Respiratory Flashcards
ARDS:
Features
Cyanosis
Tachypnoea
Peripheral vasodilation
Bilateral fine end respiratory crackles
ARDS:
Diagnostic criteria
= lung damage with (non cardiogenic) pulmonary edema +/- multi organ failure
Either from direct lung injury or from systemic illness
- acute onset
- CXR bilateral infiltrates
- low PCWP/no CHF
- refractory hypoxaemia
CURB65
Confusion
Urea >7
Respiratory rate >30
BP sys 65
CURB65:
Management
0-1 community ABx
2 admission
3-5 potential ICU admission
Hypercapnic drive:
At risk groups
- COPD
- CF
- Restrictive chest disorders (e.g muscular, neuromuscular)
- Morbid obesity >40
Hypercapnic drive:
Management
- Aim for 88-92% sats
- Decrease but don’t stop O2 2-4l
- Look for previous ABG for baseline
Pleural Effusion:
X Ray features
Blunt costophrenic angle Blunt cardio phrenic angle Fluid in fissures Meniscus Large one sided effusion --> mediastinal shift
Transudate vs Exudate
Transudate = low protein, low LDH, low specific gravity Exudate = high protein, high LDH, low specific gravity
Transudate - from increased hydrostatic pressure or decrease capillary oncotic pressure e.g HF, nephrotic syndrome, cirrhosis
Exudate - lung ca, PE, pneumonia, TB, mesothelioma
Consolidation:
Causes
Infection - pus
Haemorrhage - blood
Cancer - cells
Pulmonary edema - fluid
Consolidation:
X Ray features
Air bronchogram - homogenous opacity with dark lines running through
PE:
Risk factors
Cancer Fracture Immobility Thrombophilia Pregnancy/HRT
PE:
Sx
Tachycardia Tachypnoeic Raised JVP Breathlessness Pleuritic chest pain Haemoptysis Syncope/dizziness
PE:
Management
O2 100% Morphine 10mg+ anti emetic Critically ill --> thrombolysis alteplase /surgery Or LMWH Colloid infuse if hypotensive
ECG, CXR, ABG
D dimer
CTPA
Type I respiratory failure
Low O2 but normal CO2
From lung tissue damage; pneumonia, asthma, COPD, pneumothorax, PE, fibrosis, edema
Type II respiratory failure
Low O2 and high CO2
From poor ventilation eg: COPD, asthma, OD, MG, neuromuscular disorders, obesity
Bronchiectasis:
Definition/cause
Chronic infection of bronchi and bronchioles leading to permanent dilation of airways. From Hib, strep pneumoniae, staph A, pseudomonas
Bronchiectasis:
Symptoms
Persistent cough
Copious purulent sputum
Intermittent haemoptysis
Coarse inspiratory creps
Tram line CXR
Bronchiectasis:
Management
Postural drainage BD
Physio
ABX if needed
Surgery if severe haemoptysis
Pneumonia types:
Pneumococcal
Most common
Everyone
CXR: lobar consolidation
Amox/benpen/cephalosporin
Pneumonia types:
Klebsiella
Rare Elderly, diabetics and alcoholics Cavitating, upper lobes Drug resistant Tx: cefotaxime, imipemen
Pneumonia types:
Staphylococcal
From flu, young, elderly, IVDU, existing disease
Bilateral cavitating bronchopneumonia
Tx: fluclox
Pneumonia types:
Pseudomonas
Common in bronchiectasis
Cause HAF
Tx: anti pseudomonal penicillin
Pneumonia types:
Mycoplasma pneumoniae
Occurs in epidemics
Flu symptoms followed by dry cough
CXR: reticular shadowing, patchy consolidation worse than symptoms suggest
Can cause haemolytic anaemia, meningitis, guillain-barré
Tx: tetracycline, clarithromycin
Pneumonia types:
Legionella
Water tanks eg air con
D+v, hepatitis, anorexia, renal failure, confusion, haematuria
Tx: clarithromycin +-rifampicin
Types of pneumonia:
Chlamydophilia pneumoniae
Pharyngitis (hoarseness), otitis, then pneumonia
Tx: tetracycline or clarithromycin
Pneumonia types:
Pneumocystis pneumonia
HIV
Exertional cough and dyspnoea
CXR: bilateral creps and shadowing
Tx: high dose co-trimoxazole