Exam 2 Part 2 Flashcards

1
Q

type of spinal cord injury:

damage that eliminated all innervation below injury =

A

complete

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2
Q

type of spinal cord injury:

injury allows some function below injury =

A

incomplete

*more common

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3
Q

causes of secondary SCI

A
  • Hemorrhage
  • Ischemia
  • Hypovolemia
  • Impaired tissue perfusion
  • Edema
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4
Q

type of primary SCI

A
  • Hyperflexion (head forward)
  • Hyperextension (head backward)
  • Axial loading / vertical compression
  • Excessive rotation (twisting)
  • Penetrating trauma
  • Distraction (pulling head up from body)
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5
Q

type of sci: pulling head up from body =

A

distraction

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6
Q

CV involvement with SCI above … what does those CV changes look like?

A

T6 (bradycardia, hypotension, dysrhythmias)

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7
Q

Resp involvement with SCI above…

A

C3-c5

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8
Q

dermatome vs myotomes

A

derm = Zones of sensory motor function
myo= zones of muscle function

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9
Q

plegia vs paresis

A

plegia = paralysis
paresis = weakness

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10
Q

when does spinal shock occur and how long does it last?

A

-occurs immediately after injury
-lasts 48 hours to weeks

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11
Q

T or F: spinal shock results in temporary but complete loss of motor sensory reflex and autunomic function

A

true!

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12
Q

when and how do we use a quad cough

A
  • use hands to push on their diaphragm when they breathe out

indication: secretions that cant be suctioned or reduced coughing ability

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13
Q

autonomic dysrefelxia occurs from sustained triggering stimuli at or below….

A

T6

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14
Q

stimuli that can cause autonomic dysreflexia =

A

◦ Full bladder / UTI
◦ Bowel distension, impaction, constipation
◦ Circumferential compression - thorax, abd, scrotum, or extremities
◦ Tight clothing
◦ Temp
◦ Pain / pressure

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15
Q

(7) sxs of autonomic dysreflexia:

A
  • SNS- Increase in systolic and diastolic BP (sudden drastic increase in BP– risk for stroke!)
  • Reflex Bradycardia - comes from increasing BP
  • Severe HA
  • Nasal congestion
  • Diaphoresis (above injury) cold or goose bumps below
  • Flush skin
  • anxiety
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16
Q

non pharm intervenrtions for SCI

A
  • spinal cord stabilization
  • traction
  • log roll
  • surgery
  • rehab
  • no BLT
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17
Q

meds for SCI

A
  • muscle relaxers
  • steroids
  • pain mangement
  • BP manafgemnt
  • stool softeners
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18
Q

special muscle relaxant we give for SCI?

A

‣ Intrathecal baclofen
‣ Given right at site of spinal cord injury

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19
Q

term, TBI:

  • Sudden and profound injury to the brain
    ◦ GSW
    ◦ Blow to the head
    ◦ Fall
  • Considered “complete” at the time of impact
A

direct

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20
Q

term, TBI:

  • Injury from force applied to another body part with rebound effect
  • Movement of the brain w/i the skull

Ex
◦ Whiplash
◦ Rear end MCV
◦ Skane baby syndrome

A

indirect

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21
Q

causes of secondary TBI (4)

A

◦ Hypotension - MAP < 65
◦ Hypoxia PaO2 80-100
◦ Inc intracranial pressure (aka intracranial hypertension)
◦ Cerebral edema

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22
Q

ICP > ____ = neurons die

A

22

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23
Q

term, TBI:

  • Skull fract or pierced
  • Brain and dura contaminated

Ex
◦ Foreign obj penetration
◦ Linear
◦ Depressed
◦ Comminuted
◦ Basilar*

A

open

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24
Q

term, tbi:
* Skull maintains integrity
* Compilations with ICP

Ex
◦ Contusion
◦ Cerebral lacerations

A

closed

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25
findings of basilar skull fracture:
- leaking CSF - blood in ear - ecchymosis behind ear (battle sign) or around eye - loss of smell/hearing - impaired facial nerve
26
mild vs moderate vs severe TBI: loss of consciousness
mild: disorient, loss of consc up to 3o min moderate: loss of consc 30 min- 6 hour severe: loss of cosc >6 hour
27
mild vs moderate vs severe TBI imaging
mild= no damage moderate= focal or diffuse injury seen severe = focal or diffuse in vessels or ventricles , can see injury early on
28
GCS moderate vs severe TBI
moderate = 9-12 severe = 3-8
29
who is going to ICU for ICP monitoring- mild, mod, or severe TBI?
severe
30
s/s of TBI
neuro changes, battle/raccoon eyes, muscle changes, visual changes, N/V, loc changes, halo sign
31
most common cause of death from TBI =
increase ICP --> herniation
32
re tbi: what is an uncal herniation
◦ Shift of temporal lobe (uncus) ◦ Pressure on oculomotor nerves ◦ Dilated nonreactive pupils
33
re tbi: what is central herniation?
◦ Shift downward - towards brain stem ◦ Cheyne-strokes resp ◦ Pinpoint nonreactive pupils ◦ Hemodynamic instability
34
way to reduce ICP (non pharm)
◦ Prevent coughing / bearing down ◦ HOB 30-45 degrees ◦ do not cluster care- will increase ICP
35
TBI management general
- decrease icp - bp management - o2 - seizure precautions - mannitol - lasix - swall eval - nutrition management
36
what kind of line do we need in order to give hypertonic saline?
central line --> vessicant
37
precautions with craniectomy?
-where helmet when out of bed
38
3 criteria for breath death/organ donation
- irreversible unresponsive coma - absence of brain stem reflexes - apnea (no spont. breath over vent)
39
term: type of cirrohsis: caused by hep C or is drug induced
post necrotic cirrhosis
40
term: type of cirrohsis: caused by chronic alcoholism
Laennec's
41
term: type of cirrohsis: caused by biliary obstruction or autoimmune disease
biliary cirrhosis
42
cirrhosis patho real quick
Liver takes in blood from GI through the portal vein --> build up of blood in portal vein because it cannot move through due to scar tissue --> backs up into GI tract
43
compensated vs decompensated cirrhosis
**Compensated disease**= Scarring is present but liver can *still function* **Decompensated disease**= liver *failure *with symptoms Decompensated and acute liver failure are the same thing
44
complications from cirrhosis include
* Portal Hypertension * Ascites * Esophageal Varices * Hepatorenal Syndrome- impact on kidneys, palliative care involvement at this stage * Hepatic Encephalopathy- build up of toxins * And coagulation problems, jaundice and peritonitis
45
whats the big deal with portal hypertension? what does is allow for?
blood has resistance going through liver --> backs up into GI organs --> new veins form allowing liver bypass --> unfiltered blood enters circulation
46
2 causes of ascites re: cirrhosis
1. **Increased hydrostatic pressure** from portal hypertension pushes fluid out --->Plasma protein leaks out of vessels (less plasma protein in blood) --> not enough protein to pull fluid back into vasculature 2. Liver **can't make enough albumin** --> dependent edema in rest of the body/LE
47
esophageal varices- why do they happen? what make it worse?
backing up of blood from portal hypertension * Heavy lifting * Exercise * Dry, hard food * Intubation/NG tube ---- can also just happen without aggrevating factors so you SOL
48
hepatic enphalopathy is caused by thre backup of what chemical?
ammonia!
49
signs of hepatic enceph.
-sleep/mood/mental/speech changes progressing to altered LOC/neuromuscular/coma (stave IV)
50
things that can lead to hepatic enceph.
* GI bleed * High protein diet * Infection * Hypovolemia (third spacing) * Hypokalemia * Constipation ANYTHING that INCREASES ammonia!
51
patho of hepatorenal syndrome
Increased ascites > pressure on renal artery > decreased blood flow to kidney > renal failure > end of line
52
hepatorenal urinary output is
<500 ml/ day
53
relationship of NASH/ NAFLD/Cirrhosis
NAFLD --> NASH --> Cirrhosis
54
cirrhosis assessmnt findings
-weight loss -RUQ pain - petechiae - palmar erythema - GI bleed - jaundice - ascites - spider angioma - peripheral edema - asterixis - fetor hepaticus (fruity musty breath) - gynecomastia - amenorrhea - impotence - caput medusae
55
AST/ALT ratio for alcoholic liver disease =
>2
56
interventions for cirrhosis
* -Na 1-2 g / day * -paracentesis * -vitamins * -diuretics * -bleeding precautions * -resp support * - screening for varices
57
cirrhosis patients are at risk for what kind of peritonitis ?
Spontaneous Bacterial Peritonitis - lacks obvious source - give ABX - diagnosed with paracentesis
58
drug to give for esophageal varices?
beta blocker - propranolol Reduces Heart rate and hepatic venous pressure (reduces risk of bleeding)
59
bleeding esophageal varices are chill or not chill? and what we gonna do>?
not chill- emergency! lay on side, head down if hypovolemic , 2 large bore Ivs, get BP high enough that they can go down for procedure
60
re surgical interventions for bleeding esoph. varices: a shunt that bypasses the liver?
TIPS
61
re surgical interventions for bleeding esoph. varices: a shunt that bypasses the liver?
TIPS
62
drugs for bleeding esoph. varices?
* Vasopressors – levophed, epinephrine * IV Ocreotide- simatostatin  shunts blood away from GI * IV Protonix * Fluids * Blood products * Antibiotics --> GI bleeding is often caused by infection so treat as if
63
hepatic encph interventions
- moderate protein diet - lactulose (3-4 stools/day) - rifaximin - decreases rate of ammonia
64
3 biggest patho developments of pancreatitis
-low calcium -bleeding -pus formation/necrosis
65
assessment finding for pancreatitis
- steatorrhea - boring mid-epigastric pain that worsens in supine - n/v - fever - tachy - hypotension - diaphoresis - abd guarding - dimished bowel sounds - cullens sign - grey turners sign
66
pancreatitis complications
* Infection/ SEPSIS * Hemorrhage/ Shock * Necrosis * ARDS/ ALI: Acute Respiratory Distress Syndrome and Acute Lung Injury * Renal Failure * Pneumonia * Paralytic ileus * Jaundice * Diabetes
67
2 key labs for pancreatis
elevated amylase and lipase
68
what kind of pain meds we giving for pancreatitis?
IV opioid
69
best way to relieve pancreatisit pain?
NPO
70
pancreatitis interventions
* IVF * IV opioids * Monitor Ca * Antiemetics * Activity as tolerated * NG tube * NPO * skin integrity * education * monitor glucose
71
where do we put feeding tube in for pancreatitis?
Tube feet into small intestine below ligament of treitz --> bypasses pancreas area
72
what is chronic pancreatitis?
-progressive -remission/exacerbation -fibrosis -loss of exocrine function forever --> treat with enzymes
73
pain meds for chronic panc?
non oipoid
74
pancreatic enzyme admin
Do not crush Take with food In conjunction with antacids Prevents pain with digesting food Put in dobhoff if it is clogged
75
what is a whipple done for? what happens?
pancreatic cancer - Removal of proximal head of pancreas, duodenum, part of jejunem, part or all of stomach and gallbladder Anastomosis of pancreatic duct, common bile duct, stomach and jejunem
76
whipple complications
* Diabetes * Bleeding * Infection * Bowel obstruction * Abscess from leaking * Pneumonia * Fistula- leaking that finds its own pathway * Peritonitis