W7 - Paediatric Dermatology Flashcards

1
Q

how do you describe morphology

A
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2
Q

what is the birthmark?
small red mark at a few weeks of life - getting bigger

A

strawberry haemangioma

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3
Q

what is this

A

Strawberry Naevus/ Capillary Haemangioma

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4
Q

what is the aetiology of Strawberry Naevus/Capillary Haemangioma

A

Not present at birth - appear first month of life.
Get bigger until 6-10 months -> resolve.
Most fully resolve by 5-7 years.
Treatment: usually not needed. Near eye or obstructing airway = propranolol.
Complications: ulceration, bleeding + infection.

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5
Q

when would you treat starwberry naevus

A

over the eye - obstruct vision
in nappy area - get infected

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6
Q

what is the management of strawberry haemangioma

A

propranolol

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7
Q

what is the rash
2 day old gets spots - concern over chicken pox
baby well

A

Erythema Toxicum Neonatorum

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8
Q

what is the aetiology of Erythema Toxicum Neonatorum

A

Occurs in neonatal period (up to 28 days)
Raised yellow vesicles on an erythematous base
Occurs in 50% of babies, particularly term+
Completely benign
Settles in a few weeks

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9
Q

what is the rash
1 day old baby
has spots on head
mum has 2 ulcers
baby lethargic and not feeding

A

neonatal herpes simplex

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10
Q

what is the aetiology of neonatal herpes simplex

A

Very serious condition
Tends to affect babies whose mum has had this primary infection in pregnancy and gave birth vaginally
Usually occurs on scalp
Can disseminate to multiple organs including the brain
Needs urgent treatment with IV aciclovir
33% mortality rate (despite treatment)

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11
Q

why is neonatal herpes simplex so serious

A

urgent IV aciclovir
33% mortality even with treatment

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12
Q

what is primary herpes and why is that important

A

mums first infection with herpes - only primary herpes can be given to baby

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13
Q

what is the rash

A

stork mark \ naevus simplex

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14
Q

what is the aetiology of stork mark

A

Always midline
40% of neonates
Often: neck or forehead
A delay in the maturation of skin cells during embryonic development – dermal capillary dilatation
Usually disappears with time

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15
Q

what is this

A

milia

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16
Q

what is the aetiology of milia

A

Tiny white bumps on the nose, chin or cheeks.
Dead skin cells get trapped under the skin and form tiny cysts.
50% of neonates, but can also occur any age.
Treatment = not needed -> disappear on their own in a few weeks or months.

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17
Q

what is this

A

‘Cradle Cap’/Infantile seborrheic dermatitis

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18
Q

what is cradle cap

A

10% of infants
3 weeks -> 12 months
Cause unknown - ? Hyperactivity of sebaceous glands secondary to maternal androgens
Self limiting
Baby shampoo/oil, gentle brushing

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19
Q

what is this

A

infantile acne

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20
Q

why does this look odd for infantile acne

A

darker skin - harder to see

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21
Q

what is the aetiology of infantile acne

A

Affects cheeks, chin and forehead
Boys > girls
6w - 12 months
Papules, pustules, nodules, cysts + comedones
Aetiology unknown ? Genetic (normal hormone levels)
Benzoyl peroxide or erythromycin gel
Oral antibiotics

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22
Q

what antibiotics MUST NOT be used to treat infantile acne

A

tetracyclines - doxycycline
causes dental scaring

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23
Q

what is this

A

mongolian blue spot OR
slate grey naevus

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24
Q

what is blue spot

A

Very common birth mark in babies with darker skin tones
Occurs in 90% of east Asian babies
Area of darker pigmentation often on buttocks but can occur in thighs and arms
Doesn’t cause any harm will usually fade by 4 years of age
Always document if you see on a baby check

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25
Q

what is this

A

measles

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26
Q

what is the signs symptoms and presentation of measles

A

Maculopapular + starts behind ears
Prodromal phase: fever, corysa and conjunctivitis
Complications = encephalitis and subacute sclerosing panencephalitis

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27
Q

what are these lesions

A

coplic spots

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28
Q

what is this

A

varicellar zoster

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29
Q

what is the aetiology of varicella zoster rash

A

Papules which progress to vesicles
Often occur in crops
Infectious from 2 days before rash, until all lesions crusted over
Symptomatic management
Immunocompromised = zoster immunoglobulin

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30
Q

what is the treatemnt for varicella zoster

A

calamine/emollient, sedating antihistamine, paracetamol.

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31
Q

why must you NOT give ibuprofen

A

increased scarring and necrosis, increased risk chickenpox pneumonitis

32
Q

what is this

A

hand foot and mouth

33
Q

what is the aetiology of hand foot and mouth

A

Caused by Coxsackie virus
Tends to occur in children under 5y
Blisters on hands and feet and in mouth
Symptomatic Management

34
Q

what could cause hand foot and mouth to be more severe

A

if parents get infected
if baby has eczema

35
Q

what is this

A

Molluscum Contagiosum

36
Q

what is aetiology of Molluscum Contagiosum

A

Caused by pox virus
Umbilicated centre
Contagious
Self limiting: can take up to 2 years to clear

37
Q

how is molluscum contagiosum spread

A

normally scratching

38
Q

what associated condition MUST you consider

A

a compromised immune system
consider HIV

39
Q

what is this

A

parvovirus B19

40
Q

what MUST be considered with parvo virus

A

stay away from pregnant women - can cause miscarriage

41
Q

what is the aetiology of parvovirus B19

A

Common in children + can occur adults
Fifths disease
Fever, runny nose, sore throat, headache
Can cause miscarriage in pregnancy
Aplastic crises in haemolytic anaemia

42
Q

how would pityriasis rosacea present

A

starts with herald patch

43
Q

what is this

A

Guttate Psoriasis

44
Q

what is the aetiology of Guttate Psoriasis

A

developed widespread rash over trunk.
He is systemically well.
2 weeks ago he had antibiotics for tonsillitis.
A throat swab grew Strep

45
Q

what is this

A

Staph Scalded Skin

46
Q

what is aetiology of Staph Scalded Skin

A

Caused by a toxigenic strain of bacteria
Two exotoxins are produced which breakdown desmoglein 1
This results in separation of the skin through the granular cell layer
Tends to affect under 5 years
Skins looks red and blistered like a burn
Treat IV antibiotics

47
Q

what is this

A

axillary freckling

48
Q

what is this

A

cafe au late spot

49
Q

what is this

A

neurofibromas

50
Q

what is this

A

lisch nodules

51
Q

what links these images together

A

Neurofibromatosis Type 1

52
Q

what is the genetic inheritance of neurofibromatosis type 1

A

Autosomal Dominant

53
Q

what is this

A

adenoma sebaceum

54
Q

what is this

A

Ash Leaf patches

55
Q

what is this

A

Shagreen patches

56
Q

what condition link all 3 of these images

A

Tuberous Sclerosis

57
Q

what are some other features of Tuberous Sclerosis

A

Polycystic kidneys
Infantile spasms
Epilepsy
Learning difficulty

58
Q

what is this

A

port wine stain
(in ophthalmic division of trigeminal nerve)

59
Q

what is this

A

congenital vascular lesion in brain

60
Q

what condition links these 2 images

A

Sturge Weber syndrome

61
Q

what is Sturge Weber syndrome
associated with

A

epilepsy, learning difficulty and hemiplegia

62
Q

what is this

A

Epidermolysis bullosa

63
Q

what is aetiology of Epidermolysis bullosa

A

A rare genetic condition causing blistering of the skin and mucous membranes
Epidermis and dermis not adequately attached, so minor trauma causes separation.
Increased risk of skin cancer due to chronic inflammation.
Reduced life expectancy due to infections.

64
Q

what is this

A

meningococcal scepticaemia

65
Q

what is aetiology of meningitis scepticaemia

A

Caused by Neisseria Meningitidis, common nasal organism.
Meningococcal septicaemia occurs when the organism gets into the blood stream. It can kill a well child in hours.
Children present unwell with fever. The rash can start as blanching but usually becomes non blanching.
Lesions become non blanching + irregular with necrotic centres.

66
Q

what is the treatment for meningococcal septicaemia

A

80mg/kg of ceftriaxone IV.

67
Q

what vaccination is used for meningococcal disease

68
Q

what should be considered for family and close contacts

A

prophylactic antibiotics
rifampicin or ciprofloxacin.

69
Q

what is this

A

Henoch-Schonlein Purpura

70
Q

what is aetiology of Henoch-Schonlein Purpura

A

Raised palpable purpura on buttocks and extensor surfaces.
Most common vasculitis in children.
No fever + Child systemically well.
Associated with joint pain, joint swelling, abdominal pain and glomerulonephritis

71
Q

who gets henoch-schonlein purpura

A

Peak incidence: 4-10yrs.
M:F = 2:1
Recurrence in 1/3 patients in future.

72
Q

what is the risk in henoch-schonlein purpura

A

Risk of IgA nephropathy and renal failure.

73
Q

what should you do as an investigation for Henoch-schonlein purpura

A

Check urine for blood and protein. Measure BP – protocol.

74
Q

what is this

A

Immune Thrombocytopenic Purpura

75
Q

what is aetiology of Immune Thrombocytopenic Purpura

A

Petechial rash is caused by low platelets

Often associated with easy bruising and bleeding from mucosa

Systemically well

Tends to occur 1-2 weeks after a viral infection due to cross reacting antibodies

76
Q

what is overall treatment and management

A

Check FBC and film
Most don’t need treatment.
Treatment options include steroids and IVIG
Usually resolved in 6-8 weeks
Avoid contact sports until platelets >50