Respiratory Acidosis - EXAM 3 Flashcards

1
Q

Normal pH

A

7.35-7.45

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2
Q

Normal PaCO2

A

35-45

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3
Q

Normal HCO3 (bicarbonate)

A

24-30

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4
Q

Normal PaO2

A

75-100

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5
Q

Hypoxemic Respiratory Failure

A
  • Inadequate transfer between alveoli and pulmonary capillary beds
  • PaO2 less than or equal to 60 mm Hg on 60% O2
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6
Q

Room air oxygen level

A

21%

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7
Q

Hypercapnic Respiratory Failure

A
  • Most common ABG abnormality
  • Insufficient CO2 removal
  • PaCO2 greater than 45 mm Hg
  • pH less than 7.35
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8
Q

Causes of Hypoxemic Respiratory Failure

A
  • Intrapulmonary shunting
  • Pneumonia
  • Smoke inhalation
  • Shock
  • ARDS
  • V-Q mismatch
  • PE
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9
Q

Intrapulmonary shunting

A

Blood exits lung without participating in gas exchange

  • Pnrumonis secretions
  • Mucus Plug
  • Blood clot from PE
  • ARDS: Acute Respiratory Distress Syndrome
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10
Q

Hypercapnic Respiratory Failure

A
  • Obstructive airway diseases (asthma, COPD, difficulty getting air out)
  • CNS impairment
  • Chest wall dysfunction (problems getting air in)
  • Neuromuscular dysfunction
  • Narcotics, sedatives, barbiturates
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11
Q

What is the respiratory compensatory component?

A

carbonic acid

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12
Q

What is the renal compensatory component?

A

bicarb

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13
Q

Respiratory center

A

In brain. Detects lungs to increase or decrease RR and depth in a response to the amount of CO2. CO2 is retained or blown off.

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14
Q

How fast does compensation occur?

A

System is extremely sensitive to changes in pH and compensation begins in seconds to minutes

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15
Q

Renal Regulation of Acid Base Balance

A
  • Control buffer NaHCO3 (sodium bicarb)
  • Takes several hours to days to kick in
  • Can control HCO3 (bicarb) by either reabsorbing or excreting H+ ions
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16
Q

Respiratory Acidosis Diagnostic Findings

A
  • pH below 7.35
  • PaCO2 above 45 mm Hg
  • HCO3 is normal (24-30)
  • Hyperkalemia
  • Hypercalcemia
  • Lactic acid accumulation
  • Low PaO2
17
Q

Pulmonary Function Tests

A
  • Decreased tidal volume
  • Decreased FVC
  • Decreased FEV 1
  • Decreased minute ventilation
  • End tidal CO2 (ETCO2)
18
Q

Respiratory acidosis assessment data

A
  • Change in LOC/altered MS/headache
  • Progressive sleepiness as CO2 levels increase
  • Dyspnea
  • Rapid, shallow breathing with increased work of breathing (hypoventilation in the end)
  • Patient with HOB up at least 45 degrees
  • Leading to decreased RR and respiratory muscle fatigue (emergency)
  • Hypertension
  • Tachycardia
  • Dysrhythmia
19
Q

Acid-Base Compensation

A
  • The respiratory system attempts to compensate for respiratory acidosis
  • Increased the rate and depth of respirations to blow off CO2
  • The renal system also attempts to compensate for respiratory acidosis…slower process
  • Kidneys begin eliminating H+ ions and retaining HCO3
  • HCO3 levels rise to buffer
  • If no renal compensation is evident in the ABG analysis, the resp. problem is acute
  • Over time, the kidneys compensate by retaining bicarbonate and secreting hydrogen ions
  • If compensation is evident in the ABG analysis, the resp. problem is chronic
  • A compensated ABG differs from a normal ABG in that PaCO2 and HCO3 will both be abnormal and the pH with be normal
20
Q

What’s the relationship between acid-base compensation and correction?

A
  • Acidosis can be temporarily corrected by the medical team by administering NaHCO3
  • Compensation occurs within the body through the effects of the respiratory and renal systems
21
Q

Medical Supportive Therapy

A
  • Correct underlying cause of resp. failure
  • Manage and maintain adequate cardiac output
  • Maintenance of adequate hemoglobin concentration
22
Q

Respiratory Therapy

A
  • Intubation with mechanical ventilation (R. failure)
  • Non-invasive positive pressure ventilation
  • O2 therapy
  • Pulmonary toliet = effective cough, incentive spirometry, hydration/humidification, cchest physiotherapy, airway suctioning
23
Q

Medication Therapy

A
  • Relief of bronchospasm
  • Reduction of airway inflammation
  • Reduction of pulmonary congestion (lasix)
  • Treatment of pulmonary infections
  • Reduction of severe anxiety (lorazepam)
24
Q

Nutritional Therapy

A
  • Parenteral nutritional support
  • Enteral nutritional support
  • Need adequate calories and protein
  • May need to limit carbohydrates in patient who retains CO2
25
Q

Therapeutic Nursing Interventions

A
  • Assess LOC
  • Monitor adequacy of ventilation respiratory status: administer supplemental O2
  • Monitor cardiovascular status
  • Adequate fluids to liquefy secretions and maintain cardiac output
  • Encourage patient to cough/augment cough/suction (pulmonary toliet)
  • Administer and monitor response to medication therapy
  • Administer and assess safe nutritional intake
  • Prevent infection
  • Support patient and family
  • Education
26
Q

Gerontological Considerations

A
  • Physiologic aging of lung brings loss of effective alveoli and loss of surface area for gas exchange in alveoli
  • Decrease in ciliary activity
  • Decrease in resp. muscle strength
  • Greater vulnerability for resp. acidosis and resp. failure