Sports (2008-2019) Flashcards
- 4 contraindications to an HTO (2012)
- JAAOS 2005 - High Tibial Osteotomy
- Disease of the contra-lateral compartment
- Absent contra-lateral meniscus
- Symptomatic patellofemoral disease
- Inflammatory arthritis
- Severe angular malalignment
- Non-concordant pain pattern
- Older physiologic age (better suited to TKA)
- List 4 advantages of doing a medial opening wedge HTO over a lateral closing wedge. (2016)
JAAOS 2005 - HTO/JAAOS 2011
- Easy to control correction (can dial it in)
- Less extensive surgical dissection/no disruption to proximal tib/fib joint
- Less proximity to peroneal nerve
- Can be combined with PF procedures
- No loss of lateral bone stock
- List 5 features of traumatic proximal tibia fibula dislocation. (2013)
JAAOS 2003 - Instability of the Proximal Tibiofibular Joint
- Pain over lateral knee, radiates up biceps femoris
- Prominence of fibular head
- Pain aggravated by ankle ROM
- Instability to weightbear
- Peroneal nerve symptoms
- Posterolateral corner instability
- LCL instability
- Pain and loss of knee ROM
- In regards to an acute, traumatic proximal tibia fibula dislocation (2013):
- Explain the mechanism of injury, including position of limb and knee position. (2 marks)
- Two features seen on examination
- Explain your reduction technique (2 marks)
- Explain the mechanism of injury, including position of limb and knee position. (2 marks)
- Hyperflexed knee, plantarflexed and internally rotated foot
- Two features seen on examination
- Prominence of fibular head and lateral knee
- Instability of posterolateral corner
- Explain your reduction technique (2 marks)
- Knee at 80-110o of flexion (relax biceps femoris)
- Ankle dorsiflexed, foot everted and externally rotated (relax peroneals)
- Pressure to fibular head in appropriate direction
- List 4 things that can cause loss of extension after ACL reconstruction? (2012, 2016)
JAAOS 1999 - Loss of Extension after Reconstruction of the ACL
- Cyclops lesion
- Capsulitis leading to arthrofibrosis
- Post-operative immobilization
- Lack of post-operative rehabilitation
- Non-anatomic graft placement
- Infection
- Trauma - bucket handle of meniscal tear
- What are 4 surgical techniques for ACL reconstruction in an 11 year old female athlete? (2012, 2013)
- Iliotibial band extra-physeal reconstruction (modified McIntosh)
- IT band remains attached to Gerdy’s tubercle, passes over femoral condyle and under intermeniscal ligament to suture under flaps of anterior tibial periosteum
- All Epiphyseal Reconstruction
- Anderson technique: 4 strand hamstring all epiphyseal suspensory technique on femur, epiphyseal tibial tunnel exiting above physis but interference screw inserted below physis to tether graft
- Ganley technique - 4 string hamstring graft with epiphyseal femoral interference screw and epiphyseal vertical tibial interference screw
- Cordasco-Green - epiphyseal tunnels in femur and tibia; suspensory fixation for both
- Partial trans-physeal
- Epiphyseal femoral tunnel and trans-physeal tibial tunnel
- Trans-physeal reconstruction
- I would not answer this on an exam for an 11yo if I could avoid it; if I use it I would specify partial transphyseal with an epiphyseal femoral tunnel and a vertical, central, small diameter tibial tunnel
- When performing the Thesally physical exam on the knee, what are 3 conditions that can give you a false positive. (2011, 2013)
- OA
- Osteochondral Injury
- Collateral Ligament Injury
- Posterolateral Corner Injury
- Associated ACL Injury
- Tibial Plateau #
- Loose body
- What are 6 risk factors for recurrent patellar dislocation? (2014, 2015)
JAAOS 1997 - PF Instability
- Previous patellar instability
- Increased ligamentous laxity
- Patella alta
- Trochlear dysplasia/Hypoplastic lateral condyle
- Patellar dysplasia
- Malalignment (increased femoral anteversion, genu valgum, increase ER of tibia)
- Increased Q Angle
- VMO Atrophy
- Female gender (AAOS COR)
- Not a risk factor for initial dislocation, but risk factor for recurrence
- List 3 prognostic factors for healing of OCD of the knee. (2015)
- Status of physis
- Size of lesion (>15mm)
- Chronicity of lesion
- Integrity of cartilage
- Mechanical symptoms/loose fragment
- Atypical location (patella, lateral femoral condyle)
- Patient with a PCL and Posterolateral corner injury:
- Increased external rotation of tibia with knee at 30 degrees but not at 90 degrees
- Increased external rotation of tibia with knee at 30 degrees and at 90 degrees
- Increased laxity with varus stress
- Pivot shift positive
ANSWER: B (increase ER or Dial test at 30 AND 90 degrees)
2013, 2015
- JAAOS 2016 - Management of Posterior Cruciate Ligament Injuries
- A positive result occurring only at 30o of knee flexion indicates a PLC injury, and 90o of knee flexion indicates a combined PCL and PLC injury.
- Which of the following are not part of the posterolateral corner
- LCL
- PCL
- Popliteofibular ligament
- Popliteus
ANSWER: B (not PCL)
2013, 2014
Covey (JBJS 2001) Injuries of the posterolateral corner of the knee
- Static:
- LCL
- Popliteus Tendon
- Popliteofibular ligament
- Lateral Capsule
- Variable –> arcuate ligament, fabellofibular ligament
- Dynamic Structures:
- Biceps femoris
- Popliteus muscles
- IT band
- Lateral head of gastrocs
- What vessel provides blood supply to the PCL?
- Inferior geniculate
- Superior geniculate
- Middle geniculate
ANSWER: C (middle geniculate)
2011,2016
COR2
- Late complaint after PCL insufficient Knee?
- Medial compartment OA
- Instability
- Lateral Compartment OA
ANSWER: A (AnteroMEDIAL)
- 2009
- Old Evidence:
- “The natural history of the PCL-deficient knee leads to increased contact pressures in both the medial and PF compartments”
- ACL - posteromedial
- PCL – anteromedial
- Both medial compartment
14.All of the following are risk factors for ACL tear except:
- Female
- Cavovarus
- Increased Q-angle
- Increased BMI
ANSWER: B (Not cavovarus)
- 2016
- Really not the greatest evidence for BMI available
- JAAOS 2000 - Noncontact ACL Injuries: Risk Factors and Prevention Strategies
- Environmental:
- Equipment
- Shoe-surface interaction
- Anatomic:
- Knee angle
- Hip angle
- Laxity
- Notch size
- Hormonal
- Biomechanical:
- Muscular strength
- Skill level
- Neuromuscular control
- Body movement
- Environmental:
- When treating an infection post-ACL reconstruction, all are true EXCEPT:
- Blood cultures are usually not positive
- Every reasonable effort should be made to retain the graft and hardware
- Acute infections have the same prevalence as delayed infections
- Arthrocentesis is the diagnostic modality of choice
ANSWER: C
2016
- JAAOS 2013 - Management of Septic Arthritis Following ACL Reconstruction
- “Blood cultures, which are often ordered at presentation, are not reliable markers of infection because they are often negative or prone to contamination from skin flora”
- “All reasonable attempts are initially made to preserve the reconstructed graft and associated hardware”
- “Typically presents either acutely (<2 weeks from surgery) or subacutely (2 weeks to 2 months). Late presentation is relatively infrequent”
- “Arthrocentesis remains the standard of care for diagnosis of septic arthritis following ACL reconstruction”
- Where is the posterolateral bundle of the ACL tightest?
- Extension
- 30 degrees flexion
- 90 degrees flexion
- Same in all degrees flexion/extension
ANSWER: A
- 2014
- Amis (JBJS Br 1991) Functional anatomy of the ACL
- Anterior bundles tight in FLEXION
- Posterior bundles tight in EXTENSION
*
- What is true regarding ACL tunnel Placement
- Tibial tunnel is most important
- If femoral tunnel is too posterior, graft lengthens in flexion
- If femoral tunnel too anterior, graft lengthens in flexion
- Over the top femoral placement of graft does not change in extension
ANSWER: C
- 2008, 2012
- Depends on whether tunnel is anterior in femur or tibia
- Etiologic Factors That Lead to Failure After Primary Anterior Cruciate Ligament Surgery. Jan 2017 Clin Sports Med
- TRICK: femur rotates during flexion, what is posterior becomes anterior and vis versa
- Chronic ACL all associated except:
- Increased risk of OA
- ACL recon has not been shown to decrease OA
- More chance of damaging cartilage and meniscus
- More likely to damage lateral as opposed to medial meniscus
ANSWER: D (chronic ACL = posteroMEDIAL OA)
- 2013 (two variants)
- Cipolla M (KSSTA 1995) Different patterns of meniscal tears in acute Acl ruptures and in chronic ACL deficient knees
- Retrospective study of 1103 reconstructions
- “Acute injuries show a higher rate of lateral meniscus tears, chronic laxities are very frequently associated with severe medial meniscal lesions”
- Gillquist J (Sports Med 1999) ACL reconstruction and the long-term incidence of gonarthrosis
- 10-20% incidence of arthrosis
- 10x higher than age matched controls
- Menisectomy doubles risk (50-70% incidence at 15-20 years)
- Not necessarily symptomatic
- Lohmander LS (AJSM 2007) The long-term consequence of ACL and meniscus injuries: osteoarthritis
- “there is lack of evidence to support a protective role of repair or reconstructive surgery of the ACL or meniscus against osteoarthritis development”
- 50% of patients with ACL/meniscus injuries have OA at 10-20 years
- Oiestad BE (AJSM 2009) Knee OA after ACL injury: a systematic review
- Review of 7 prospective and 24 retrospective
- 0-13% incidence of OA for ISOLATED ACL injury at at least 10 years
- Combined injury 21-48%
- What is not associated with a poor prognosis after ACL injury?
- Hearing a pop at time of injury
- Smoking
- Gain 15lbs in first year
- Wearing a brace for 1 year post-op
ANSWER: D
- 2010
- OKU 9:
- Independent poor predictors after ACL reconstruction:
- Smoking
- Recollection of hearing a pop at injury
- Weight gain of 15lbs
- No change in educational level since injury
- Independent poor predictors after ACL reconstruction:
- Spindler K (JBJS 2005) Clinical outcome at a minimum of five years after reconstruction of the ACL
- Independent predictors of a worse outcome…..included the patient’s recollection of hearing or feeling a pop, a weight gain of >15lbs, and no change in educational level since surgery
- What is true regarding pediatric ACL reconstruction:
- Transphyseal tibial tunnel is an option
- Bone-patellar-tendon-bone graft is a good option
- Screw placement across the physis is acceptable
- Non-operative treatment has good outcomes
ANSWER: A
2015
JAAOS - ACL in the Skeletally Immature
- “Conservative management of skeletally immature patients with complete ACL injuries has been less successful. The high non-compliance rate is associated with secondary damage of the menisci and/or articular cartilage, and a large percentage of patients eventually require surgical reconstruction”
2018 IOC consensus on pediatric ACL:
- OK to go transphyseal BUT need to be central, vertical and no hardware crossing physis
- Non-operative
- Kocher found that partial tears-1/3 of them required surgery for persistent instability
- 50% will drop out of sports b/c of instability
- Increased rate of meniscal and chondral damage
- Hamstrings tendon grafts in ACL reconstruction, all are true except:
- Previous MCL injury is a contra-indication to using hamstring tendon
- Ligamentous laxity is a contraindication to using hamstring tendon
- Sprinter is a contraindication to using hamstring tendon
- Previous hamstring injury is a contra-indication to using hamstring tendon
ANSWER: A
2008
JAAOS 2005 - Graft Selection in ACL Reconstruction
- Contra-indications to hamstring autograft:
- Generalized ligamentous laxity
- Competitive sprinters
- Previous hamstring injury
- Acute MCL
- In performing an opening wedge HTO, all of the following are true except:
- In an ACL deficient knee, placing the bone wedge posteromedially will decrease the tibial slope and decrease anterior translation
- In a PCL deficient knee, placing the bone wedge anteromedially will increase the tibial slope and increase posterior translation
- Placing the bone wedge direct medial will not affect the slope
- Smaller anterior gap with a larger posterior gap will preserve the native tibial slope
ANSWER: B (best answer) but C also not true
2015
Charles: C is also NOT true
- JAAOS 2011 – Role of the HTO in the varus knee
- “The tibia has a triangular shape and to preserve the anatomic slope, the anterior opening shoulder be smaller than the posterior medial opening.
- My understanding is that you are opening more lateral posteriorly (base of triangle) and therefore are opening LESS central posterior then central anterior.
Increased tibial slope will DECREASE posterior translation
JAAOS 2011 - Role of the HTO in the varus knee
- ACL deficiency –> decrease tibial slope
- PCL deficiency –> increase tibial slope
- When doing a proximal tibial osteotomy for a varus knee, what is the difference between doing a fibular osteotomy and a partial resection of the fibular head
- Increased peroneal nerve injury with osteotomy
- Increased non union
- Difficult to do the tibial osteotomy
- Increased risk of popiteal artery injury
ANSWER: A (old answer C)
2008
-less chance of damage to CPN: at tib/fib joint (zone 1) or at junction of middle and distal thirds of fibula (zone 4)
- JAAOS 2005 - HTO
- Incidence of peroneal nerve palsy after lateral closing wedge 0-20%
- Osteotomy in vicinity of the fibular head associated with higher incidence of peroneal nerve palsy than fibular osteotomy >15cm distal to head