quiz 14 Flashcards

1
Q

what is Alexia or Acquired Dyslexia

A
  • the ability to read is lost due to brain damage
  • often read letter by letter
  • developmental dyslexia is different, similar symptoms but present in childhood and not due to brain damage
  • damage due to angular gyrus-ventral part of parietal lobe near sylvian fissure
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2
Q

what is agraphia

A
  • the ability to write is lost due to brain damage

- damage to angular gyrus

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3
Q

explain alexia and agraphia

A

usually co-occur, caused by damage to same brain region

  • sometimes there is one without the other-they are doubly dissociable
  • in alexia without agraphia, one can write a sentence and then not be able to read that same sentence
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4
Q

what are the two routes in reading

A

phonological and direct

they are doubly dissociable

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5
Q

what is the phonological route

A

sounding out the words

  • how we learn new words we have not seen before, and non-words such as “glimay”
  • phonological dyslexia: selective damage to phonological route; must memorize all words and cannot sound out new ones
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6
Q

what is the direct route

A

directly read previously memorized words

  • the only way we can read irregular words like “yacht”
  • surface alexia: selective damage to “direct” route; cannot read irregular words and can confuse homophones (beat and beet); often misspell words
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7
Q

what is deep dyslexia

A

similar to phonological dyslexia with additional problems

  • one symptoms is semantic paralexias (reading “forest” as “woods” & difficulty with abstract words like “sympathy” and “faith”)
  • due to over reliance on right hemisphere for language
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8
Q

what is writing evidence for two routes

A
  • phonological agraphia: can manually or orally spell regular and irregular words, but cannot spell non-words
  • lexical agraphia: reasonable spelling for any regular word and non-words, but cannot spell irregular words
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9
Q

what is acquired surface alexia

A

damage to lexical-semantic route (Direct route) and reliance on grapheme-phoneme conversion

  • able to read non-words
  • frequency x regularity interaction
  • regularization errors (yacht read as “yachted”)
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10
Q

explain brain imaging of reading

A
  • MEG signal at about 150 ms after presentation; localized to left inferior occipitotemporal region
  • fMRI: “visual word form area” (VWFA) is more active for words than non-words
  • *there is considerable overlap of networks**
  • *what we use and when depends on the language (ex. Italian is more regular than English so direct route not needed)**
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11
Q

explain Kana and Kanji

A

Kana: phonological mapping (phonological route)
Kanji: symbol not related to phonology (direct route)
-brain damage can cause impairment in Kana and not Kanji, or vice versa

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12
Q

explain intact patients in Kana and Kanji

A
  • fMRI of Kana is slightly more dorsal route, angular gyrus
  • fMRI of Kanji is more ventral route including VWFA
  • similar findings in stimulation of epilepsy patients in brain surgery; however, there is still much overlap in brain regions
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13
Q

how is the right hemisphere used in language

A
  • prosody- tone/ inflection of voice as well as timing
  • narrative-following overall story
  • inference- filling in blanks, figuring out connotations
  • jokes, humor and idioms
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14
Q

explain music and language

A
  • aphasia can occur without amusia, and vice versa; however, aphasia occurs in 80% of cases of amusia
  • spatial aspects of music engage right hemisphere more often
  • pitch processing occurs in right hemisphere
  • musical structure / syntax may rely on same syntax regions as language
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15
Q

explain musical syntax

A
  • ERP component “ERAN” responds to unexpected musical chord
  • comes out of Broca’s area, but bilaterally (both sides)
  • much like the P600
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