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Pathology > SEQUELAE OF PULPAL DISEASE > Flashcards

SEQUELAE OF PULPAL DISEASE Flashcards

1
Q

PULPITIS (4)

A

Inflammation of the pulpal tissue
Reversible vs irreversible
Acute vs chronic
Symptomatic vs asymptomatic

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2
Q

MECHANISM OF ACTION:
INFLAMMATION OF THE PULP (4)

A

 Limited blood supply
 No collateral support
 D e s t ruct ive
 E x p ansi le p roces s

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3
Q

E x p ansi le p roces s
(3)

A

▪ Blood vessel dilation
▪ Leakage of fluid into surrounding tissue
▪ Migration of cells

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4
Q

PERIAPICAL LESIONS
Presence of (2) pulpitis

A

opened or closed

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5
Q

PERIAPICAL LESIONS
— of involved microorganism

A

Virulence

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6
Q

PERIAPICAL LESIONS
Extent of — of dentinal tubules

A

sclerosis

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7
Q

PERIAPICAL LESIONS
— of host immune response

A

Competency

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8
Q

PERIAPICAL GRANULOMA

A

mass of chronically inflamed granulation tissue

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9
Q

PERIAPICAL GRANULOMA
location

A

apex of non vital tooth

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10
Q

PERIAPICAL GRANULOMA
most are

A

asymptomatic

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11
Q

PERIAPICAL GRANULOMA
misnomer

A

not true granulomatous inflammation

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12
Q

PERIAPICAL GRANULOMA
may develop as the

A

initial periapical pathosis or arise after an initial periapical abscess

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13
Q

PERIAPICAL GRANULOMA
may transform into (2)

A

periapical cyst
abscess

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14
Q

DEFINITION OF A CYST

A

pathologic cavity located in soft tissue or bone lined by epithelium

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15
Q

APICAL PERIODONTAL CYST
(PERIAPICAL CYST)
an — cyst

A

inflammatory

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16
Q

APICAL PERIODONTAL CYST
(PERIAPICAL CYST)

A

asymptomatic, slow growing lesion associated with the root apex of a non-vital tooth

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17
Q

APICAL PERIODONTAL CYST
(PERIAPICAL CYST)
— possible

A

external root resorption

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18
Q

PERIAPICAL ABSCESS
accumulation of

A

acute inflammatory cells at the apex of a nonvital tooth

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19
Q

PERIAPICAL ABSCESS
may arise as the initial

A

periapical pathosis or as an acute exacerbation of chronic periapical lesion (phoenix abscess)

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20
Q

PERIAPICAL ABSCESS
generally,

A

symptomatic, but may be asymptomatic if there is a lack of accumulation of purulent material due to a chronic path of drainage

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21
Q

DRAINAGE PATHWAYS OF ACUTE
PERIAPICAL INFECTIONS (5)

A

1 . S ur f ace o f t h e g i ngi va ( p arulis )
2 . P al ate ( p al atal a b s ces s)
3 . M ax i llar y s i nus
4 . S o f t t i s s ue s p aces ( c e ll ulit is)
5 . F l oor o f m ou t h ( Lu dw ig A n g ina)

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22
Q

PARULIS (GUM BOIL) (2)

A

acute periapical inflammation
purulent material perforates through bone, periosteum, soft tissue, epithelium and drains through intraoral sinus

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23
Q

a parulis is the

A

intraoral opening of a sinus tract

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24
Q

parulis consists of

A

a mass of inflamed granulation tissue with an epithelialized sinus tract

25
Q

ABSCESS

A

a localized collection of pus that has accumulated (consolidated) in a tissue cavity, producing fluctulance

26
Q

CUTANEOUS SINUS TRACT

A

a dental abscess that drains extraorally, through the overlying skin

27
Q

SINUS TRACT (2)

A

from within to the exterior
parulis is the end of a sinus tract

28
Q

FISTULOUS TRACT (1)

A

connects two anatomic cavities

29
Q

connects two anatomic cavities (2)

A

oroantral, oronasal fistula
tracheo-esophageal fistula

30
Q

CELLULITIS

A

 D i f f us e s p read o f a n a c u te
i n flamm ator y p roces s
t h rough t h e f a s c ial p l anes
o f s o f t t i s s ue p roduc ing
e r y t hema, e d ema, wa r m t h,
a n d p a i n
 A b s c ess u n abl e to e s t ablis h
d r ainage i n to t h e o r al c av it y
o r o n to t h e s u r f ace o f s k i n
( s i nus t r ac t )

31
Q

LUDWIG’S ANGINA

A

A n a g g ress ive, r a p idly
s p read ing c e l lulit is
i nvolvi ng m u lt iple a n a tomic
s p aces – t h e s u bmental ,
s ub lingual a n d
s u b mandib ular s p ac es

32
Q

LUDWIG’S ANGINA
produces

A

m a s sive s we ll ing
o f n e c k t h at m ay ex tend
c l ose to c l av icl es a n d c a u se
a i r way o b s truc t ion

33
Q

CAVERNOUS SINUS THROMBOSIS

A

Va l veless ve nous s y s tem m ay a l l ow r et rograde s p read o f
i nfec t ion f rom m i d dle t h i rd o f t h e f a c e

34
Q

Formatio n o f a — w i t hi n t h e c avernous s i nus , a m a j or
d ural ve nous s i nus , a l i fe - th reatening i n fect ion

A

blood clot

35
Q

M ay b e a s s oc iated w i t h s p read o f i n fec tion f rom

A

m ax i llar y
te et h

36
Q

OSTEOMYELITIS

A

B a c terial i nfec t ion o f b o ne

37
Q

OSTEOMYELITIS
B a c terial i nfec t ion o f b o ne
(4)

A

▪ Odontogenic infection
▪ Traumatic fracture of bone
▪ NUG, NOMA
- Developing countries

38
Q

OSTEOMYELITIS
P re dis pos iti on
(3)

A

▪ Chronic systemic diseases
▪ Immunodeficiency
▪ Decreased vascularity of bone

39
Q

Ac u te o s te o mye l i t i s
(2)

A

▪ Spread through medullar y spaces
▪ Minimal tissue reaction

40
Q

C h ro n i c o s te o mye l i t i s
(3)

A

▪ Prominent tissue reaction
▪ Granulation tissue
▪ Fibrosis

41
Q

SEQUESTRUM

A

f r agment o f n e c rot ic b o ne s e p arated f rom
a d j ac ent v i t al b o ne t h at u s u ally u nd ergoes s p ont aneous
ex fol iati on

42
Q

INVOLUCRUM

A

nonvital b o ne, e n c as ed by v i t al b o ne

43
Q

PROLIFERATIVE PERIOSTITIS
form of

A

chronic osteomyelitis

44
Q

osteomyelitis with proliferative periostitis,

A

garre osteomyelitis

45
Q

PROLIFERATIVE PERIOSTITIS
a periosteal rxn in which layers of

A

reactive vital bone are formed, producing cortical expansion

46
Q

PROLIFERATIVE PERIOSTITIS
seen most frequently in

A

children and young adults in the mandibular molar and premolar area involving the lower bored or buccal cortex

47
Q

CONDENSING OSTEITIS
(FOCAL CHRONIC
SCLEROSING OSTEOMYELITIS)

A

localized area of bone sclerosis associated with the apices of teeth with pulpal disease

48
Q

CONDENSING OSTEITIS
(FOCAL CHRONIC
SCLEROSING OSTEOMYELITIS)
seen most frequently in

A

children and young adults involving the mandibular molars and premolars

49
Q

A c t inomyces s p e cies
(2)

A

▪ Actinomyces israelii
▪ Actinomyces viscosus

50
Q

F i lam entous b a c teria
(3)

A

▪ Branching
▪ Gram positive
▪ Anaerobic

51
Q

ACTINOMYCOSIS – “RAY FUNGUS” (3)

A

actinomyces species
filamentous bacteria
normal oral flora

52
Q

CLASSIFICATION OF ACTINOMYCOSIS
 C e r vico - Faci al –
 A b d omino - p elvic –
 P u l monar y –

A

55 %
25 %
15 %

53
Q

CERVICO-FACIAL ACTINOMYCOSIS
A r e a o f p r i o r t r a u m a – e n t r y f o r o r g a n i s m
(5)

A

▪ Sof t tissue injur y
▪ Periodontal poc ket
▪ Non -vital tooth
▪ Extraction soc ket
▪ Infected tonsil

54
Q

CERVICO-FACIAL ACTINOMYCOSIS
direct extension through

A

soft tissue

55
Q

CERVICO-FACIAL ACTINOMYCOSIS
disregards (2)

A

fascial planes, lymphatics

56
Q

CERVICO-FACIAL ACTINOMYCOSIS
induration and fibrosis

A

woody

57
Q

CERVICO-FACIAL ACTINOMYCOSIS
draining

A

sinus tracts

58
Q

CERVICO-FACIAL ACTINOMYCOSIS
suppuration with

A

sulfur granules

Pathology (23 decks)

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