Tricuspid Regurgitation Flashcards

1
Q

What is the difference between Functional and Organic etiologies of TR?

A

functional: valve is normal but there’s secondary causation of TR

organic: primary caused of abnormal TV

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What are the FUNCTIONAL CAUSES of TR?

A
  1. annular dilatation due to RVE/RAE
  2. pulmonary HTN
  3. RV ichemia/infarct leads to PM dysfunction & tenting
  4. AICD or Pacer Leads
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What measurement is considered SEVERE for Annuluar Dilatation?

A

> 40 mm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What causes PULMONARY HTN?

A
  • primary: idiopathic ( cause unknown, genetic)
  • secondary: heart/lung disease
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What are the COMPLICATIONS WITH AICD leads?

A
  1. worsening TR in 25%
  2. thrombus formation
  3. vegetations
  4. perforation
  5. subclavian obstruction
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Leadless pacers are available for?

A

bradyarrhythmia that can be paced from one chamber

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Where are the leads placed through and placed?

A

through subclavian vein into RA, RV and LV

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What are the ORGANIC CAUSES of TR?

A
  1. rheumatic
  2. carcinoid
  3. endocarditis
  4. ebstein anomaly
  5. myxomatous TV Prolapse
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is EBSTEIN ANOMALY?

A

congenital condition where on or more tricuspid leaflets displaced toward ventricular apex

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What do you visualize with Ebstein Anomaly?

A
  • septal leaflets most often involved
  • RAE
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What are the SIGNS and SYMPTOMS of TR?

A
  • signs of overload to right heart and cava
  • jugular venous distension
  • hepatomegaly
  • peripheral edema
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What types of AUSCULTATION are heard in TR?

A
  • holosystolic high pitch blowing murmur at xyphoid LSB
  • right sided S3
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What is RIGHT SIDED S3?

A

when TV is opened trying to move overloaded RA volume into RV - heard at mid diastole

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What are the TREATMENTS of TR?

A

none required bc usually tolerated but if significant TR - repair/replacement as MV

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What are the 2D FINDINGS of TR?

A
  1. RVE
  2. RAE
  3. dilated IVC/HV
  4. paradoxic septal motion due to RV volume overload
  5. flattening of IVS and D shaped LV in PSSAX due to RV volume overload
  6. abnormality of leaflet or chord
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Whata are the M MODE findings of TR?

A
  1. RVE
  2. paradoxic septal motion due to RV volume overload
  3. B bump on TV
17
Q

What are the COLOR/DOPPLER findings of TR?

A
  1. aliasing jet into RA during systole
  2. sign TR demos holosystolic flow below baseline in systole
18
Q

In what ways to we QUANTIFY TR?

A
  1. Jet area/RA area x 100
  2. jet area (color)
  3. vena contract
  4. simplified PISA radius
  5. EROA (PISA) and RV
19
Q

What indicates SEVERE TR?

A

presence of flow reversal in IVC/Hepatics/SVC

  • in systole, s wave above baseline
20
Q

What would indicate SEVERE TR of CW Doppler signal?

A

when as bright as the antegrade flow

21
Q

What would indicate SEVERE TR of shape of Doppler Envelope?

A

dagger or v shaped TR jet

22
Q

Dominant E wave indicates?

A

severe TR of > 1 m/s