(!) Supraventricular tachycardia Flashcards
Define Supraventricular tachycardia
group of heart electrical abnormalities-defines as having normal sinus tachycardia with abnormal (often absent) p waves
AVRT-wolff parkinson white-via bundle of his
this focus on AV NODAL re-entry tachycardia (AVNRT
Aetiology and risk factors of Supraventricular tachycardia
In AV node, 2 pathways-fast and slow that connect.Normally goes via fast, and fast stops slow. Fast repolarises slow, so can have premature signal-goes down slow pathway, and arrives at end as fast repolarises-> goes in circle-> activating ventricle everytime and escaping control
most common are focal atrial tachy-p waves present
Or AVNRT-no p waves
risk factors:
substance missuse (alcohol, cocaine)
Digoxin toxicity
Caffeine
Epidiemology of Supraventricular tachycardia
Focal atrial tachy is relatively uncommon, but represent 10% of Supraventricular tachy
Signs and Sx of Supraventricular tachycardia
Mainly based on ECG
Identify prior Hx of cardiac problems
or substance missuse
Sx-SOB, palpitations, dizziness/syncope, fatigue, nausea
ECG-AVNRT-sinus tachycardia, regular, but with no visible p waves
Focal atrial tachy-p waves visible, rate under 250bpm
Short duration with abrupt stop and start
P wave morphology weird-esp in v1 or aVL
response to vagal manoeuvres and adenosine are big diagnostic clues
Investigations of Supraventricular tachycardia
ECG-AVNRT-sinus tachycardia, regular, but with no visible p waves. or p waves immediately before/after qrs. 140-280bpm
Focal atrial tachy-p waves visible, rate under 250bpm
Short duration with abrupt stop and start
P wave morphology weird-esp in v1 or aVL
rates should vary with pain, respirations and changes in ANS tone
response to vagal manoeuvres and adenosine are big diagnostic clues-
AVNRT will abruptly stop with vagal manoeuvres (only one to respond like that)
digoxin levels-can be high
Hypokalaemia can be cause
Management of Supraventricular tachycardia
Differentiating between AVNRT and Sinus tachy (or A flutter)-AVNRT should stop abruptly after vagal manoeuvres (1st line) or use of adenosine (warn patient they’ll feel awful)
focal atrial tacky-respond to removal of causing agent (cocaine, alcohol, etc) but resistant to shock or drugs
Complications of Supraventricular tachycardia
Congestive heart failure can be a consquence
resistance to therapy in longer term
cardiomyopathy
tachycardia induced ischemia/MI
Prognosis of Supraventricular tachycardia
AVNRT-if not caused by structural defect-good-often stopped by vagal manoeuvres or adenosine
Focal AT-eventually always controlled but can take some time and combination of drugs