Voluntary Feed Intake Flashcards
Why is measuring voluntary feed intake of production animals important?
- Production (maximise intake)
- Economics (optimise intake)
- Weight control (balance intake and expenditure)
- Weight loss (reduce/minimise intake)
What is distension or fill feedback in ruminants?
The more the rumen becomes distended/filled, the more the feed intake reduces
- Rumen wall stretch
- Message sent to brain to stop eating and start ruminanting
What suggests distension/fill feedback in ruminants is not the only factor in determining voluntary feed intake?
If it was the only factor the cow would eat as and when the rumen empties bu they do tent to eat in phases
What are the volatile fatty acids produced in ruminants? How do they impact on VFI?
\+ Acetate % depression = (0.39 x acetate) - 5.18 More acetate = less intake \+ Propionate Almost as effective as acetate in causing depression \+ Butyrate Absorbed much slower from rumen, so more effective in causing ruminal stasis Problem with poorly fermented feeds
What factors decrease voluntary feed intake in ruminants?
Body fat Pregnancy Inert fill Diet Neutral Detergent Fiber Age Growth rate Production level Environment Stress Food palatability
When does water affect VFI?
Water drunk with food has little effect on VFI but water in plant material can have a significant effect on reducing VFI
What is the water holding capacity of feed?
May be loosely regarded as soluble fibre or non-starch polysaccharides
What is the effect of NSP (non-starch polysaccharides) on the GI of ruminants?
Slowing down the passage of feed through the intestines - this is good as allows for more absorption but must be balance to maximise intake
What is inert fill?
Effect of bulky volume in cows abdomen either with rumen or outside the GI (e.g. foetus)
How does inert fill affect rumen outflow rate?
Particle size reaching the omasum (<1 cm able to pass).
Depends on ease of feed breakdown by chewing/fermentation and pressure within rumen
What disturbs rumen outflow rate?
Ruminal acidosis
What is the glucostatic theory?
Short term control.
Glucoreceptors in CNS sensitive to rate of glucose use.
Low glucose use - excite neural activity to stimulate hunger sensations and vice versa for high.
What is the hepatic oxidation theory?
ATP levels in liver determine vagal stimulation; low levels stimulate hunger centre
What are sources for ATP in the liver in ruminants?
VFA
BHBA (Beta-hydroxy-butyric-acid)
NEFA (Non-esterified fatty acids)
How does leptin affect VFI?
Decreased feed intake but in domestic animals selected for increased growth and energy conservation, role of leptin is largely unknown.