Pulmonary Embolism / Pulmonary Edema Flashcards

1
Q

Excess fluid in the lungs (not the pleural space) =

A

pulmonary edema

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2
Q

causes of pulmonary edema

A

• Happens a lot in older folks –> decreased heart/kidney function
◦ HF
◦ Renal failure
◦ ARDS
◦ High altitudes
◦ Brain trauma
◦ Rapidly expanding lungs
— pneunothorax –> Pulling fluid off lungs too quickly when re-expanding the lung
◦ Most common: When giving too much fluid too quickly

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3
Q

what happens when fluid accumulates in the alveoli

A

cant exchange O2/Co2

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4
Q

on a patho level what is causing pulmonary edema (not diseases)

A

• Increased pulmonary pressure increases, fluid leaks across pulmonary capillaries into airway and tissue

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5
Q

which is bigger deal- acute pulmonary edema or pulmonary edema

A

Acute Pulmonary Edema (formerly known as flash pulmonary edema)
=Life threatening emergency

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6
Q

who is highest risk of acute pulmonary edema

A

• HF, renal, older folks
• Laboring parents on a lot of fluid can happen
◦ Results from severe fluid overload

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7
Q

acute pulmonary edema assessment findings

A

• Coarse Crackles (especially in bases)
• Cough
• SOB
• Pink, frothy sputum**
• Dyspnea
• Confusion
• Tachy/Dysrythmias
• Altered BP (low , high, or normal)
• Reduced urinary output (low cardiac output)
• Restlessness/anxiety
• Lethargy

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8
Q

interventions for pulmonary edema

A

• Reassurance
• HOB raised
• O2 increase
• Monitor SpO2, Vital signs
- Meds
• FIX UNDERLYING CAUSES
• Ultrafiltration
• Use ABC’s
• O2
◦ Face mask
◦ Noninvasive positive pressure ventilation (Bipap cpap)
◦ Intubation/Mechanical ventilation

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9
Q

meds we might give for pulmonary edema

A

nitroglycerin, morphine, diuretics, antihypertensives, dobutamine

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10
Q

which drug reduces preload (pulmonary venous return) :

diuretic, antihypertensive, or dobutamine

A

diuretic

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11
Q

which drug provide ionotropic support (heart contraction):

diuretic, antihypertensive, or dobutamine

A

dobutamine

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12
Q

which drug reduces the afterload (systemic vascular resistance):

diuretic, antihypertensive, or dobutamine

A

antihypertensive

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13
Q

goals for managing pulmonary edema long term? who do you want on the team for helping to manage them?

A

-manage underlying diseases that cause pulmonary edema
-HF core measures
◦ Discharge instructions
◦ Left ventricular systolic function
◦ ACE or ARB
◦ Smoking cessation
-• Activity as tolerated / Work up to routine exercise

-case management/social worker

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14
Q

HF core measures re: managing pulmonary edema

A

◦ Discharge instructions
◦ Left ventricular systolic function
◦ ACE or ARB
◦ Smoking cessation

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15
Q

A PE is caused by a blockage- what kind of things can block your vessels?

A

air, liquid, solid

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16
Q

Take me through the patho of what happens when you have a PE

A

• Reduced gas exchange
• Reduced oxygenation
• Pulmonary tissue hypoxia
• Decreased perfusion –> increases resistance in pulmonary vasculature –> increase work of R side of heart to push blood out into the lungs –> R sided HF –> poor perfusion to rest of body
• Possible death

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17
Q

Most common cause of a PE =

A

DVT! VTE (venous thromboembolism)

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18
Q

Path of DVT to PE

A

DVT → dislodges → vena cava → right atrium → right ventricle → pulmonary vessels → platelets aggregate → triggers other substances that cause vessel constriction
→ Impaired gas –> impaired tissue perfusion –> hypoxemia

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19
Q

When the DVT dislodge and goes into your pulmonary vasculature what happens to the right side of the heart?

A

becomes hard to the right ventricle to push blood into the lungs

20
Q

What puts someone at increase risk of DVT/PE

A

• Increased age
• Hypercoagulable states
• Obesity
• hypercoagulable state
• Prolonged immobility
• Central venous catheter
• IV drug use
• Sepsis

21
Q

virchow’s triad =

A

= increase risk of developing a blood clot from
1.) damage to vessel (trauma)
2.) immobility (stasis of blood flow)
3.) hypercoagulability

22
Q

The best way to manage a DVT is to ….

A

prevent it!!

use anticoagulant, mobility, SCD’s

23
Q

asssesment findings for PE

A

• Resp compromise
• Dyspnea
• Chest pain – stabbing, sharp
◦ heart attack = squeezing, crushing
• Restlessness
• Agitation
• Cough
• Bloody sputum from infarcted lung (Pulmonary Edema = pink frothy)
• Abnormal breath sounds
• Tachypnea
• Tachycardia
• Diaphoresis
• Fever- inflammation
• Petechiae (throwing blood clots, hemorrhaging of small capillaries)
• Showering clots
• Increased O2 demands
• Hypotension from decreased cardiac output
• Abnormal heart sounds
• EKG changes

24
Q

sputum with PE vs sputum with Pulmonary edema

A

PE = bloody from infarcted lung
Pulm Edema = pink frothy

25
Q

heart attack chest pain vs PE chest pain

A

heart attack = squeezing crushing
PE = stabbing, sharp

26
Q

diagnostics for PE (long list)

A

• ABG
• BMP
• Troponin
• BNP
• D-Dimer
◦ All it tells us is that there is a blood clot somewhere
• Pulmonary angiography = looks for blood clot in the lungs
• CT-PA
• Ventilation – perfusion scan (V/Q)
• There would be a mismatch
• Chest x-ray
• Ultrasound → Looking for an underlying DVT

27
Q

this lab value tells us there is clot somewhere but not specifically where

A

D- dimer

28
Q

gold standard for diagnosing a PE

A

pulmonary angiography

29
Q

What do you do if you suspect someone has a PE?

A

get some help!!

30
Q

3 priority responses for PE management

A

• ABG’s with normal limits
• Give O2 to maintain SpO2 > 95%
• Patient maintain baseline cognitive status

31
Q

Interventions for PE

A

• HOB
• Increase O2 to maintain SpO2 > 95%
• Call RR
• Reassure your patient
• Assess, assess, assess
◦ Respiratory
◦ Cardiac
◦ Skin
• Imaging
• Prescribed anticoagulants (monitor bleeding!!)

32
Q

Which anticoag med works the fastest?

A

Heparin

33
Q

what labs do we check with heparin ? what labs do we check with warfarin?

A

heparin = ptt or APTT
warfarin = INR

34
Q

how soon do we check aptt or ptt with heparin

A

within 6 hours check ptt or aptt

35
Q

if your patient is going for a prcedure and they are heparin how long do you have to wait after stopping the heparin for them to go have the procedure?

A

stops working within 1 hour of turning off the infusion

36
Q

antidote for heparin? antidote for warfarin?

A

heparin= protamine sulfate
warf = vitamin k

37
Q

how long after stopping lovenox can patient or procedure?

A

12 hours

38
Q

which are the 2 acute anticoags and who is our typical extended?

A

acute = heparin, lovenox
extended = warfarin

39
Q

other extended anticoags you might d/c someone with

A

◦ Rivaroxaban
◦ Dabigatran
◦ Apixaban

40
Q

How do we know the anticoagulant meds are working to treat the PE?

A

clot is getting smaller = breathing better

41
Q

surgical intervention for PE (2) - which one is a temporary solution and why would we do it?

A

• Embolectomy
• Inferior vena cava filtration
◦ Filter to catch clot if they cannot receive anticoagulants , temporary measuer

42
Q

what do we do to treat hypotension related to HF from a PE

A

-in fluids
-iv drug therapy
-positive inotropic meds
-vasopressors
-vasodilators

43
Q

managing and preventing bleeding measures

A

• Assess for bleeding
• Education your patient
• Antidotes
• Monitor CBC, aPTT, PT, INR, platelets
• Bleeding preventions

44
Q

if you have a patient on apixaban do they require lab monitoring?

A

nope!

45
Q

2 tools to manage PE anxiety

A

• Communication!!
◦ stay with the patient
• Pharmaceuticals
◦ morphine, ativan

46
Q

when d/cing a patint with PE what things do we need to consider?

A

Going Home
• Anticoagulation therapy
• Home oxygen?
• Home health
• Follow up care