Theme 3 - ECG Flashcards

1
Q

what type of junctions allow electrical coupling of cells?

A

gap junctions

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2
Q

what is the most numerous cell type in the heart?

A

contractile cells

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3
Q

what is the function of the fibrous skeleton?

A

electrically isolates the A from V so they don’t contract at the same time

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4
Q

how are cardiomyocytes linked?

A

by gap junctions at intercalated disks

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5
Q

what speed to A and V myocytes propagate at?

A

0.3-0.5m/s

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6
Q

what speed do modified myocytes in the purkinje fibres propogate at?

A

5m/s

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7
Q

what three things are intercalated disks made up of?

A

fascia adherens, gap junctions and desmosomes

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8
Q

what allows synchronous conduction of impulses from SA to AV node?

A

internodal bundles

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9
Q

how many specialised bundles are there in the atria and what do they contain?

A

four bundles that contain purkinje like cells

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10
Q

where is Bachman’s bundle found?

A

inner wall of the LA

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11
Q

what is the speed of transmission at the AV node and why?

A

slow to allow atria to empty blood into the ventricles so that they are full at systole

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12
Q

what sequence does depolarisation pass in the ventricular wall?

A

septum then apex then AV groove

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13
Q

what does an ECG show?

A

individual currents of cardiomyocytes that work together in a functional syncytium

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14
Q

what is an ECG lead and where is it connected?

A

a configuration of electrodes - positive (ankle or leg), negative (wrist) and sometimes a ground electrode

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15
Q

how many electrodes does a 12 lead ECG use?

A

10

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16
Q

what type of leads make up 12 standard lead ECG?

A

3 bipolar leads (frontal plane), 3 augmented leads (coronal plane) and 6 pre cordial leads (thorax near the heart)

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17
Q

what does the P wave represent?

A

depolarisation in atria from SA node

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18
Q

what does the PR segment represent?

A

SA to AV delat to allow ventricular filling

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19
Q

what does QRS represent?

A

movement of electrical activity through the ventricles (both ventricles need to be simultaneously depolarisation)

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20
Q

what does ST represent?

A

beginning of ventricular repol (should be flat)

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21
Q

what is the T wave?

A

ventricular repolarisaton

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22
Q

what are escape beats and what do they appear as on ECG?

A

when ventricles dont get a signal so signals itself and the myocardium beats ineffectively (misshaped QRS)

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23
Q

what is a possible cause of wide QRS?

A

problems in the purkinje system

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24
Q

what is a large/deep Q wave a sign of?

A

dead tissue - old MI

25
what is sinus tachycardia?
fast heart rate due to SA node beating too quickly
26
how is atrial rate calculated?
P to P wave
27
how is ventricular rate calculated?
RR interval (QRS to QRS)
28
how can rate be calculated using boxes on ECG?
this is ventricular rate so counting how many big boxes occur between two R waves then 300/big boxes
29
what are the names of pre cordial leads and what plane do they record from?
transverse - V1-V6
30
what is the main NT in psym control of the heart and what receptors does it work on?
vagal drive from Ash which stimulates muscarinic receptors
31
what drug is a muscarinic antagonist and therefore increases heart rate?
atropine
32
is heart vasculature innervated by the psym system?
no
33
what type of nerves does symp autonomic control work on?
stellate nerves
34
what is the effect of a beta agonist on heart rate and what receptors does it work on?
increases rate via b2 adrenergic receptors
35
what is the effect of beta blockers on heart rate and what receptors does it block?
decreases rate via adrenergic receptors
36
what is AV block?
a delay due to failure of atrial signal to stimulate ventricles
37
what three main things is AV block caused by
- ischaemia of AV node or AV bundles - POOR PERFUSION - compression of AV bundle by scar/calcified tissue - inflammation of AV node or bundle
38
what are the symptoms of AV block?
palpitations, hypotension like - dizziness, malaise, syncope and risk of sudden death - patient can be asymptomatic
39
How is 1st degree heart block characterised on ECG?
increased PR interval (more than 5 small boxes) but all Ps are followed by QRS
40
what is first degree heart block usually due to?
delayed AV node transmission
41
how is Mobitz type I 2nd degree heart block characterised on ECG?
- some P waves are blocked and not followed by QRS | - some missing QRS (P wave gets longer until QRS fails to follow it - wenckebach)
42
what is the cause of mobitz type I 2nd degree heart block and what treatment is usually given?
AV node damage and usually no treatment
43
what are the two types of second degree heart block?
mobitz type I and II
44
how is Mobitz type II 2nd degree heart block characterised on ECG?
consistent PR interval and some P waves blocked/not followed by QRS
45
what causes Mobitz type II second degree heart block?
problems with ventricles or bundle of his
46
is mobitz type II 2nd degree heart block high risk and what is the treatment?
yes it is high risk and treated by implanting a pacemaker
47
how is 3rd degree heart block characterised on ECG?
distance of P to QRS is unrelated and PR interval varies radically
48
what causes 3rd degree heart block?
AV node isn't conducting so ventricles do their own thing (ectopic complex)
49
what are they features of ventricular rate and atrial beats in 3rd degree heart block?
ventricular rate and atrial beats are consistent
50
what is a premature beat?
early neats that can be triggered by irritable tissue
51
what are escape beats and what triggers them?
late beats when the atrial signal is delayed or prevented that are triggered by natural rhythmicity of non atrial tissue
52
what is the purpose of escape beats?
an extra beat to escape arrhythmia
53
How is a premature ventricular contraction characterised on ECG?
Very wide QRS and no T wave, no S wave | - negative dip where T wave should be
54
where are premature ventricular contractions often triggered and what does this result in?
in the middle of the myocardium (non purkinje) | - results in electrical unsynchrony, delayed and insufficient conduction
55
what is atrial fibrillation and what happens?
disorganised electrical activity in the atria | - ventricular rate is faster and irregular as many signals reach the AV node but only some are transmitted
56
how is atrial fibrillation characterised on ECG?
No P wave - flat or wiggly line instead
57
what can AF lead to?
thrombus formation in the atrium due to slow flow of blood | - increased risk of stroke
58
what is respiratory sinus arrhythmia?
heart beat is slight fasting during inspiration and slightly slower during expiration
59
what is respiratory sinus arrhythmia a sign of and who is it most commonly seen in?
a healthy heart mostly seen in children and athletes