Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

General Pathology > 4. Postmortem changes > Flashcards

4. Postmortem changes Flashcards

1
Q

Cooling off

A

algor mortis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Pale color

A

pallor mortis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Desiccation

A

exsiccatio postmortalis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Soaking

A

maceratio postmortalis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Postmortem clot

A

cruor postmortalis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

PM blood sedimentation

A

hypostasis postmortalis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Discoloration

A

imbibition

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Selfsoftening

A

autolysis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Grave wax

A

adipocere

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What does PM cooling off depend on?

A
  • hair, subcutaneous fat tissue
  • temperature of surroundings
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Reasons of increased PM temperature:

A
  • overtemperature (heat stroke, septicaemia)
  • spusmic muscle contraction (tetanus, strychnine or DiNitroOrtoCrezol poisoning)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Why corpse becomes pale?

A
  • blood is settling to lower parts of an animal
  • contraction of small vessels

NOT AN INFALLIBLE SIGN OF DEATH (ANEMIA)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Why dessication happens and can it happen in alive animal?

A
  • happens bc of evaporation (skin, mucous membranes, cornea)

*can happen in alive animals in case of necrotized skin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Why soaking happens?

A

*skin, organs filled with fluid

  • foetuses (aseptic autolysis)
  • carcasses staying in water
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Stiffness in skeletal muscles

A

– 2-4 hours the beginning

– 5-8 hours becomes general

– 24-48 hours starts to disappear

– 48-60 passes off

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Specificity of rigor mortis in spleen:

A

Has smooth muscle interstitium (trabeculae of spleen) which will contract and spleen will be rigid. Can be confused with lesion (septicaemia -› septic splenitis -› rigid spleen)

Is stiffness of a spleen a PM change or lesion???

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Stiffness in heart muscle

A

– Standstill in diastole – looks like systole

– Develops fast (30 minutes)

– Lasts for 1 day

18
Q

Stiffness in smooth muscles

A

– Quick process (10-15 minutes)

– Lasts for 1 – 4 hours

19
Q

Onset and duration of RM. Rapid and short

A

– High environmental and/or inner temperature

– Prolonged muscular activity

– Young and elder animals

– Septicaemia, wasting diseases

20
Q

Onset and duration of RM. Delayed

A

– Asphyxial death (notably by carbon monoxide poisoning)

– Severe hemorrhage, cold surroundings

21
Q

Onset and duration of RM. Fails to develop

A

– In case of degenerative muscle changes

22
Q

Postmortem clot. Most imp inf:

A
  • Not attached to the intima!
  • After death blood clots in 15-30 minutes (Heart, large blood vessels)
  • No clotting in small blood vessels - fibrinolysin
23
Q

Why self softening happens?

A
  • Autolytic ferments of the cell in the cytoplasm
  • Autodigestio (esophagus and stomach mainly)
  • type of autolysis
24
Q

Postmortem destruction. The most imp inf

A

Activity of saprogenic bacteria

Suffocation supports the putrefaction– blood remains liquid

25
Q

What factors increase the speed of putrefaction?

A
  • warm environment
  • obesity (body stays warmer for longer)
  • hyperemic organs (fulfilled with blood)
  • infection
  • injuries (port of entries for bacteris)
  • oedematous tissues
26
Q

Other types of discolourations:

A
  • sulph-hemoglobin (hemoglobin + hydrogen sylphid (H2S) -› greyish green color
  • pseudomelanosis (iron (from hemoglobin) + hydrogen sulphyd (H2S) -› black color)
27
Q

Are post mortem changes dependent on fatal disease?

A

No !

28
Q

Are post mortem changes influenced by endogenous and environmental factors?

A

Yes. Very much.

29
Q

Stiffness

A

rigor mortis

30
Q

Postmortem decomposition

A

putrefaction

31
Q

Average speed of cooling off in dogs

A

1 celcius degree / hour

32
Q

Sequence of appearing and disappearing of rigor mortis:

A

Head -› Neck -› Body -› Limbs

33
Q

2 most imp forms of imbibition:

A
  1. from blood (hemoglobin)
  2. from gall bladder (bilirubin)
34
Q

What organ is very easily putrefied? And why?

A

Liver. Bc there are saprogenic bacteria and through vena portae they will enter the liver.

35
Q

At what temperature putrefaction stops?

A

Under 5 degree celcius

36
Q

What is the difference between autolysis and putrefaction?

A

Autolysis - by own enzymes of the organism

Putrefaction - by bacteria

37
Q

Suffocation <-> putrefaction - ?

A

Suffocation supports putrefaction because blood remains liquid

38
Q

“Way” of putrefaction

A

Intestine -> vena portae -> liver

39
Q

What makes putrefaction rapid?

A
  • obesity (retaining of the body heat)
  • warm environment
  • hyperaemic organs
  • widespread infection
  • injuries (portals of entry)
  • oedematous tissues
40
Q

What makes putrefaction slow?

A
  • lean
  • dehydtration

General Pathology (19 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions