[acute respiratory distress syndrome] Flashcards
release of inflammatory mediators
widespread inflammation on the lung
non-cardiogenic
changes in permeability to the pulmonary capillary membrane
pneumonia gastric aspiration inhalation trauma vasculitis
bruise (laymans)
sepsis
bilateral fine inspiratory crackles
CXR
bilateral inflammatory infiltrates
low PaO2
an indirect measure of left atrial pressure
gold standard for determining causes of pulmonary oedema (oedema + normal wedge pressure = ARDS/non-cardiogenic)
Acute onset
CXR +ve
PCWP
Refractory hypoxaemia
less than 19mmHg
or lack of clinical CHF signs
fraction of inspired oxygen
Acute onset
CXR +ve
PCWP
Refractory hypoxaemia (PaO2:FiO2)
fraction of inspired oxygen
comparison between [O2] in blood and the [O2] that is breathed
less than 200
between 200-300 indicates acute lung injury
low tidal volume mechanical ventilation
reduced lung compliance = high pressure –> potential pneumothorax
trigger (shock/trauma)
widespread inflammation/release of inflammatories
damaged alveoli/reduced surfacant
BILATERAL (widespread inflammation)
basically anything
Sepsis –> malaria –|> raised ICP