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Step One!!!! Kat's kontributions > Rhythm is gonna get you > Flashcards

Rhythm is gonna get you Flashcards

1
Q

Quinidine

A

Class IA

Toxicity: Cinchonism (HA, tinnitus)

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2
Q

Procainamide

A

Class IA

Toxicity: Reversible SLE-like syndrome

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3
Q

Disopyramide

A

Class IA

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4
Q

Lidocaine

A

Class IB

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5
Q

Mexiletine

A

Class IB

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6
Q

Tocainide

A

Class IB

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7
Q

Flecainide

A

Class IC

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8
Q

Propafenone

A

Class IC

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9
Q

Class IA MOA

A

Na+ channel blocker

Increases AP duration, increases effective refractory period, & increases QT interval

Affect both atrial & ventricular arrhythmias, especially reentrant & ectopic supraventricular & ventricular tachycardia

Toxicity: Thrombocytopenia; torsades de pointes

Double Quarter Pounder

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10
Q

Class IB MOA

A

Na+ channel blocker

Decreases AP duration

Preferentially affect ischemic or depolarized Purkinje & ventricular tissue. Useful in acute ventricular arrhythmias (esp post-MI) & digitalis-induced arrhythmias

Toxicity: Local anesthetic; CNS stimulation/ depression, cardiovascular depression

Lettuce, Tomato, Mayo

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11
Q

Class IC MOA

A

Na+ channel blocker

No effect on AP duration

Useful in VTachs that progress to VF & in intractable SVT. Usually only used as last resort in refractory tachyarrhythmias. For pts w/o structural abnormalities

Toxicity: proarrhythmic, especially post-MI. Significantly prolongs refractory period in AV node

More Fries Please

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12
Q

Which class is affected by hyperkalemia?

A

Hyperkalemia causes increased toxicity for all class I drugs

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13
Q

Propanolol

A

Non-selective beta blocker

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14
Q

Esmolol

A

Selective beta blocker

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15
Q

Metoprolol

A

Selective beta blocker

SE: Dyslipidemia

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16
Q

Atenolol

A

Selective beta blocker

17
Q

Timolol

A

Non-selective beta blocker

18
Q

Carvedilol

A

Beta blocker

19
Q

Acebutolol

A

Selective beta blocker

20
Q

Class II MOA

A

Beta blockers

Decreases cAMP, decreases Ca2+ currents

Suppresses abnormal pacemakers by decreasing slope of phase 4. AV node particularly sensitive. Prolongs PR interval

Used for VTach, SVT, slowing ventricular rate during atrial fibrillation & atrial fibrillation

Toxicity: impotence, asthma exacerbation, cardiovascular effects (bradycardia, AV block, CHF), CNS effects (sedation, sleep alteration). May mask signs of hypoglycemia

Treat OD w/ glucagon

21
Q

Ibutilide

A

K+ channel blocker

Toxicity: Torsades

22
Q

Sotalol

A

K+ channel blocker

Toxicity: torsades de pointes, excessive beta block

23
Q

Bretylium

A

K+ channel blocker

Toxicity: New arrhythmias, hypotension

24
Q

Amiodarone

A 
K+ channel blocker 
BUT has class I, II, III, & IV effects b/c it alters lipid membrane

Toxicities: Pulmonary fibrosis, hepatotoxicity, hypothyroidism/hyperthyroidism, corneal deposits, skin deposits (blue/gray) resulting in photodermatitis, neurologic effects, constipation, cardiovascular effects (bradycardia, heart block, CHF)

25
Q

Dofetilide

A

K+ channel blocker

26
Q

Class III MOA

A

K+ channel blocker

Increases AP duration, increases effective refractory period. Used when other antiarrhythmics fail. Prolongs QT interval

27
Q

Verapamil

A

Class IV

Cardioselective Ca2+ channel blocker

28
Q

Diltiazem

A

Class IV

29
Q

Nifedipine

A

Class IV

Selective for peripheral vasculature

30
Q

Class IV

A

Ca2+ channel blockers

Decreases conduction velocity, increases effective refractory period, prolongs PR interval

Used to prevent nodal arrhythmias (Ie SVT)

Toxicity: Constipation, flushing, edema, CV effects (CHF, AV block, sinus node depression)

31
Q

Adenosine

A

Increases K+ efflux out of cells –> hyperpolarizing the cell

Drug of choice in diagnosing/abolishing SVT

Very short acting (~15 sec)

Toxicities: flushing, hypotension, chest pain

Effects blocked by theophylline

32
Q

Mg2+

A

Effective in torsades de pointes & digoxin toxicity

Step One!!!! Kat's kontributions (28 decks)

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