Session 12: nerve injuries in l/ limb and gait abnormalities Flashcards

1
Q

common l/limb examples of nerve compression

A

meralgia paraesthesia - compression of lateral cutaneous nerve of theigh in inguinal region

tarsal tunnel syndrome - compression of posterior tibial nerve at the ankle

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2
Q

classification of nerve injuries according to Herbert J Seddon

A

Neuropraxia: transient interruption, recovery, no Wallerian degeneration, caused by eg biochem. lesion from concussion/compression/blunt blows/shock-like injuries, temporary loss of function -> reversible w/in hours - months, greater involvement of motor than sensory (autonomic is retained)

Axonotmesis: severe crush/contusion > neuropraxia, loss of continuity of myelin sheath and axon, epi and perineureum are retained, Wallerian degeneration, equal loss in both motor and sensory, element of retrograde/proximal degeneration of axon, axonal regen. occurs down intact neural tubes, 1-3mm per day

Neurotmesis: severe contusion/stretch/lacerations, epi and perineureum damaged, no gross loss instead there is internal disruption, loss of motor, sensory and autonomic, axonal regen. causes a neuroma to form

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3
Q

neuropraxia

A

transient interruption, recovery, no Wallerian degeneration, caused by eg biochem. lesion from concussion/compression/blunt blows/shock-like injuries, temporary loss of function -> reversible w/in hours - months, greater involvement of motor than sensory (autonomic is retained)

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4
Q

axonotmesis

A

severe crush/contusion > neuropraxia, loss of continuity of myelin sheath and axon, epi and perineureum are retained, Wallerian degeneration, equal loss in both motor and sensory, element of retrograde/proximal degeneration of axon, axonal regen. occurs down intact neural tubes, 1-3mm per day

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5
Q

neurotmesis

A

severe contusion/stretch/lacerations, epi and perineureum damaged, no gross loss instead there is internal disruption, loss of motor, sensory and autonomic, axonal regen. causes a neuroma to form

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6
Q

what stages do these represent?

A

1 normal

2 Neuropraxia: transient interruption, recovery, no Wallerian degeneration, caused by eg biochem. lesion from concussion/compression/blunt blows/shock-like injuries, temporary loss of function -> reversible w/in hours - months, greater involvement of motor than sensory (autonomic is retained)

3 Axonotmesis: severe crush/contusion > neuropraxia, loss of continuity of myelin sheath and axon, epi and perineureum are retained, Wallerian degeneration, equal loss in both motor and sensory, element of retrograde/proximal degeneration of axon, axonal regen. occurs down intact neural tubes, 1-3mm per day

4 Neurotmesis: severe contusion/stretch/lacerations, epi and perineureum damaged, no gross loss instead there is internal disruption, loss of motor, sensory and autonomic, axonal regen. causes a neuroma to form

5 5th degree lesion

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7
Q

sunderland system

A

1st degree/Seddon’s neuropraxia: transient interruption, recovery, no Wallerian degeneration, caused by eg biochem. lesion from concussion/compression/blunt blows/shock-like injuries, temporary loss of function -> reversible w/in hours - months, greater involvement of motor than sensory (autonomic is retained)

2nd degree/Seddon’s axonotmesis: severe crush/contusion > neuropraxia, loss of continuity of myelin sheath and axon, epi and perineureum are retained, Wallerian degeneration, equal loss in both motor and sensory, element of retrograde/proximal degeneration of axon, axonal regen. occurs down intact neural tubes, 1-3mm per day

3rd degree: endoneureum disrupted, epi and perineureum intact, recovery can range from poor -> complete, depends on degree of intrafascicular fibrosis, nerve may not appear seriously on gross inspection

4th degree/Seddon’s neurotmesis: severe contusion/stretch/lacerations, epi and perineureum damaged, no gross loss instead there is internal disruption, loss of motor, sensory and autonomic, axonal regen. causes a neuroma to form

5th degree: complete transection w/ loss of continuity

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8
Q

what is lesion of common peroneal nerve associated w/?

A

foot drop -> high steppage gait

loss of sensation of lateral leg and dorsum of foot

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9
Q

what may stiffness/poor mobility/pain in a joint result in?

A

antalgic gait: walk in a manner that reduces the painful sensations

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10
Q

what are the clinical presentations of nerve injuries?

A

bruising/crushing/entrapment -> compression (eg piriformis syndrome, meralgia parasthetica, below knee POP- Paris of plaster, prolapsed/herniated IV disc)

stretching/traction (dislocated hip, THR surgery- total hip replacement, leg lengthening)

anoxia- complete loss of oxygen, ie extreme hypoxia (dead leg, saturday night palsy, compartment syndrome)

division/transection (penetrating wounds, laceration by bone fragments, surgery)

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11
Q

deep peroneal nerve damage results

A

motor loss of tibialis posterior, EDL, EHL, fibialis tertius

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12
Q

piriformis syndrome

A

bruising/entrapment/crushing and conpression

piriformis muscle irritates sciatic nerve

pain in the buttocks

referring pain along course of sciatic nerve

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