Traumatic Head Injury Flashcards

1
Q

Definitiation of head injury

A

1 of following

Loss of consciousness
Amneisa after traumatic event
Confirmed brain injury
Skull fractie

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2
Q

Gender and age with head injury

A

Risk is 2X greater in males and risk of fatal 4X in males

Most in 15-24 y/o

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3
Q

Causes of head injury

A

Most a vehicular accidents

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4
Q

Lesions in various spots

A

Frontal - asympto
Spianl cord - paralysis
Brainstem - death

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5
Q

Penetrating
Blunt
Open

A

Penetrating - agent directly pierces into scalp, skull, meninges and brain tissue

Blunt - object deforms the head but no penetration…accidents and falls

Open - skull fracturd

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6
Q

Closed
Coup
Contrecoup

A

Closed - scalp may be cut but everything else in tact
Coup - injurys lie directly beneath POI
Contrecoup - injury to those that lie opposite to point…when brain moves and strikes other side of skull

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7
Q

Types of head injury

A

Skull fracture
Parenchymal brain injury
Vaqscular injury

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8
Q

Skull fractures location, types

A

Most are in thin parts
Most are linear (radiate opposite direction from POI)
Most do not cross suture lines of skull…tend to isolatei n one bone

Diastatic fracture crosses suture lines and involves more than 1 bone

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9
Q

Depress skull fracture

A

When a portion of skull bone displaced into cranial cavity by distance greater than thickness of the bone

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10
Q

COmplications of skull fractures

A

If no damage to brain or meninges, then heals

Damage to parenchyma (risk with depressed frcture)

Trauam to intracranial blood vessels with hemorhhage (esp epidural hematoma)

Bacterial meningitis in open

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11
Q

Concussion

A

Immediate disruption of CNS

Temportary and reversible

Blunt injury causes change in momentum of head

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12
Q

Concussion syns and smyptoms

A

Loss of consciousness, temporary resp arrest, loss of reflexes, loss of muscle tone

Recovery in 24 hours

Amnesia almost always accompanies

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13
Q

CTE

A

Chronic traumatic encephalopathy

Degen dz from repeated brain trauam

Histologic changes mimic alzheimers (tau protein)

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14
Q

Contusion

A

Bruise of brain surface by blunt trauma

Dura may be torn but leptomeninges intact

Coup or contrecoup

Occur at gyri of cerebral cortex

Wedge shaped area of hemorrhae with bae of wedge at brain surface

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15
Q

Clinical contusion

A

Headache
Dizziness
mental status changes
Seizures

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16
Q

Lacerations

A

Disruptions of leptomeninges and brain parenchyma

Pentrating head wounds like depressed skull fractures

Heals by gliosis and can serve as focus for seizure activyt

17
Q

Epidural hematoma

A

Spacce occupying intracranial lesions
5-15% of all fatal
Typically coup
Accompanied by skull fractures

18
Q

Mech of epidural hematoma

A

Most are trauam to dural arteries

Middle meningeal is most common

19
Q

Epi dural comps and clinical course and tx

A

Compression of prain
Increased can lead to tonsillar hernation which can lead to compression and necrosis of brainstem

Acute and rapidly progressive due to high pressure bleeding…emergency

Typical clinical picture is loss of consciousness after injury followed by lucid interval followed by loss of consciousness again

Neurosrugery where hole drilled in skull

20
Q

Subdural hematoma and groups affected

A

Space occupying
Bridging veins

Battered infants
Alcoholics
Boxers
Elderly - brain atrophy leads to strethcing of veins

21
Q

Clinical course and tx of subdural hematoma

A

Slowly bleeding because of veins

Confusion, loss of consciousness, disoreintation, hemiplegia, coma, death

Occur weeks or months after injury

tx is same

22
Q

SAH

A

Always results in bloody CSF
Most common are veins in subarachnoid space

Blood is in contact iwht meninges and can cause chemical meningitis

Blood can cause vasopasm of other blood veseels leading to ischemic injury and infarcts of other brian areas