Week 1 Fluid Electrolyte Balance Flashcards
Hypervolemia
- Too much fluid
- Fluid volume overload
Hypovolemia
- Too little fluid
- Fluid volume deficit
Edema
- Excess fluid in interstitial space
Osmosis
- Movement of water
- High concentration to low concentration
- Across semi-permeable membrane
Diffusion
- Movement of particles
- High concentration to low concentration
- Electrolytes require active (facilitated) diffusion - ion pumps
- Across semi-permeable membrane
Filtration
- Movement of fluid through cell/vessel membrane
- Hydrostatic pressure differences
Hydrostatic Pressure
- Pressing of water molecules outwards from confined space
- High pressure to low pressure
Angiotensin II Function
- Vasoconstriction
- Stimulate thirst
Stimulate aldosterone to retain water & sodium
Atrial Natriuretic Peptide (ANP)
- Inhibits RAAS when in a state of overdrive
Hypovolemia Causes
- Insufficient intake
- Excessive loss
- Fluid shifts in body
Intravascular Space Deficit
- Difficulty perfusing body
- Systems activate to raise BP
Hypervolemia Causes
- Excessive intake
- Abnormal retention - kidney/heart failure
Intravascular Space Overload
- Raise BP
- Stress on systems
Cardiovascular Hypovolemia Findings
- Increased HR
- Thready pulse
- Decreased BP - orthostatic hypotension
- Flat veins
- Dysrhythmias
- Decreased peripheral pulses
Respiratory Hypovolemia Findings
- Increased RR
- Dyspnea
Neuromuscular Hypovolemia Findings
- Confusion
- Dizziness, weakness, lethargy
- Decreased LOC - coma
Integumentary Hypovolemia Findings
- Dry mouth
- Poor skin turgor - tearing
GI Hypovolemia Findings
- Decreased bowel sounds & motility
- Constipation
- Weight loss
- Thirst
Cardiovascular Hypervolemia Findings
- Increased HR
- Bounding pulse
- Increased BP
- Distended veins
- Dysrhythmias
Respiratory Hypervolemia Findings
- Increased RR
- Dyspnea
- Crackles on auscultation
Neuromuscular Hypervolemia Findings
- Confusion
- Headache
- Decreased LOC - coma
- Muscle spasms (electrolytes)
Integumentary Hypervolemia Findings
- Cool, pale skin
- Edema
GI Hypervolemia Findings
- Increased bowel sounds & motility
- Diarrhea
- Weight gain
Nursing Management: Fluid Deficit
- Correct underlying cause of deficit
- Replace fluids & electrolytes
- Prevent & assess inadequate perfusion
Nursing Management: Fluid Overload
- Prevention in at-risk clients
- Correct underlying cause
- Limit sodium/fluid intake
- Administer diuretics
Furosemide (Lasix) Action
- Increases renal excretion
- Mobilize excess fluid
- Decreases BP
Furosemide (Lasix) Side Effects
- Dizziness
- Headache
- Hypotension
- Electrolyte imbalance
Furosemide (Lasix) Nursing Considerations
- Fall risk of older clients
- Electrolyte imbalance
- Pre-existing kidney function & impact
- Monitor weight
- Avoid taking at night nocturnal urination disrupts sleep
Shock Causes
- Hypovolemic - hemorrhage
- Cardiogenic - myocardial infarction
- Distributive
- Neurogenic - spinal cord injury
- Anaphylactic - severe allergic reaction
- Septic - systemic infection
Compensatory Shock Symptoms
- Normal BP
- Increased HR & RR
- Blood shunting to vital organs
- Pale skin
- Hypoactive bowel sounds
- Decrease urine output
- Confusion
Progressive Shock Symptoms
- Decrease BP
- Decrease LOC
Healthy Tissue Perfusion
MAP - 70 to 100 mmHg
Hypovolemic Shock
- Decreased intravascular fluid volume
- External fluid losses
- Fluid shifts between intravascular & interstitial compartments (internal fluid losses)
Nursing Management: Hypovolemic Shock
- Call for help
- Notify MPR
- Position client in modified Trendelenburg
- Administer IV fluids, meds, blood products - according to provider orders
- Apply oxygen
Trendelenburg Position
- Head of bed down
- Blood moves to brain with less gravity
- Head of bed up - less gravity restricting breath, increases breath quality
Potassium K+
- 3.5-5mmol/L
- Intercellular electrolyte
- Maintains heart & muscle contraction
Hypokalemia
- <3.5mmol/L
- Not consuming enough K
- Loss (vomit, GI suction, sweat)
- Medications that move K (diuretics, insulin (K+ move into cell)
Hyperkalemia
- > 5.0
- Excessive intake
- Renal failure
- Medications that retain (ACE inhibitors, sparing diuretics, NSAIDs)
Hypokalemia Symptoms (7 L’s)
- Low BP & HR
- Lethargy
- Low shallow respirations (decreased ability to use accessory muscles)
- Lethal cardiac dysrhythmias (ST depression, shallow T wave, projecting U wave)
- Lots of urine
- Leg cramps
- Limp muscles (decrease deep tendon reflexes)
Hyperkalemia Symptoms
- Weak pulse, low HR
- Muscle twitches, cramping
- Resp failure
- Peaked T waves
- Prolonged PR interval
Hypokalemia Interventions
- Supplement K
- IV admin, high alert, administer slow (NO PUSH)
- Falls prevention
- Cardiac monitoring
Hyperkalemia Interventions
- Meds to lower potassium & support cardiac health (insulin, diuretics)
- Falls prevention
- Cardiac monitoring
Sodium Na+
- 135-145mmol/L
- Extracellular electrolyte
- Regulates water inside & outside cells
Hyponatremia Causes
- <135mmol/L
- Not consuming enough Na
- Hypovolemic - excessive losses, diuretics, vomiting, sweating
- Hypervolemic - excess fluid dilutes sodium
- Decrease serum osmolality
- Cells swell as water moves in
Hypernatremia Causes
- Overconsumption
- Syndromes that causes high cortisol/aldosterone = retain Na
- Water loss
- Increase serum osmolality
- Cellular dehydration
Hyponatremia Impact on CNS - SALT LOSS
- Seizures & stupor (decreased LOC, confusion)
- Abdominal cramping
- Lethargic
- Tendon reflexes diminished - trouble concentrating
- Loss of urine & appetite
- Orthostatic hypotension, overactive bowel sounds
- Shallow respirations
- Spasms of muscles
- Hypertension
Hypernatremia Impact on CNS - no FRIED foods for you
- Fatigue
- Restless, agitated, confused
- Increased reflexes
- Extreme thirst
- Decreased urine output, dry mouth/skin
- Tachycardia/hypotension
Hyponatremia Interventions
- Isotonic (non-severe)
- Na+ fluids <120
- Normal/excess fluids: meds (diuretics promote water loss)
Hypernatremia Interventions
- Health teaching - Na restricted diet
- Administer IV infusion - volume loss (hypotonic/isotonic)
- Meds (diuretics promote Na loss - loop)
Chloride Cl-
- 95-105mEq/L
- Blood pressure & volume maintenance
- pH balance
- Goes with Na, Na low = Cl low
Magnesium Mg2+
- 1.6-2.6mg/dL
- Neuromuscular contractility
Calcium Ca2+
- 4.5-5.5 mEq/L
- Neuromuscular contractility
- Coagulation
- Bone health
Phosphate P
- 1.9-2.6mEq/L
- Bone & teeth health
- Muscle & RBC function
- Inverse relation to Ca, Ca high = P low
Vascular Access Device Selection
- Duration: PIV (short) CVAD (long)
- Patency: PIV more risk for loss of patency
- History of vascular access & comorbidities: difficult prior access, skin, vessels
- Types of therapy: vesicant/irritants, pH
- Patients preference
- Always select smallest gauge (22 for PIV) & minimum # of lumens
PIV Selection
- Access to upper extremity
- Short term therapy <7 days
- Monitor for repeated failed/loss access
CVAD Selection
- PIV access unavailable
- Long term therapy
- Suitable for vesicant/irritant medications/nutrition
CVAD Access
- Large vein central circulation system
- Tip of catheter sits in superior vena cava
- Inserted by HCP with specialized knowledge
- Ultrasound guided technique
Common CVADs
- PICC
- Non tunneled
- Tunneled
- Implanted
PICC
- Enters body on upper arm
- Catheter runs to superior vena cava
- Very common in clinical settings
- RNs can insert & remove
- Medium term use
Non-Tunneled CVAD
- Enters body at vessel site (internal/external jugular, subclavian, femoral)
- Catheter outside body at injection site
- Common in critical care (shorter term)
Tunneled CVAD
- Hickman or broviac
- Proximal end tunneled subcutaneously from insertion site & brought out through skin at exit site
- Antimicrobial cuff
- Long term use
CVAD Infections
- High risk for sepsis
- PPE
- Anti-microbials
VAD Insertion
- All can cause phlebitis at insertion site
- Skin assessment