Unit 12 - Positioning & Nerve Injury Flashcards
how does the awake patient compensate for moving from sitting to standing position
SNS activated (baroreceptor reflex), which combats the effect of gravity (venous pooling) and ensures perfusion of brain and other vital organs
how does GA attenuate the body’s protective mechanisms against position changes
impaired baroreceptor responsiveness and decreased SNS tone
now does neuraxial anesthesia attenuate the body’s protective mechanisms against position changes
sympathectomy
how does positive pressure ventilation attenuate the body’s protective mechanisms against position changes
increased intrathoracic pressure results in decreased venous return
how do muscle relaxants attenuate the body’s protective mechanisms against position changes
decreased skeletal muscle tone = decreased venous return
Frank-Starling curve in trendelenburg and lithotomy positions
shifts to the right
blood shifts toward central circulation, increasing venous return
MAP in trendelenburg and lithotomy positions
stays the same or increases - venous return initially increases but is followed by vasodilation and a slower HR
complications of increased venous pressure in Trendelenburg and lithotomy positions
hydrostatic pressure leads to edema of face, eye, and airway
increased ICP
Frank Starling curve in sitting, flexed lateral, and prone positions
curve shifts to the left
blood shifts away from central circulation, resulting in venous pooling and decreased venous return
why are sitting, flexed lateral, and prone positions associated with higher incidence of hemodynamic instability?
anesthesia impairs baroreceptor responsiveness, so SNS is unable to fully compensate for preload reduction
results in decreased SV, CO, and BP
what 4 positions are assoc. with higher incidence of hemodynamic instability under GA?
- reverse trendelenburg
- sitting
- flexed lateral
- prone
name 5 common anesthesia techniques that attenuate the body’s compensatory mechanisms for maintaining CV stability in Trendelenburg position
- general anesthesia
- neuraxial anesthesia
- positive pressure ventilation
- PEEP
- muscle relaxants
4 consequences of abdominal shift in Trendelenburg position
- diaphragm moves cephalad
- FRC decreased
- pulmonary compliance decreased
- risk of endobronchial intubation increased
3 respiratory changes in the awake and spontaneously breathing patient vs. anesthetized and spontaneously breathing patient
- decreased Vt
- decreased FRC
- increased closing volume
changes to perfusion in the dependent lung in lateral decubitus position
- increased blood flow
- increased vascular pressure
- decreased vascular resistance
ventilation changes in the dependent region of the lung in upright position
- increased alveolar ventilation
- increased alveolar compliance
- increased PACO2
- decreased PAO2
ventilation changes in non-dependent lung region in upright position
- decreased alveolar ventilation
- decreased alveolar compliance
- decreased PACO2
- increased PAO2
perfusion changes in the non-dependent region of the lung in lateral decubitus position
- decreased blood flow
- decreased vascular pressure
- increased vascular resistance
how do neck flexion and extension affect ETT placement
flexion pushes the ETT tip towards the carina
extension pulls the ETT tip towards vocal cords
how does airway edema form in prone, trendelenburg, and sitting positions
prone and trendelenburg: increased hydrostatic pressure results in edema formation
sitting: neck flexion impaires venous drainage from head and results in edema
where may the tip of the endotracheal tube settle when a patient is shifted into Trendelenberg position?
mainstem bronchus - abd contents shift cephalad and pushes diaphragm towards ETT, increasing risk of endobronchial intubation
why does every surgical position increase the risk of brachial plexus stretching/injury
when is the risk of stretch injury highest?
the brachial plexus is anatomically fixed at the cervical vertebrae and axillary fascia
risk of injury highest when arms ABducted > 90 degrees and head is rotated to other side
what causes compression injury of the brachial plexus
brachial plexus is compressed as it passes between the clavicle and first rib or by an external force (ex. shoulder braces, improperly placed axillary roll)
when is there a risk of brachial plexus compression during open heart surgery?
excessive sternal retraction during median sternotomy can compress the brachial plexus under the first rib
what is a safer option than shoulder braces in the Trendelenburg position?
if shoulder braces are used, where should they be placed?
non-sliding mattress
if used, place at the distal end of each clavicle over the acromion
when might the arms need to be tucked in prone position?
assess for thoracic outlet syndrome in preop - ask pt to clasp hands behind their head. if they c/o pain, may be prudent to tuck arms in prone position
where is the axillary roll placed in lateral decubitus position
what is a good monitor for neurovascular compression with an ax roll
distal to the axilla
weak SpO2 signal in dependent arm
leg positioning in lateral decubitus position
downside thigh and knee are flexed and padded
upside thigh and leg are extended and separated from lower leg with pillows
where should the retaining strap be placed on the patient in lateral decubitus position
placed across the hip and fixed to the underbelly of the OR table. strap should be placed between iliac crest and head of femur
second strap can be placed over thorax or shoulders
what type of brachial plexus injury can occur when a bean bag is used for positioning?
compression
how high above the head can the arms be safely positioned in prone position?
arms should not be extended over the head - keep shoulders and elbows at 90 degrees or less
presentation of ulnar nerve injury
- impaired sensation of 4th and 5th digits
- inability to ABduct or oppose pinky finger
- chronic injury presents with claw hand (muscular atrophy)
most commonly injured peripheral nerve
ulnar nerve
mechanisms of ulnar nerve injury
- external compression (tight arm strap on forearm)
- elbow flexion = increased distance between medial epicondyle and olecranon = decreased cubital tunnel size = increased pressure on ulnar nerve
5 risk factors for ulnar nerve injury
- male (esp. > 50 yrs old)
- preexisting ulnar neuropathy
- body habitus extremes
- prolonged hospital stay/bedrest
- cardiac surgery
when does ulnar neuropathy typically present
> 24 h postop
what should you do if a pt has ulnar sensory deficits postop?
- neurology consult within first week of injury
- typically resolve in 5 days or less
recovery time for ulnar nerve injuries with motor deficits
4-6 weeks if demyelination involved
some are irreversible