Pulmonary Emergencies Flashcards

You may prefer our related Brainscape-certified flashcards:
1
Q

Sound that indicates airway obstruction is incomplete

A

stridor

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Where can the infection of a retropharyngeal abscess spread to?

A

mediastinum causing pleural or pericardial effusion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

extends from the base of the skull to the tracheal bifurcation

A

retropharyngeal space

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Xray finding that is consistent with retropharyngeal abscess

A

expansion of the prevertebral soft tissues

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What is the imaging modality of choice for a suspected retropharyngeal abscess (ie gold standard)?

A

CT scan of neck

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is the managemnet of a patient with a retropharyngeal abscess?

A

IV abx (clinda or unasyn) started in ER and IV hydration. ENT consult for I&D.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Subdermal or submucosal swelling that is diffuse and nonpitting affecting the face, lips, mouth, throat, larynx, extremities, genitalia and possibly the bowel (colicky abdominal pain) . Can occur in isolation, with urticaria or as a component of anaphylaxis

A

angioedema

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What is the treatment of allergic (mast cell mediated) angioedema after assessing the airway?

A

epi 0.3 mg IM, methylprednisolone IV or oral prednisone, and diphenhydramine 25-50 mg IV

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

what is the difference in the between angioedema that is mast cell or bradykinin mediated?

A

mast cell responds to epi, glucocorticoids, antihistamines whereas bradykinin mediated is due to ACEI or hereditary angioedema

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What is the treatment of ACEI (bradykinin) induced angioedema after assessing the airway?

A

discontinue ACEI and reassess in 24-72 hrs if sx haven’t resolved

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is the treatment of hereditary (bradykinin mediated) angioedema after assessing the airway?

A

C1 inhibitor concentrate, Bradykinin receptor antagonist or kailikrein inhibitor

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Acute, potentially lethal, multisystem syndrome from the sudden release of mast cells and basophils into the circulation

A

anaphylaxis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What symptoms are commonly observed in patients experiencing anaphylaxis?

A

pruritic urticaria, angioedema, flushing and hypotension

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What is the treatment of anaphylaxis that reverse the process?

A

epi every 5-15 minutes for up to 3 doses.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What are other pharmacological treatment options in addition to epi that may help treat anaphylaxis?

A

albuterol nebulizer, H1 (ranitidine)/H2 blocker (benadryl), glucocorticoid (solumedrol)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Noise during assessment of head and neck trauma that indicates pooling of liquids in the oral cavity or hypopharynx

A

gurgling

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

What causes wheezing?

A

Narrowing of lower airways

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Why do patients in a stupor or coma have an inability to protect their airway?

A

lack of gag reflex

19
Q

lack of critical cognitive function and level of consciousness wherein a sufferer is almost entirely unresponsive and only responds to base stimuli such as pain.

A

stupor

20
Q

Accumulation of air in the pleural space. Can be spontaneous or trauma induced. Sudden onset of dyspnea and pleuritic chest pain that occurs at rest

A

pneumothorax

21
Q

Describe the patient who is at greatest risk of a pneumothorax

A

tall, thin males between 20-40 yrs (Marfans)

22
Q

Associated with mediastinal shift to the contralateral side and impaired ventilation leading to cardiac compromise

A

tension pneumo

23
Q

Patient who has h/o of heavy smoking presents with sudden stabbing pain in right pectoral and right lateral axillary regions and SOB. PE reveals hyperresonance to percussion. What is the appropriate management?

A

needle aspiration/decompresion followed by chest tube placement (unstable) or primary treatment with chest tube (stable)

24
Q

What locations can be used to accomplish decompression of a pneumothorax?

A

2nd or 3rd ICS at the midclavicular line or at the 5th ICS at the anterior axillary line

25
Q

From sudden increase in left sided intracardiac filling pressures or increased alveolar capillary membrane permeability

A

acute pulmonary edema

26
Q

Major cause of noncardiogenic pulmonary edema

A

ARDS

27
Q

Initial management of acute pulmonary edema both cardiogenic or noncardiogenic

A

O2, lasix 40-80mg IV if stable, then tx cause

28
Q

requires immediate protection of the airway from further injury by intubation. Once intubated can then lavage and suction the lower airway

A

massive aspiration

29
Q

Reduction in airway diameter caused by smooth muscle contraction, vascular congestion, bronchial wall edema, and thick secretions.

A

asthma

30
Q

Quantification of severe hypoxemia

A

SpO2 ≤ 95% despite high flow O2 by nonrebreather

31
Q

Helps give an objective measurement as to the severity of airflow obstruction. Less than 40% of predicted is severe. Should be measured before and after each nebulizer or MDI tx

A

peak flow

32
Q

Bronchodilators used for medical therapy of asthma. Given every 20 minutes via neb or puffs

A

beta 2 agonist (albuterol) or antichoinergic (atrovent)

33
Q

For life threatening asthma exacerbations that remain severe after 1 h of intense bronchodilator therapy. 2 g IV over 20 min

A

magnesium sulfate

34
Q

For severe asthma unresponsive to standard therapies. 0.25mg SQ q 20 min X 3 doses. Administration contraindicated with epi

A

terbutaline

35
Q

Most often precipitated by a viral or bacterial infection. Increase or change in character of usual symptoms of dyspnea, cough or sputum production.

A

COPD exacerbation

36
Q

What is the initial management of an acute COPD exacerbation?

A

O2, solumedrol, levaquin (includes pseudo coverage), bronchodilators

37
Q

EKG findings consistent with a PE

A

S waves in lead 1, Q waves in lead III, and inverted T waves in lead III (S1Q3T3)

38
Q

Pharmacological treatment of hypotension in patient with a PE

A

Fluid bolus of 500 to 1000 ml NS and Vasopressors

39
Q

use in unstable patients for treatment of acute PE in case you need to stop anticoagulation and trial thrombolytics

A

Unfractionated Heparin (UFH)

40
Q

Risk factors for PE described by Virchow’s triad

A

hypercoaguable state, venous stasis, endothelial injury

41
Q

Patient presents with pleuritic chest pain, cough, hemoptysis, diaphoresis. PE reveals tachycardia, tachypnea, crackles, and accentuation of the 2nd heart sound. What is the imaging of choice to work-up this patient?

A

CT scan

42
Q

Give an example of abx options for treating non-ICU pneumonia. Include a respiratory fluoroquinalone and antipneumococcal beta-lactam + a macrolide

A

levoflaxacin or ceftriaxone plus azthromycin

43
Q

What is the abx option for a patient with pneumonia in the ICU who is allergic to PCN?

A

Respiratory fluoroquinolone PLUS aztreonam

44
Q

How does the differentiation between inspiratory and expiratory stridor help distinguish level of airway obstruction?

A

inspiratory- level of larynx

expiratory- level of trachea