Shock Flashcards
(30 cards)
Shock stages
-compensatory
-progressive
-irreversible
Compensatory Shock stage
-multi system response to decreased tissue perfusion
-the body can compensate for changes
-if the cause is corrected, then minimal residual effects
-pt is experiencing fight/flight = high HR, normal BP
-beta blockers might prevent high heart rate
Progressive Shock stage
-when compensator mechanisms fail
-cardiac profoundly affected, decreased BP and CO, increased capillary permeability, leaky vessels
-Goals: prevent MODS (e.g., add pressors
Irreversible Shock stage
-profound hypotension & hypoxemia
-failure of organs r/t waste products (lactate, urea)
-recovery unlikely
early signs
-pallor
-tachypnea
-confusion
-tachycardia
late signs
-cold, moist skin
-weak, thready pulse
-anuria
-hypotension
-metabilic acidosis
Vasopressors
increase cardiac output
-multiple side effects
-different pressors for different types of shock
Hypovolemic Shock types
-External: fluid losses (fluid lost from the body completely)
-external causes: stabbing, hemorrhage, diarrhea, vomiting
-Internal: fluid shifts (third spacing)
-internal causes: ascites
Hypovolemic Shock causes
-hemorrhage
-GI loss
-Fistula drainage
-DI
-diuresis
-burns
-ascites
-internal bleeding
Hypovolemic Shock presentation
-tachypnea
-hypotension
-tachycardia
-peripheral hypoperfusion
-decreased urine output
-mental status changes
Hypovolemic Shock patho
-decreased venous return to heart leads to decreased CO
Hypovolemic Shock treatment
-Rapid fluid replacement
-correct the cause
-safety of blood transfusion
-rapid infuser
rapid fluid replacement in Hypovolemic Shock
-2 large bore IV lines (14-16g), IO, or central venous catheter
-restore fluids (Blood, IVF)
Cardiogenic Shock types
-coronary (more common, most MI patients)
-noncoronary (conditions that stress the myocardium)
Cardiogenic Shock presentation
-tachypnea
-crackles
-hypotension
-tachycardia
-peripheral hypoperfusion
-decreased urine output
-mental status changes
Cardiogenic Shock causes
-MI (number one cause)
-cardiomyopathy
-cardiac tamponade
-blunt cardiac injury
-hypoxemia
-acidosis
-tension pneumothorax
Cardiogenic Shock patho
decreased cardiac output
-impaired tissue perfusion
-weakens heart and ability to pump
Cardiogenic Shock treatment
GOAL: correct underlying cause
-cardiac Cath/fibrinolytics/CABG/IABP/VAD
-2-6L NC, SpO2 goal: 95%
-morphine for pain control
-use A-line or PA Cath for hemodynamic monitoring
-Labs: BNP, cardiac enzymes, lactate
-fluids: monitor for overload
Cardiogenic Shock medications
-drug of choice: Dobutamine (inotropic) to increase contractibility
-nitrates
-dopamine: increase HR & contractility
-pressors
-antiarrhythmic meds
Distributive shock
-intravascular volume pools in peripheral blood vessels
-leads to relative hypovolemia b/c not enough blood returns to the heart –> inadequate tissue perfusion
-septic, neurogenic, anaphylactic
Septic shock
-persistent hypotension despite adequate fluid resuscitation
-requires vasopressors
-inadequate perfusion resulting in tissue hypoxia
Septic shock major pathophysiologic effects
-vasodilation
-misdistribution of blood flow
-myocardial depression/decreased cardiac output
Septic shock treatment
-aggressive fluid resuscitation (30mg/kg)
-blood cultures before antibiotics
-Vasopressors for BP (norepinephrine is the 1st line)
-monitor temp, glucose, nutritional therapy
-stress ulcer prophylaxis
Neurogenic shock manifestations
-hypotension & bradycardia
-difficulty regulation body temp