Acidosis Flashcards

1
Q

rumen temp

A

38-41 with decrease between meals

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2
Q

normal rumen pH range

A

5.5-6.9

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3
Q

rumen microbiota relative populations

A

most bacteria, then bacteriophages, then protozoa, then methanogens, then fungi,

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4
Q

3 types of microbe communities

A

particle associated: up to 75% high enzymatic activity
liquid associated: up to 50%, utilize soluble nutrients
epimural associated: more stable, urease activity

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5
Q

SCFA

A

major energy supply
production released ATP for microbial growth
difficult to measure production

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6
Q

major VFA, what changes between forage and concentrate diets

A

always mostly acetate
when on concentrate propionate increases

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7
Q

SCFA and lactic acid pKA
what does this mean

A

SCFA = 4.9
lactic acid = 3.9
at lower pH SCFA doesn’t grow as well and lactic grows better

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8
Q

predominant lactate isomer in the rumen

A

L lactate

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9
Q

absorption difference between L and D lactate

A

L lactate absorbs better. D is more predominant at lower pH and sticks around longer snowballing acidity

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10
Q

SARA pH

A

5.2-5.6 for atleast 3h in a day

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11
Q

acute acidosis pH

A

below 5.0 (5.2)

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12
Q

bacteria linked to liver abscess increase when pH

A

decrease

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13
Q

lactic acid utilizers trend for SARA and acute

A

lactic acid utilizers are increased in SARA and decreased in acute
cant deal with acute issues

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14
Q

main and other acid removal methods from rumen

A

main is absorption
others are bicarb and passage

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15
Q

relation between salvia and acidosis

A

more saliva does not prevent acidosis as more saliva comes from more intake which means more ferm to deal with

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16
Q

how does HCO3- get into rumen

A

antiport with VFA between rumen and blood stream

17
Q

is bicarb higher in roughage or concentrate diets for salivary bicarb and rumen bicarb

A

salivary bicarb is higher in roughage
rumen bicarb is higher when on concentrate

18
Q

main mechanisms for the removal of acid from ruminal contents (5)

A

saliva
passive diffusion of SCFA across rumen wall
SCFA absorption antiport with bicarb
carbonic anhydrase
passage of acid out of rumen

19
Q

why consider ruminal acidosis

A

animal welfare
decreased performance
associated disorders
mortality

20
Q

causes of acidosis

A

incr VFA production
excess grain
increase in dietary carb
insufficient fiber
insufficient buffering
sorting feed

21
Q

SARA consequences

A

metabolic acidosis
death
poor performance

22
Q

chronic SARA acidosis consequences

A

poor bunk management
cyclic intake pattern
reduced performance

23
Q

types of bloat

A

free grass
frothy (can be feedlot due to microbial factors or pasture due to plant components)

24
Q

consequences of acidosis

A

decr feed intake
decr absorptive capacity
incr liver abscesses

25
how do liver abscesses happen
low pH deteriorates rumen wall, bacteria invade systemic circulation, hepatic blood stream takes to liver where it becomes encapsulated 1-2 small abscesses is severe
26
laminitis
low pH degrades rumen wall allowing bacteria to enter systemic circulation and translocate causing infection vascular seepage causes edema, hypoxia, degradation of corium and irreversible damage
27
cost to inflammation
induces glucose utilization that should be used for growth instead increases liver amino acid uptake decreased growth and feed efficiency
28
where does acidosis have a significantly different pH than normal
Rumen Cecum Proximal colon distal colon
29
mucin cast
bandaid on LI to try and recover from pH damage comes out in feces
30
what areas should you target when trying to mitigate ruminal acidosis
reduce fermentability feeding management
31
how to reduce fermentability
increase fiber, byproducts, and fat
32
what to change in feeding management to reduce acidosis
frequency bunk management avoiding restriction improve dietary adaptation
33
cascade of grain processing speed
wheat barley corn sorghum
34
factors for variation in daily feed intake
weather missed feedings poor processing stale feed increasing too quickly
35
when would you feed the higher grain diet if you are having acidosis issues
in the afternoon feeding