5.1.5 Plant and animal responses Flashcards

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1
Q

CNS made up of and role

A

central nervous system = brain + spinal cord

coordinating response

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2
Q

brain mostly made out of

A

non-myelinated relay neurones (grey matter)

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3
Q

spinal cord mostly made out of

A

myelinated (white) and non-myelinated (grey) relay neurones

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4
Q

PNS made out of and role

A

sensory and motor neurones

connects receptors to CNS and to effector to bring about response

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5
Q

sensory nervous system structure

A

connects receptor to CNS
sensory neurones enter spinal cord at dorsal root (where cell body is also)
short axon connects to relay neurones in CNS

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6
Q

motor neurones structure and role

A

connects CNS and effectors

split into autonomic and somatic nervous systems

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7
Q

somatic nervous system features and role

A

motor neurones under voluntary control
e.g. controlling skeletal muscles
mostly myelinated neurones (fast)
single motor neurones connect CNS and effectors

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8
Q

autonomic nervous system features and role

A

motor neurones under involuntary control
mostly non-myelinated neurones (slower)
at least 2 neurones between CNS and effector

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9
Q

examples of actions controlled by autonomic nervous system

A
controlling glands
cardiac muscle
smooth muscles in gut
eyes
blood vessels
airways
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10
Q

ganglia obvious features

A

swelling

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11
Q

sympathetic vs parasympathetic nervous systems in general

A

sympathetic more active in times of stress whereas parasympathetic in times of rest

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12
Q

how autonomic nervous system is split

A

sympathetic and parasympathetic
antagonistic to each other
balance depending on internal conditions and stress to bring about appropriate response

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13
Q

sympathetic nervous system features

A
short preganglionic neurone 
ganglia near CNS
many nerves leave CNS
noradrenaline is neurotransmitter
active in fight/flight or stress
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14
Q

parasympathetic nervous system features

A
long preganglionic neurone
ganglia near organs
few nerves leave CNS then split up to go to effectors
acetylcholine is neurotransmitter
active in calm
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15
Q

human brain 4 main parts

A

cerebrum
cerebellum
hypothalamus + pituitary complex
medulla oblongata

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16
Q

cerebrum function

A

organises most higher thought process e.g.
conscious thought/actions
memory
emotions
intelligence, reasoning, judgement, decision making

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17
Q

cerebellum function

A

coordinates balance and fine movement e.g. tensioning muscles for playing music, judging positioning of objects while moving
complex nervous pathways become stronger with practice (becomes “second nature”)

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18
Q

hypothalamus structure and role

A

organises homeostatic responses and control physiological processes
e.g. temperature regulation and osmoregulation
contains own receptors, osmoreceptors, thermoreceptors
regulates feeding and sleeping patterns

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19
Q

medulla oblongata function

A
coordinates many autonomic responses 
controls cardiac muscles and smooth muscles by sending action potentials 
through autonomic nervous system
regulates many vital processes e.g.
cardiac centre (regulates heart rate)
vasomotor centre (regulates circulation + blood pressure)
respiratory centre (controls rate + depth of breathing)
centres receive sensory information and coordinate vital functions through negative feedback
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20
Q

cerebrum structure

A

2 cerebral hemispheres connected via major tracts of neurones called corpus callosum
outermost layer consists of thin layer of nerve cell bodies called cerebral cortex

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21
Q

cerebral cortex structure

A

sensory areas
association areas
motor areas

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22
Q

how cerebrum and cerebellum connected

A

the pons

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23
Q

pituitary gland structure and role

A

posterior lobe linked to hypothalamus by specialised neurosecretory glands
secretes hormones (produced in hypothalamus) into blood
anterior lobe produces own hormones (for physiological processes e.g. stress), released in response to releasing factors produced by hypothalamus

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24
Q

sensory area function

A

receive action potentials from sensory receptors, size related to sensitivity of area to inputs received

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25
Q

association area function

A

compares and interprets sensory inputs with previous experiences to judge appropriate response

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26
Q

motor area function

A

send action potentials to effectors, size related to complexity of movements needed in parts of body, left side of brain controls effectors on right side and vice versa

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27
Q

knee jerk reflex definition

A

reflex action that straightens leg when tendon below kneecap is tapped

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28
Q

reflex action definition

A

response that doesn’t involve any processing by the brain

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29
Q

why reflex occur

A

need to be quick for survival

e.g. get out of danger, prevent damage, maintain balance

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30
Q

nervous pathway of reflex actions

A

sensory neurone -> relay neurone -> motor neurone

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31
Q

cranial reflex definition

A

reflex where nervous pathway passes through part of the brain but doesn’t involve any thought processes

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32
Q

reflex arc definition

A

receptor and effector are in the same place

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33
Q

blinking stimulus examples

A

foreign object touching eye (corneal reflex)
sudden bright light (optical reflex)
loud sounds
sudden movements close to eye

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34
Q

optical reflex

A

protects light-sensitive cells of retina from damage
stimulus detected by retina
reflex mediated by optical centre in cerebral cortex
slower than corneal reflex

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35
Q

corneal reflex

A

mediated by sensory neurone from cornea, entering pons
synapse connects sensory to relay neurone, carrying action potential to motor neurone
motor neurone passes back out of brain to facial muscles, causing eyelids to blink
short and direct pathway so very rapid

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36
Q

why corneal reflex can be overridden

A

sensory neurone involved in corneal reflex also passes action potentials to myelinated neurones in pons
these neurones carry a.p. to sensory area in cerebral cortex
informs higher centres of brain that stimulus has occurred
allows reflex to be overriden by conscious control
myelinated neurones carry a.p. faster than non-myelinated neurones

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37
Q

how knee jerk reflex works

A

muscle at bottom of thigh contracts to straighten leg
muscle spindle (specialised stretch receptors) detect increase in length of muscle
if unexpected, reflex causes contraction of same muscle to remain balanced

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38
Q

why knee jerk reflex is strange

A

nervous pathway only involves 2 neurones
sensory neurone -> motor neurone
much quicker as 1 less synapse

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39
Q

spinal reflex definition

A

nervous pathway passes through spinal cord rather than through brain

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40
Q

why brain cannot inhibit knee jerk reflex

A

no relay neurone to carry a.p. to brain
sensory neurone stimulates motor neurone directly
insufficient delay to enable inhibition by brain sending inhibitory action potentials to synapse before motor neurone is stimulated

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41
Q

fight or flight response physiological changes

A

+heart rate and blood pressure (increased blood flow, more O2 and glucose to respiring cells for more respiration)
+breathing rate and depth (faster rate of gas exchange = more O2 in blood = more respiration)
arterioles to skin+digestive system vasoconstrict (less blood to skin and DS, not needed in response)
arterioles to muscles vasodilate (more blood for more respiration, needed in response)
pupils dilate (more light into retina to see better)
+glycogenolysis (more glucose released into blood from liver, more respiration)
+metabolic rate
erector muscles in skin contract (make hairs stand up to look bigger, potentially more intimidating, prevent conflict)

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42
Q

role of brain in fight or flight response

A

receptor sense threatening stimulus
a.p. sent to sensory centres in cerebrum
then association centres to coordinate response
cerebrum stimulated hypothalamus in response to threat
hypothalamus stimulates sympathetic nervous system + anterior pituitary gland

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43
Q

role of sympathetic nervous system in fight or flight response

A

increases activity of effectors via nervous impulses (more rapid response)
stimulates adrenal medulla to release adrenaline (which brings about responses in effectors) for longer response

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44
Q

action of adrenaline

A

adrenaline acts as first messenger (travels through blood to target cells)
binds to receptors on cell surface membrane of target cells
binding causes a G-protein on membrane to activate adenyl cyclase (enzyme)
this converts ATP into cAMP (cyclic AMP)
brings about effect in cell

45
Q

role of anterior pituitary gland in hypothalamic-pituitary-adrenal cortical axis

A

hypothalamus secretes corticotropin-releasing hormone (CRH)
causes release of adrenocorticotropic hormone (ACTH) into blood
stimulates adrenal cortex to release corticosteroids e.g. cortisol

46
Q

role of anterior pituitary gland in hypothalamic-pituitary-thyroid axis

A

thyrotropin‐releasing hormone (TRH) causes the release of thyroid‐ stimulating hormone (TSH) into blood stimulates the thyroid gland to release more thyroxine

47
Q

role of anterior pituitary gland in hypothalamic-pituitary axis in general

A

hypothalamus secretes releasing hormones into blood to pituitary gland
stimulates release of tropic hormones

48
Q

cortisol effect

A

increases metabolism of carbohydrates -> glucose
increases blood glucose levels
increases blood pressure and suppresses immune system

49
Q

thyroxine effect

A

increases metabolic rate

makes cells more sensitive to adrenaline

50
Q

how heart rate is controlled

A

cardiovascular centre in medulla oblongata sends nervous impulses to SAN via autonomic nervous system to alter the frequency of waves of excitation (changes heart rate)

51
Q

how SAN alters heart rate

A

heart beat always same length of time

heart rate increased by higher frequency and larger stroke volume

52
Q

how heart rate increased during exercise method

A

produce more CO2
more carbonic acid formed when reacting with water
more H+ which reduces pH
lower pH detected by chemoreceptors in carotid arteries, aorta and brain
increased action potential frequency in sensory neurone to medulla oblongata
cardiovascular centre sends nervous impulse to SAN via sympathetic nervous system
noradrenaline released at SAN
causes heart rate to increase

53
Q

other ways heart rate is increased examples

A

hormones from adrenal medulla bind to adrenoreceptors on cardiac muscle
stretch receptors detect movement in muscles, sends impulses to cardiovascular centre
heart rate increases

54
Q

why heart rate is increased

A

faster exchange of oxygen and glucose

faster removal of CO2 and other waste

55
Q

decreasing heart rate when stopping exercise method

A

conc. of CO2 decreases (pH rises)
higher pH detected by chemoreceptors in carotid arteries, aorta and brain
decreased ap. frequency in sensory neurone to medulla oblongata
cardiovascular centre sends fewer nervous impulses to SAN via parasympathetic nerve
heart rate decreases

56
Q

decreasing heart rate when increase in blood pressure method

A

monitored by baroreceptors in carotid sinus
if b.p. too high, sensory nerve carries signal to medulla oblongata
cardiovascular centre sends nervous impulses to SAN via vagus nerve (parasympathetic)
acetylcholine (neurotransmitter) released at SAN
cause heart rate to decrease
blood pressure to decrease

57
Q

muscle cell grouping

A

group together to form fibres which contract and relax

groups arranged in antagonistic pairs (one contracts as one elongates)

58
Q

skeletal muscle function and location

A

attached to bones

contract to move bones

59
Q

cardiac muscle location and function

A

found in heart

contract to make the heart beat to pump blood

60
Q

smooth (involuntary) muscle location and function

A

walls of bronchi/bronchioles, blood vessels and organs (e.g. small intestine, stomach)
control diameters of arteries/arterioles, bronchi/bronchioles
peristalsis (and passing of other substances)

61
Q

cardiac muscle structure

A

cells branch to ensure electrical stimulation spreads evenly over the walls so contraction is 3D
cells joined by intercalated discs to ensure synchronised contraction
muscles striated in appearance
do not easily fatigue

62
Q

smooth muscle structure

A

controlled by autonomic NS
contract slowly
non-striated
longitudinal cells arranged in circular shapes around lumen

63
Q

skeletal muscle structure

A

controlled by somatic NS
striated, cylindrical shaped cells

muscle cells join up to make long muscle fibres that share sarcoplasm (cytoplasm, contains lots of mitochondria) and sarcolemma (membrane)
between myofibrils are mitochondria, sarcoplasmic reticulum (Ca^2+ store), glycogen granules
short striated section of myofibrils called sarcomeres made out of actin+myosin filaments

64
Q

structure of sarcomeres

A

thin filaments of light bands (I bands) of striations
thin + thick filaments overlapping make up dark bands (A bands)
area in the middle of dark band that has no over lap (H zone) with dark M line in midle

65
Q

sliding filament hypothesis and contracted sarcomere under electron micrograph

A

shorter I band, shorter H band, Z discs get closer together, sarcomere shorter
A band remains the same

66
Q

stimulation of contraction method

A

a.p. arriving at end of axon open calcium ion channels
allows Ca^2+ ions to flow into axon tip
vesicles of acetylcholine move towards and fuses with the cell surface membrane
acetylcholine diffuses across gap and bind to receptors on muscle fibre
sodium ion channels open and Na^+ ions enter muscle fibre, causes depolarisation
wave of depolarisation creates a.p. that passes along sarcolemma and down transverse tubules
a.p. reaches sarcoplasmic reticulum, causing it to release Ca^2+ ions
causes muscle contraction

67
Q

thickness of filaments

A
myosin = thick filaments
actin = thin filaments
68
Q

thin filament structure

A

consists of 2 actin subunits and tropomyosin molecules twisted around each other attached to troponin
anchored to Z-disks

69
Q

troponin structure

A
globular
made up of 3 polypeptides
1 binds to actin
1 to tropomyosin
1 to calcium ions when they are available
70
Q

thick filament structure

A

bundle of myosin molecules
each have 2 protruding heads at each end of molecule
heads are mobile and bind to actin when binding sites are exposed
anchored to M-line

71
Q

muscle contraction model answer

A

action potential passes along sarcolemma and down transverse tubules into muscle fibre
action potential carried to sarcoplasmic reticulum, released calcium ions into sarcoplasm

calcium ions bind to troponin
causes troponin to change shape, pulls tropomyosin aside and exposes binding sites to actin
myosin heads bind to actin to form actin-myosin cross-bridges when ATP is present
causes myosin head to move and actin filament to slide past the stationary myosin filament (power stroke)
ADP and Pi released from myosin during power stroke
ATP attaches to myosin head and causes it to detach from actin
ATP hydrolysed by ATPase on myosin head into ADP and Pi, provides energy to return myosin head to original position
myosin can reattach further up actin and repeat

when stimulus stops, calcium ions actively transported back into sarcoplasmic reticulum
calcium concentration falls until troponin and tropomyosin move back to cover binding sites

72
Q

role of ATP during contraction

A

supplies energy for contraction
during power stroke, ADP and Pi released from myosin head
new ATP molecule attached to myosin head, breaks cross-bridge
ATP is hydrolysed, releasing energy for myosin head to return to original position and repeat contraction mechanism further along actin filament

73
Q

maintaining supply of ATP

A

ATP available in muscle tissue needs to be regenerated very quickly to allow continued contraction via:

aerobic respiration in mitochondria (limited by delivery of oxygen to muscle tissue during intense activity)

anaerobic respiration in sarcoplasm (releases smaller amounts of ATP, leads to production and build up of lactic acid, toxic and causes fatigue)

creatine phosphate in sarcoplasm (reverse store of phosphate groups that can bind to ADP to form ATP rapidly, enzyme required, enough to support muscular contraction for 2-4 more seconds)

74
Q

enzyme required for creatine phosphate

A

creatine phosphotransferase

75
Q

why plant need to respond to their environment

A

avoid abiotic stress
maximise photosynthesis (obtain more sunlight/water)
germinate in suitable conditions
respond to and protect against predation or invasion by pathogens

76
Q

what plants respond to in environment

A

abiotic stress
tropisms
avoid herbivory/grazing

77
Q

how plants respond to abiotic stresses (environmental)

A

higher temperatures = more waxy layer
very windy = more lignification of xylem vessels
drought = root growth slows, stomata close (abscisic acid)

78
Q

how plants respond to tropisms

A

geotropism/gravitropism (roots grow towards soil to obtain more minerals and water)
hydrotropism (roots grow towards water to absorb more water required for photosynthesis)
phototropism (shoots grow towards sunlight to maximise sunlight absorbed for photosynthesis)
thigmotropism (grow up and around structures for support + anchor and obtain reactants for photosynthesis)
chemotropism (pollen grows towards ovule)

79
Q

how plants respond to herbivory/grazing

A
thigmonasty (e.g. folding in response to touch in Mimosa pudica)
chemical defences (tannins, alkaloids, pheromones)
80
Q

tannins

A

makes plant taste bad

defends roots against pathogen

81
Q

alkaloids

A

make tips of roots and shoots and flowers taste bitter

82
Q

pheromones in plants

A

can be produced when one leaf is eaten

communicates with other leaves to produce chemical defences

83
Q

cytokinins effect

A

promote cell division
delay leaf senescence
overcome apical dominance
promote cell expansion

84
Q

abscisic acid effects

A

inhibits seed germination and growth
causes stomata closure when plant is stressed by low water availability
inhibit lateral bud growth (promote apical dominance)

85
Q

auxins effects

A

e.g. IAA (indole-3-acetic acid)
promote cell elongation
promotes apical dominance (keeps abscisic acid levels high)
inhibit leaf abscission (leaf fall) by reducing ethene production

86
Q

gibberellins effects

A

promote seed germination and growth of stems

87
Q

ethene effects

A

promotes fruit ripening and leaf abscission

88
Q

nastic movement

A

plant movement that occurs in response to environmental stimuli but the direction of response is not dependent on direction of stimulus

89
Q

leaf abscission definition

A

leaf fall

90
Q

leaf senescence definition

A

ageing of leaves

chlorophyll degrades, causes leaves to change to autumnal colour

91
Q

apical dominance definition

A

inhibits lateral buds growing further down the shoot

causes shoots and buds to grow upwards

92
Q

plant hormone action

A

made in many plant tissues
act on wide variety of target tissues
move in xylem vessels or phloem tissue by mass flow up and down plant
then diffuse or active transport from cell to cell
binds to complementary-shaped receptors on plasma membrane
binding causes series of enzyme-controlled reactions (sometimes causing genes to be switched on/off) that brings about response

93
Q

differences between plant and mammalian hormone action

A

made in endocrine glands vs made in many tissues
move in blood vs move in xylem/phloem, from cell to cell
act on few/specific target tissues vs add on most tissues, act in cells where produced
act more rapidly vs acts more slowly

94
Q

similarities in plant and mammalian hormone action

A

binds to complementary-shaped receptor
causes cascade of events / enzyme reactions
may involve switching on/off of genes
only present in small quantities to cause effect
may have effect on more than one location/tissue
may involve interaction with more than one hormone

95
Q

auxin experiment method

A

take 15 seedlings, cut off tip and measure them
to 5 seedlings, cover end of tip with lanolin (wax) (A)
to another 5, cover end with lanolin infused with IAA
leave final 5 untreated (C)
after 3 days measure them
both A and C needed as lanolin alone is not causing effect and only IAA causes effect
height doesnt matter by measuring % change in height

96
Q

how to prove phototropism of plants method

A

collect 20 seedlings
mark stems every 2mm
plant 10 in one pot and 10 in another
set up lamps so one pot (A) gets light from all direction and the other (B) only gets light from one side
leave to grow for 4 days
measure distances between each mark and calculate mean distances for A and B for both shady and light sides

97
Q

tropism definition

A

plants responding to stimuli via growth

response is directional

98
Q

elongation method

A

auxins produced at apex of shoot
diffuse down shoot to zone of elongation
binds to receptors on cell surface membrane of cells
causes H+ ions to be actively transported into cell wall
low pH causes wall-loosening enzymes to catalyse breaking of bonds in cellulose
walls become more flexible
water enters cell, flexible wall allows cell to elongate

99
Q

phototropism method

A

auxins produced at apex of shoot
more phototropin enzymes activated on side with more light shining on it
phototropin enzymes cause PIN proteins to transport more auxins to shaded side
cells on shaded side of shoot elongate more quickly
shoot bends towards light

100
Q

mica vs gelatin

A

mica is impermeable (doesn’t allow diffusion)

gelatin is permeable (allows diffusion)

101
Q

what Darwin’s work showed about phototropism

A

tip was responsible for phototropism

102
Q

what Boysen-Jensen’s work showed about phototropism

A

substance responsible for phototropism

auxins must pass from the tip down to cause response

103
Q

what Went’s work showed about phototropism

A
showed chemical (auxin) from tip causes response
effect can be caused artificially if chemical was allowed to diffuse into agar block
104
Q

geotropism of plants

A

plant shoots show negative geotropism (up and against from gravity)
plant roots show positive geotropism (down and with gravity)

105
Q

showing geotropism experiment

A

collect 10 seeds
embed 5 in one Petra dish of moist cotton wool
other 5 in another
place one group (A) in the klinostat, allow to turn very slowly for 4 days
place B into klinostat, without turning
observe results

106
Q

auxin mechanism for geotropism

A

auxins produced at apex (tip) of shoot
if roots lying flat, auxin collects on lower side
auxin inhibits cell elongation in roots
upper side cells elongate, so root bends downwards

107
Q

where acetylcholine receptors are found

A

postsynaptic membrane in neuromuscular junction

108
Q

auxins commercial uses

A

weedkiller (promotes rapid shoot growth, plant can’t support itself, falls and does)
cuttings of plants dipped in rooting powder (promotes root growth)
micropropagation
make seedless fruits