URR other cerebral abnormalities Flashcards

1
Q

vertebral artery stenosis

A

-difficult to diagnose with US due to portions of the a are located within the trans proceses of the vertebrae
-normal flow is low resistive
-most common location of stenosis is at the vertebral origin
-can lead to steal phenomenon

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2
Q

diagnostic criteria for vertebral artery stenosis/occlusion

A

-abnormal PSV > 100 cm/sec in most stenosis
-ratio of pre and post stenotic velocities >2
-abnorm pulsatility (biphasic or triphasic)

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3
Q

-occlusion or stenosis or prox subclavian artery
-most common on left side
-dist subclavian artery fed by reversed flow through ipsilateral vert artery
-retrograde vert flow on affected side
-if suspected, bilat brachial pressures should be obtained, decreased or greater than 20 mmHg will be seen on affected side
-symptoms include limb paralysis, vertigo, ataxia, and syncope

A

subclavian steal

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4
Q

subclavian steal happens more often on what side?

A

left

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5
Q

proactive maneuvers can be done to make partial or complete steal

A

-if bidirectional or biphasic in vert , exercising the arm or applying a blood pressure cuff can be used to convert the flow to complete steal
-if suspected subclavian a should be evaled, mono flow detected on affected side
-ask patient to perform 40 flexion/ extensions of arm on affected side then re-eval the vert flow
-inflate above syustolic pressure on ipsilat arm and maintain for 1-2 mins, replace cuff and asess flow in abnormal vert a, if steal present, the flow should change from biphasic to complete reversal

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6
Q

early systolic deceleration in vert a

A

-stenosis of subclav a can cause changes in vert a flow
-systolic deceleration is a sign of a significant stenosis in ipsilat side
-systolic flow starts with sharp peak followed by a sharp decrease in flow to baseline
-then resumes, increase to decrease through diastole
-this is caused by stenosis in subclav a and less commonly the prox vert A
-waveform is considered a sign of “pre steal”
-provacative maneuvers can be done to make partial or complete steal

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7
Q

-inflammation of A wall
-causes difffuse or focal thickening of muscle layer
-may cause increased vasc in wall of inflamed segment
-increased velocities with decrease lumen size

A

arteritis

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8
Q

buerger, takayasu, polyarteritits, temporal (giant cell)

A

types of ateritits

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9
Q

the superficial temporal A courses anterior to the ear and can be palpated just above what?

A

the zygomatic arch

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10
Q

-inflammation of the superficial temp A leading to flow reduction to frontal and parietal branches
-stiffness in neck, jaw, claudication, headaches, and sudden uni blindness
-biopsy necessary for diagnosis

A

temporal arteritis (GIANT CELL)

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11
Q

what does temporal arteritis look like?

A

-images have thickened walls
-halo sign = ring of thickened vessel wall surrounding vessel
-normal flow patterns mimic ECA
-diffuse vessel wall thickening and distal ischemia cause increase velocity and decreased flow resistance
-areas of greater wall thickening can cause focal stenosis with increased resistance prox to stenosis and decrease resistance distal to stenosis

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12
Q

aneurysms area rarely seen where?

A

cervical carotid branches

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13
Q

if an aneurysm is found in intracranial vessels, it is what kind?

A

berry

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14
Q

-blood escapes into surrounding tissues and becomes encapsulated
-risk of hemorrhage/death, trauma, infection, or true aneurysm

A

pseudoaneurysm

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15
Q

what is an AVM?

A

congenital connection between arterioles and venules (lack of capillary bed); usually has multiple connections and surrounding venous collaterals and dilated veins

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16
Q

severe form of intracranial AVM in fetuses?

A

vein of galen aneurysm

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17
Q

what is an AV fistula?

A

direct connection between aterial and venous systems caused by tauma or vascular procedure, usually single connection and no surrounding collaterals
-causes increased volume of flow in venous system with pulsatility and turbulence
-reduces resistance in arterial inflow and increased diastolic flow
-large or chronic arteriovenous connections can lead to heart failure

18
Q

-acute tear in vessel intima
-creates false lumen
-associated with marfan syndrome and ehler danlos syndrome

A

dissection

19
Q

if ICA affected by dissection, usually affects which part?

A

first 2-4 cm

20
Q

CCA dissection can by continuation into where?

A

aortic arch

21
Q

dissection is more common in ICA or ECA?

22
Q

-collagen growth in medial layer
-most common in renal A
-usually occurs mid-distal ICA (atherosclerosis more common in prox)
-causing flow limited lesions

A

fibromuscular dysplasia

23
Q

what does fibromuscular dysplasia look like?

A

-string of pearl appearance
-abnorm growth of medial layer
-doesnt shadow
-causes stenosis/occlusion

24
Q

carotid body (sinus):

A

-located at CCA bif, near angle of jaw
-above this numerous baroreceptors are located to monitor BP

25
what is the therapeutic and diagnostic carotid sinus massages?
therapeutic: -used to interrupt paroxysmal atrial tachycardia -performed to reduce heart rate -causes transient block in conduction of the electrical signal in heart diagnostic -used to eval patients hypersensitivity of the sinus -may be performed if no stenosis in extracranial As -abnormal response will lead to hypotension, bradycardia, and dizziness cant be performed on patients with hx of TIA, bruit, or stenosis
26
carotid body tumor/glomus tumor, chemodectoma/paraganglionoma?
-rare neoplasm composed of paraganglion tissue -more common in females -seen in people living in high altitudes -high vasc structure fed by ECA -overgrowth of normal structure that is responsible for detecting blood gasses/pH -can compress ICA -pulsatile lat cervical mass -dysphasia, syncope, hoarseness, dyspnea, cough -horner syndrome -pharyngeal bulge
27
thromboembolic disease?
-thrombus accumulation within arterial system that produces embolisms that travel distally -symptoms vary with occlusion site of embolism -can cause multiple TIAs
28
neointimal hyperplasia (myointimal hyperplasia)
-intimal thickening post endarterectomy or stent placement (smooth muscle rapid thickening) -flow limitations lesions -causes higher velocities in ICA -leads to restenosis or occlusion within 2 years of surgery
29
PHACE syndrome
-Posterior fossa malformations (such as dandy walker) , Hemangioma, Arterial anomalies, Coarctation of the Ao and other cardiac anomalies, Eyes -arterial stenosis or occlusion of cerebral As -abarent origin or coarse -saccular aneurysms of any cerebral As
30
-elongation of the styloid process or a calcified sytlohyoid ligament causes carotid compression -most common bilat -visual disturbances, syncope, carotid dissection
eagle syndrome
31
increased heart output can cause ?
increased flow in As
32
decreased heart output can cause ?
reduced resistance and flow velocity in A system and pulsatile flow in venous system
33
right heart valvular disease can cause abnormal?
pulsatile decreased flow
34
80-90% of atrial emboli are from what?
cardiac source
35
atrial fibrilation (irregular HR) can lead to clot formation in the left atrium which could lead to what?
-if it breaks off, can float anywhere -if foramen ovale is patent, could go into right heart
36
aortic stenosis?
-usually degenerative disase causes but can also be from congenital malformation of bicuspid valve -flow increases in velocity -flow into ascending aorta will be turbulent -brachial pressures decrease -characteristics can be passed to carotids
37
aortic regurgitaiton
-blood flows back through aortic valve to left ventricle during diastole -caused by disease valve, congenital defects, and chamber of aortic dilatation -significant can lead to volume overload occurs in left vent -pulsns bisferiens can occur in carotis -waveforms have double diastolic peaks -severe regurg is associated with diastolic flow reversal in carotids (water hammer pulse)
38
mitral stenosis
-bilat tardus parvus in CA can indicate severe -blood flow reduced in left atrium to left vent -significant stenosis can cause left atrial pressure overload with reduced cardiac output -CA will have low resistance, low velocity
39
mitral regurg
-blood moves back through mitral valve into left atrium during systole -caused by diseased valve, congenital defects and changer dilatation -significant regurg results in volume overload in chamber recieving the retrograde flow -with mitral regurg, volume overload occurs in left atrium -usually has no effect on atrial or venous doppler
40
cardiac temponade?
-critical -severe pericardial effusion within fibrous pericardium causes intrinsic compression of right heart chambers -respiratory variation in arterial flow -flow decreases with inspiration and normalizes with expiration -another indication: pulsus alternans(peak systolic velocity alternates btwn 2 levels with each beat), also associated with dilated cardiomyopathy and constrictive pericarditis
41
intra-aortic balloon pump (IABP)
-for severly and left vent function -implanted in descending ao and expands and contracts to cause blood to flow -have 2 systolic peaks for each pulse -2nd : balloon inflation -EDV cant be determined because of flow reversal
42
left vent assist device (LVAD)
-used for end stage heart failure -can prolong life for transplant patients and those who cant transplant -pulls blood from LV into a pump transports blood to AO -brachial measurements must be recorded -diminished PSV, increased diastolic -can cause fake negative for CA stenosis