W6 - Sleep Opnoea - Abdalla Flashcards

1
Q

Describe the sleep cycle

A

Stage 1 - light sleep “twitches, jerking”

REM - first occurs after 90 min, then every 90 min thereafter

Stage 2 - brainwaves slow, assoc w/ memory consolidation

Stage 3+4 final and deepest stages of sleep

REM gets longer through the night

Deep sleep reduces through the night

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2
Q

Sleep disorders (6)

A

Insomnia

Parasomnia (sleep walking, talk, etc)

Sleep-related breathing disorders (OSA, snoring)

Sleep-related mvmt disorders - nocturnal bruxism

Narcolepsy

Circadian rhythm sleep disorders (jet lag)

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3
Q

What other chronic conditions can be worsened by OSA (4)

A

Increased risk of developing:

Heart failure

Heart attack

Schizophrenia

Anxiety/depression

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4
Q

What causes snoring in adults

A

Vibration of loose soft tissues in the airway as air passes over them

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5
Q

What causes snoring in children

A

Enlarged tonsils or adenoids

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6
Q

Snoring = sleep apnoea?

A

No

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7
Q

How does sleep apnoea get subclassified

A

Central sleep apnoea (CSA)

Obstructive sleep apnoea (OSA)

Complex (combination)

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8
Q

Features of central sleep apnoea

A

Airflow ceases due to a temporary lack of inspiration

CNS issue

  • Polymyelitis
  • Spinal cord injury
    • Encephalitis
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9
Q

Apnoea vs hypopnea

A

Hypopnea - reduced airlow for at least 10s, accompanied by arousal or drop in o2 sat

Apnoea - Total cessation of airflow for at least 10s

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10
Q

Central sleep apnoea vs obstructive sleep apnoea

A

CSA = CNS issue

OSA = physical obstruction of airway

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11
Q

Describe severity of apnoea for children and adult ranges

A
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12
Q

Describe the pathophysiology cycle of OSA

A
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13
Q

How does apnoea affect the sleep cycle and lead to symptoms?

A

Patients are unable to complete the sleep cycle - keep getting woken up

  • 27 min in
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14
Q

Symptoms of sleep apnoea (8)

A

Poor concentration

Low mood

Restless sleep

Heartburn

Waking up in the morning with dry mouth

Night sweats

Weight gain

Daytime fatigue

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15
Q

Modifiable risk factors of OSA adult

A

Obesity

Smoking

alcohol

Upper airway collapsibility

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16
Q

How can lifestyle be modified to reduce OSA (4)

A

Weight management

no drinking/smoking

sleeping position alteration (not supine)

CPAP

17
Q

Describe the comorbidities of OSA (4)

A

Hypertension

CVD

Stroke

Metabolic syndromes

18
Q

Dental risk factors of OSA (7)

A

High narrow palate

Narrow dental arches

Increased anterior face height

Increased overjet

Retrognathia

Large tongue

Tonsillar hypertrophy

19
Q

Why doesnt CPAP work very effectively in adults?

A

Poor pt compliance

20
Q

Differences btw adult and children OSA

A
21
Q

Concern with CPAP machine use in children

A

Hypomaxillary development

Class 3 / midface def

22
Q

Management of OSA children (4)

A

Adenotonsillectomy

Pharmacological agents to reduce lymphoid tissue

Weight loss if obese

CPAP (caution - maxillary retrusion)

23
Q

Why is assessing OSA important for dentists? (4)

A
  • Under diagnosed and reported - GP’s dont screen for it
  • Dentists see their pts regularly
  • Dentist can manage pts with provision of mandibular advancement devices
  • Should always be done under guidance of specialist sleep physician
24
Q

How to screen adults (mallampati score) for OSA

A
25
Q

How to screen adults for OSA using STOP BANG questions

A
26
Q

What anatomical feature to screen for OSA

A

Scalloped tongue

  • 70% diagnostic for OSA
  • Also sign of nocturnal bruxism
  • Tongue presses up against teeth during sleep
27
Q

3 ways of screening adults of OSA

A
  1. Check for scalloped tongue
  2. STOP-BANG questionnarie
  3. Mallampati Score
28
Q

How to screen children for OSA (5)

A

Epworth Sleepiness Scale

Paediatric sleep questionnaire

History of snoring, daytime sleepiness, ADHD

Brodsky scale for enlarged tonsils

Skeletal risk factors (narrow mx, mandibular retrognathia)

29
Q

What is the only way to truly diagnose OSA

A

Dentists can’t do it - we can only screen

Polysomnogram / Polysomnography test

30
Q

TG advice on how to manage OSA (4)

A

Diagnosis of facial skeletal retrusion (retrognathia)

Refer for medical examination and sleep lab investigation

Construction of md advancement splints BUT must be done in association with a multidisciplinary team led by resp specialist physician

Be mindful of GA - OSA pts are at increased risk of respiratory arrest with GA and sedation - do it in hospital if required

31
Q

Consideration of sedation and GA for dental treatment in OSA pts

A

increased risk of respiratory arrest

  • treat in hosp if required
32
Q

What is the role of the dentist/ortho in tx of OSA (3)

A
  • Skeletal problems causing malocclusion should be corrected (RME, functional appliance, etc) in adults
  • Mandibular advancement splints to be used in adults with mild-mod OSA who cannot tolerate CPAP
  • Bimax protrusion orthognathic surgery if all else fails
33
Q

Should ortho appliances and mandibular advancement splints be used to treat OSA in children?

A

No - insufficient evidence

34
Q

Features of mandibular advancement splints for OSA (4)

A
  • Better tolerated than CPAP
  • Less effective in severe cases
  • Can cause changes to occlusion over time
  • Adults need to be dentate for most appliances
35
Q

Risk factors for smaller upper airway in children (2)

A

Narrow maxilla

Mandibular retrognathia

Class 2

36
Q

What is the Brodsky scale for enlarged tonsils in kids

A