Dysrhythmia Flashcards

1
Q

this is an early rhythm complex that occurs for the next SA impulse

A

premature complexes

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2
Q

PVC/PAC that occurs every other beat / every 2 beats / every 3 beats?

A

bigeminy , trigeminy, quadrigeminy

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3
Q

2 regular beats then PVC or PAC =

A

trigeminy

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4
Q

3 regular beats then PAC PVC =

A

quadrigeminy

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5
Q

sxs of prematures complexes

A

Palpitations or symptoms of low cardiac output –> check BP

*note: may be asxs as well

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6
Q

premature complexes can be generated by other parts of the heart, what do we see when the PAC starts in the atrium?

A

see P wave that is abnormal, narrow QRS complex

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7
Q

premature complexes can be generated by other parts of the heart, what do we see when the PAC starts in the ventricles?

A

no P wave, wider QRS complex

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8
Q

brady dysrthymia = HR < ____

A

60!

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9
Q

concerns with brady-dysrhythmia - what does it to diastole? what are 3 things we are checking in patient to assess their perfusion?

A

• Prolonged diastole → improved perfusion to heart

OR

• Decreased perfusion to heart if Cardiac Output decreases (low BP)
◦ check BP! to determine if CO is poor perfusion
◦ check LOC
◦ check urine output

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10
Q

tachy dysrhythmia = HR >_____

A

100!

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11
Q

what does tachy-dysthymia do to diastole? what do we need to assess?

A

• Shortens diastole –> heart itself doesn’t get as much O2
• Increased work of heart (heart needs more O2)
◦ uses too much energy
• get BP - low from decrease stroke volume

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12
Q

what will BP be with tach and brady dysthrhytmia

A

low if poor perfusion

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13
Q

causes of sinus tachy

A

• Anxiety, Pain, Fever, Hypoxia, Anemia, Drugs
• May be compensatory

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14
Q

sinus brady s/s

A

• Low BP, Confusion, SHOB, chest pain, dizzy, syncope

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15
Q

who might naturally have a low HR?

A

arthletes

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16
Q

is sinus brady always bad?

A

has therapeutic benefit of reducing myocardial O2 demand and allws for increased perfusion time

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17
Q

when do we use pacing- HR too slow or HR too fast?

A

hr too slow! –> patient not perfusing

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18
Q

type of pacing =

◦ Two large electrodes (Pads)
◦ Stimulates ventricular depolarization
◦ Emergency
A

• Temporary Transcutaneous Pacing

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19
Q

type of pacing: Lead Wires are threaded to right atrium of heart

=

A

• Transvenous Pacing

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20
Q

type of pacing- pacemaker Placed in SubQ pocket =

A

permanent pacemaker

21
Q

pacemakers can pace different parts of the heart -

◦ Atrial (Sick Sinus) - initiates p wave
◦ Ventricular
◦ Biventricular (Heart failure)

the take home from this is you need to know….

A

what your patient is pacing

22
Q

wtf are pacer spikes?

A

= initiates something to happen in the heart

23
Q

3 types of atrial dysrthymias

A

• Premature Atrial Complexes (see above)
• Supraventricular Tachycardia
• Atrial Fibrillation

24
Q

this type of dysrhythmia =
• Rapid stimulation of atrial tissue
• 100 bpm to 280 bpm
- No visible P wave

A

Supraventricular Tachycardia (SVT)

25
what is • Paroxysmal Supraventricular Tachycardia
short run of SVT and got back to normal
26
interventions for SVT
- take BP -identify cause -Vagal maneuver -Adenosine (6mg, 12mg, 12mg) -Fluids - BB, CCB -Cardioversion (= synchronous shock)
27
adenosine protocol for SVT?
◦ causes period of asystole. ◦ feels like you are getting kicked in the chest ◦ Give 6 then 20 cc fluid, 12 then 12 if needed
28
cardiovert or defib SVT?
cardiovert --> synchronous
29
Atrial fibrilation- what are the main causes?
Related to atrial fibrosis and muscle mass ◦ Hypertension ◦ Heart Failure ◦ CAD
30
this dysrhythmia = irritable atria, multiple rapid impulses depolarizing the atria causing decreased CO and NO P WAVE
atrial fibrillation
31
do you see a P wave with A fib?
no P wave on afib
32
A Fib interventions
- O2 -decrease anxiety -Meds (dilt (drip) + amiodoarone (drip) + anticoag) -Cardioversion (synchronized) -Ablation
33
type of dysrhytmia with HR of 140-180 fired from ventricles
V tach
34
risk factors for V tach
◦ Myocardial Ischemia ◦ Cardiomyopathy ◦ Low K+ ◦ Drugs ◦ Shock
35
V tach + pulse = vs V tach + no pulse =
V tach + pulse = check BP , cardioversion, ablation (stable enough) V tach + no pulse = defibrillate, Antidysrhythmic (Amiodarone)
36
type of dysrhythmia with electrical chaos is ventricles meaning the ventricles can't contract =
v fib
37
V fib is chill or not chill?
EMERGENCY, not okay, not chill -- no blood perfusing to body
38
V fib risk factors
• CAD • MI • Low K+ • Low Mg • Surgery, Procedure • Trauma
39
interventions fr V fib =
defibrilate CPR • Antidysrhythmic
40
you have a person in V fib and you shock their ass. Are they gonna be cool now?
must fix underlying cause--- can go back into v fib after you defibrillate
41
type of dysrthymia with no impulses being conducted in the ventricle leading to no cardiac output =
ventricular asystole
42
ventricular aystole is a _____ rhythm
terminal
43
interventions for ventricular asystole
CPR, ventilate, epinephrine
44
type of defibrillator: ◦ Emergency ( have in pyblic places) ◦ BLS
• Automated External Defibrillator
45
type of defibrillator: ◦ Similar to pacemaker ◦ Patient remains on an antidysrhythmia
• Implantable Cardioverter/ Defibrillator
46
type of defibrillator ◦ Vest worn at all times other than bathing ◦ Ability to prevent shock
• Wearable Cardioverter Defibrillator
47
3 types of defibrillators
-AED -Implantable -Wearable
48
dysrhythmias are typically the result of another.....
pathology or disease state so we have to treat that!