Unit 2 - Cardiovascular System Part 2 Flashcards

1
Q

myocardial infarction

A

occurs when there is a complete interruption in blood/O2 delivery to the heart muscle from a blockage in an artery
-result is necrosis (tissue death)

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2
Q

acute MI (AMI)

A
  • typically result of significant CAD that culminates in a complete blockage
  • ischemia often a precipitating factor
  • often fatal - 30%
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3
Q

transmural

A

full thickness MI affecting all 3 layers of the heart

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4
Q

nontransmural

A

partial thickness MI involving sub endocardial layer

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5
Q

most common sites of coronary artery occlusion

A
  1. left anterior descending artery (LAD)
  2. left circumflex artery (LCX)
  3. right coronary artery (RCA)
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6
Q

STEMI

A

“ST Elevation Myocardial Infarction”. Typically “transmural” infarctions
~70% of AMI’s are STEMI

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7
Q

non-STEMI

A

MI that does not demonstrate ST segment elevation on the EKG – Typically “subendocardial” infarctions

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8
Q

pathogenesis

A

The rupture of a vulnerable atherosclerotic plaque with subsequent thrombus formation, appears to be most common cause of AMI.

“soft” plaques are more likely to rupture than “hard”

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9
Q

diagnosis of MI

A
  • similar to angina but more profound/severe
  • chest pain, jaw/neck/shoulder discomfort, dyspnea, overwhelming fatigue
  • **may also include nausea and diaphoresis
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10
Q

EKG changes

A

“injury” pattern is often noted

hallmark characteristic is ST segment elevation (indicator of tissue damage)

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11
Q

Serum enzyme levels

A

elevations in:

  • troponin
  • CPK-MB
  • LDH “flipped ratio”
  • AST
  • myoglobin
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12
Q

major complications of MI

A
Dysrhythmias
Heart Failure (HF)
Sudden Death Syndrome
Mural thrombus / CVA
Ventricular aneurysm
Ventricular rupture with tamponade
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13
Q

dysrhythmias

A

Multiple PVC’s - indicates myocardial irritability
Ventricular Tachycardia
Ventricular Fibrillation

requires immediate treatment - medication or defibrillation

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14
Q

heart failure

A

heart is unable to pump enough blood through the body

causes: decrease myocardial capacity, intrinsic muscle disease, increased BP, valvular disease

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15
Q

sudden death syndrome

A

death occurring within 1 hour of the onset of cardiac symptoms

in adolescents/young adults under 30, “hypertrophic cardiomyopathy” is most common cause

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16
Q

mural thrombus/CVA

A

blood clot on endocardial wall

if the thrombus breaks loose, it can travel to the brain causing a CVA/stroke

17
Q

ventricular aneurysm

A

when necrosis occurs during MI, the tissue is vulnerable during the healing stage and may develop an abnormal ballooning

18
Q

ventricular rupture with tamponade

A

heart becomes compressed and can no longer contract and relax normally

19
Q

medical and surgical management of MI

A
  1. treat symptoms (rest, O2, meds)
  2. limit damage (meds, surgery)
  3. secondary prevention (lifestyle changes, rehab)
20
Q

sternal precautions

A

No lifting, pulling, pushing (10# limit) for 6 wks
Log roll technique in/out bed
No driving (4-8 wks)
ROM exercises – neck, shoulders, torso (“caution with sternectomy”)
Scar mobilization when incision is healed
Be conservative if: osteoporosis, diabetes, advanced age

21
Q

Phase I of cardiac rehab

A

= Inpatient phase: (typically 3-7 days) - review sternal precautions, initiate physical activity and provide home exercise/activity guidelines. Refer to comprehensive out-patient cardiac rehabilitation program!!

Note: monitor vital signs pre, during, and post exercise. Be aware of contraindications to exercise in the cardiac patient.

22
Q

Phase II of cardiac rehab

A

Acute outpatient: (may last up to 12 weeks) comprehensive program including individually prescribed and monitored exercise, and individual and group educational sessions aimed at reducing risk factors and secondary events.

23
Q

Phase III of cardiac rehab

A

Follows phase II: (may last 6 months or more) a continuation of phase II but patients no longer receive continuous telemetry monitoring during exercise and are more independent.