Unit 7 Eating Obesity Flashcards
What is the difference between homeostatic and non-homeostatic eating?
a) Only non-homeostatic eating involves the brain
b) Homeostatic is based on energy needs, non-homeostatic on emotion/reward
c) Homeostatic is unhealthy
d) Non-homeostatic eating is triggered by blood glucose
b) Homeostatic is based on energy needs, non-homeostatic on emotion/reward
Explanation: Homeostatic eating is metabolically driven, while non-homeostatic eating is influenced by stress, emotion, and external cues.
What does leptin do?
a) Increases appetite
b) Inhibits insulin secretion
c) Stimulates ghrelin release
d) Signals satiety and inhibits feeding behavior
d) Signals satiety and inhibits feeding behavior
Explanation: Leptin is released by adipose tissue and helps reduce food intake by acting on the hypothalamus.
What is ghrelin’s role in appetite regulation?
a) Inhibits feeding centers
b) Stimulates satiety
c) Increases hunger when the stomach is empty
d) Promotes fat storage
c) Increases hunger when the stomach is empty
Explanation: Ghrelin is secreted by the empty stomach and stimulates appetite through hypothalamic centers.
What is leptin resistance and how does it relate to obesity?
a) Brain fails to respond to high leptin, hunger persists
b) Low leptin levels promote fat storage
c) Leptin inhibits insulin sensitivity
d) Leptin increases in underweight individuals
a) Brain fails to respond to high leptin, hunger persists
Explanation: In obesity, high leptin levels fail to suppress appetite due to resistance in hypothalamic pathways.
What are the ob/ob and db/db mouse models used to study?
a) Ghrelin signaling
b) Insulin resistance
c) Pancreatic beta cell damage
d) Leptin deficiency and leptin receptor deficiency
d) Leptin deficiency and leptin receptor deficiency
Explanation: ob/ob mice lack leptin; db/db mice have nonfunctional leptin receptors. Both become obese and hyperphagic.
