Wound Management Flashcards

1
Q

Othopedic and some soft tissue injuries will compromise this

A

Structural support

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2
Q

How do you restrain/analgesia a wounded horse for assessment?

A

a2 agonist + opioid

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3
Q

Good sedation, reversible, fast onset, potent analgesia

A

a2 agonists

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4
Q

Synergistic w/a2s

A

Opioids

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5
Q

How do you vaccinate for clostridium tetani?

A

Tetanus toxoid vaccine + booster 3wk later

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6
Q

How should you treat C. tetani if the vaccination status of the patient is unknown?

A

Booster w/tetanus toxoid

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7
Q

What is the most common reason that wound repairs fail?

A

Improper prep and assessment of the wound

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8
Q

Partial/full thickness wounds are classified as ______

A

Open

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9
Q

Crushing/contusion wounds are classified as ______

A

Closed

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10
Q

Period of time before there is more than 10^5 bacteria/g of tissue in the wound

A

Golden period

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11
Q

Is there a golden period for wounds in equine management?

A

No

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12
Q

Immediate closure for clean and clean-contaminated wounds

A

Primary (intention) closure (healing)

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13
Q

Closing a wound 2-5d after injury but before granulation tissue is produced

A

Delayed primary closure (healing)

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14
Q

Make sure wound bed is clean and as healthy as possible prior to closure

A

Delayed primary closure (healing)

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15
Q

Choice of closure for contaminated wounds or wounds w/questionable viability

A

Delayed primary closure (healing)

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16
Q

Closure >5d after injury

A

Secondary closure

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17
Q

Choice of closure for contaminated/infected wounds

A

Secondary closure

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18
Q

Granulation tissue, wound contracture, and epithelialization

A

Second intention healing

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19
Q

How do you decide when to close a wound?

A

Time, location, tissue availability, type of injury

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20
Q

What is the goal of wound closure?

A

Return damaged tissue to best fxn and appearance via manipulation of the healing process

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21
Q

What are the 3 phases of wound healing?

A

Inflammation/lag, proliferation, remodeling

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22
Q

Healing phase of hemostastis and acute inflammation

A

Inflammatory/lag phase

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23
Q

Healing phase of tissue formation

A

Proliferation

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24
Q

Healing phase of regaining strength

A

Remodeling

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25
Q

How is hemostasis initially affected by a wound?

A

Initial vasoconstrction, then vasodilation

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26
Q

How does platelet aggregation affect a wound?

A

Seals vessels, releases growth factors, fibrin deposition

27
Q

What cells release wound repair mediators?

A

Platelets

28
Q

What wound repair mediators do platelets release?

A

PDGF, TGF-B

29
Q

What cells remove damaged tissue and release chemoattractants?

A

PMNs, macrophages, fibroplasts

30
Q

What cells remain present for days to weeks and are imperative for wound healing?

A

Macrophages

31
Q

What do macrophages release to initiate the proliferative phase of wound healing?

A

Tissue growth factors

32
Q

When does angiogenesis, collagen deposition, and granulation tissue formation occur?

A

Proliferative phase

33
Q

What initiates angiogenesis?

A

Decreased O2 tension, high lactate, low pH in wound

34
Q

Release proteases to digest fibrin clot and arrange collagen into bundles parallel to the wound surface

A

Fibroblasts

35
Q

What type of collagen is initially produced and why?

A

Initially type 3 d/t dense blood vessel population

36
Q

What type does collagen production shift to as wound remodels?

A

Type 1

37
Q

How soon after injury does epithelialization begin?

A

Immediately

38
Q

How soon after injury does contraction begin?

A

2nd week after injury

39
Q

How much does contraction reduce a wound?

A

40-80%

40
Q

When does contraction stop?

A

When skin tension is greater than the ability to contrat

41
Q

Why are scars weaker than original skin?

A

New epidermis lacks dermal layer

42
Q

When does the remodeling phase begin and how long does it persist?

A

During 2nd week, lasts up to 2 years

43
Q

How much weaker is the final scar compared to the original skin?

A

15-50% weaker

44
Q

What is the single most important factor in successfully healing a wound?

A

Debridement

45
Q

How do you minimze sequestrum formation?

A

Debride outer cortex of bone to max blood flow to area

46
Q

Where do sequestrums most commonly occur?

A

Distal limbs

47
Q

What are the 3 tyeps of debridement?

A

Sharp, irrigation, direct contact

48
Q

What type of solution should you use to lavage a wound? What PSI/set-up?

A

Non-toxic (LRS, isotonic slaine, water) @ 7-8 PSI (1L bag set to 300mmHg)

49
Q

Slows down wound healing

A

Steroids

50
Q

Abx + steroid potion

A

Panalog

51
Q

Abx ointment potion that is carcinogenic

A

Nitrofurazone

52
Q

One of the ingredients in this potion is bleach

A

Vetricyn

53
Q

What are the topical treatments of choice for wounds?

A

Silver sulfadiazine, polysporin ointment

54
Q

Used for exudative or necrotic wounds

A

Hypertonic saline dressing

55
Q

Curasalt

A

Hypertonic saline dressing

56
Q

What is the best type of wound dressing?

A

AMD

57
Q

What is the active agent in Kerlix AMD?

A

PHMB

58
Q

How often do you need to change an AMD?

A

Q3-7d

59
Q

How often do you need to change a hypertonic saline dressing?

A

Q24-48hr

60
Q

How are wound vacuums beneficial?

A

Negative pressure = more rapid healing

61
Q

What should you NEVER use for wound dressing?

A

Corticosteroids

62
Q

Why is manuka honey the best honey to use for wound dressing?

A

UMF >15

63
Q

Requires sx excision, few topical agents, and bandaging

A

Chronic wounds