Thermoregulation Flashcards

1
Q

The thermoregulatory centre is located where in the brain

A

The hypothalamus

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2
Q

Heating mechanisms

A

Basal metabolic rate
Absorption from surrounding
Heat retention mechanisms

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3
Q

3 ways heat is absorbed from the environment

A

Conduction: direct contact

Convection: heat transfer from body to surrounding air

Radiation: absorption from infra-red energy

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4
Q

Heating retention mechanisms

A

Piloerection

Vasoconstriction

Behaviour responses (herding)

Shivering

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5
Q

Cooling mechanisms

A

Decrease metabolic rate

Heat in deep tissues is transferred to skin via blood flow

Evaporation

Vasodilation

Behaviour response (shade, decreased activity)

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6
Q

Normal body temp of cats and dogs

A

38.5 +/- 0.5 degrees Celsius

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7
Q

Normal body temp of cattle

A

38.5 +/- 0.5 degrees Celsius

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8
Q

Normal body temp of horses

A

38 +/- 0.5 degrees Celsius

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9
Q

True or false

Pain and extreme stress can cause a mild increase in temperature

A

True

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10
Q

Dangerous temperatures for all species

A

Hot: 43 degrees Celsius and above (rapidly dying)

Cold: 23 degrees Celsius (dying)

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11
Q

Fever (Pyrexia)

AKA physiological hyperthermia

A

Purposeful increase in body temp in response to something abnormal occurring in the body

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12
Q

Why is a fever a normal part of the body’s immune response

A

Higher temps inhibit virus and bacteria from replicating by denaturing viral and bacterial proteins

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13
Q

Why is treatment of true fever not always recommended

A

To allow the body’s natural immune response to do its job

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14
Q

True or false
Treating a fever is only indicated if the temperature is high enough or sustained long enough to pose a risk to the patients proteins

A

True

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15
Q

Proteins in the cells start to become damaged at

A

42 degrees Celsius

And irreversible damage occurs at 43 degrees Celsius

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16
Q

Treatment options for fever

A

Treat underlying problem

Non-steroidal anti-inflammatories

Passive cooling

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17
Q

Pathological hyperthermia occurs when

A

There is excessive heat generation or the body cannot cool off fast enough

AKA

The increase in temperature is not supposed to be happening and it’s causing damage

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18
Q

Intrinsic risk factors for hyperthermia

A
Obesity
Hair coat
Dehydration
Underlying issues 
Brachycephalic dogs (respiratory issues)
Genetic predisposition to drug reactions 
Exercise
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19
Q

Extrinsic risk factors for hyperthermia

A

Environmental temp approaches body temp
>80% humidity (decreases evaporation)
Over crowding/poor ventilation/transport/ capture stress
Water deprivation

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20
Q

Heat stress

Mild hyperthermia=heat exhaustion

A

Often subclinical
Signs include: lethargy, sweating, panting, decreased performance, decreased production, changes in hydration

39.1-41.5 degrees Celsius

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21
Q

Treatment of heat stress

A

Passive cooling (shade, ventilation, decrease ambient temps, fans, misting, water)

Prognosis is good if it does not progress

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22
Q

Heat stroke

Severe hyperthermia

A

Core body temp exceeds 41.5 degrees Celsius for a long period of time

Anything above 43 degrees Celsius is critical

23
Q

disseminated intravascular coagulation

A

DIC= DEATH IS COMING

24
Q

Goal when treating heat stroke

A

To decrease to 39.5 degrees within 30-45 minutes and stop there

25
Passive surface cooling
``` Cool room Shade Fans/ventilation Soak with tepid water Drink fluids ```
26
Active surface cooling (if <43 degrees)
Apply cold packs to neck, foot pads, inguinal regions and axilla (where major blood vessels are) Goal is to cool blood as it moves through the largest vessels
27
Active core cooling -for critical heat stroke
Chilled IV fluids Cool water enemas
28
Why should you not immerse a heat stroke patient into an ice bath?
It will cause vasoconstriction and shivering: things that will create more heat
29
Mu-agonist induced hyperthermia in cats
A class of opioids that can cause hyperthermia in cats
30
Signs of mu-agonist hyperthermia
Temp does not drop as expected during anesthesia Gradual increase in temp
31
Treatment of mu-agonist hyperthermia
Intervene at 41 degrees Treat by reversing the opioid
32
What is the most common reason patients will die from mu-agonist hyperthermia?
Because it is not caught early enough
33
Malignant hyperthermia
Fatal hyperthermia caused by exposure to inhalant anesthetics Most common in pigs Inherited defect in skeletal muscles metabolism triggered by exposure: causes excessive release of calcium causes prolonged muscle contraction
34
Hypothermia due to increased heat loss
Heat production is normal ``` Related to environmental conditions Cold temps Vet Low BCS neonates born in snow Small animals ```
35
Hypothermia due to lack of heat production
Can occur with severe illness or shock Brain trauma Adverse effect of anesthesia
36
Mild hypothermia
34-37 degrees Celsius Signs: Lethargy, depression, shivering, loss of suckle reflex in young
37
Moderate hypothermia
28 to 33 degrees Celsius (loose physiological warming responses at 33 degrees) Signs: altered LOC, decreased metabolism, loss of thermoregulation, decreased HR and cardiac output, decreased muscle function,
38
Severe hypothermia
<28 degrees Celsius Signs: loss of consciousness, coma, death, organ dysfunction, DIC, CNS shut down
39
Prevention for hypothermia
Proper shelter Avoid wetness in cold temps Minimize anesthesia times, warm IV fluids, monitor temps
40
Treatment for hypothermia
Passive surface methods Support vital functions Active warming methods if needed Stop warming at 37 degrees
41
Why do we want to stop warming a hypothermia patient at 37 degrees Celsius
So they don’t get hot and start to thermoregulate to cool themselves down (sweating, panting etc)
42
Passive surface warming methods
Get dry Get out of cold area Blankets Foil wrap
43
Active surface warming methods
Mild heat to surface, which transfers to core Warm packs on axilla, inguinal, neck, etc
44
Active core warming methods
Breathe in warm air Warm IV fluids Warm enema Monitor temp every 15 minutes
45
After drop phenomenon
Occurs when the patient reaches 37 degrees but heat sources are not removed Patient becomes too warm at the core, to try and cool down again blood from the periphery (which is cool) will move to the core and cause a sudden drop in temp which can affect heart and CNS
46
Re-warming shock
Occurs when there is external warming without core warming Causes vasodilation in the skin, blood moves towards the skin and away from organs so the patient goes into shock
47
Frostbite
Frozen tissue after exposure to extremes cold
48
Rick factors of frostbite
Cold, wet, windchill Neonates <48 hours of age Really old or sick If they have poor perfusion to the extremities
49
Pathology of frostbite
Ice crystals and expansion of frozen water cause permanent tissue damage Lack of blood flow increases risk of necrosis Increased risk of bacterial infection upon thawing
50
Necrosis
Dying or dead cells or tissue
51
Signs of frostbite
Skin is hard, cold, pale, loss of sensation Sounds solid when tapped Red(not as severe) or black (extreme)
52
Treatment of frostbite
``` Take out of cold Slow passive surface rewarming Protect damaged areas from patient (might chew on them) Support organs Anti inflammatories ```
53
What should you never do with frostbite
Never rub Never immerse in hot water Never thaw and then refreeze