( social self-consciousness about changes occurring in their bodies)