Exam 2 Part 2 Flashcards

1
Q

type of spinal cord injury:

damage that eliminated all innervation below injury =

A

complete

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2
Q

type of spinal cord injury:

injury allows some function below injury =

A

incomplete

*more common

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3
Q

causes of secondary SCI

A
  • Hemorrhage
  • Ischemia
  • Hypovolemia
  • Impaired tissue perfusion
  • Edema
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4
Q

type of primary SCI

A
  • Hyperflexion (head forward)
  • Hyperextension (head backward)
  • Axial loading / vertical compression
  • Excessive rotation (twisting)
  • Penetrating trauma
  • Distraction (pulling head up from body)
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5
Q

type of sci: pulling head up from body =

A

distraction

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6
Q

CV involvement with SCI above … what does those CV changes look like?

A

T6 (bradycardia, hypotension, dysrhythmias)

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7
Q

Resp involvement with SCI above…

A

C3-c5

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8
Q

dermatome vs myotomes

A

derm = Zones of sensory motor function
myo= zones of muscle function

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9
Q

plegia vs paresis

A

plegia = paralysis
paresis = weakness

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10
Q

when does spinal shock occur and how long does it last?

A

-occurs immediately after injury
-lasts 48 hours to weeks

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11
Q

T or F: spinal shock results in temporary but complete loss of motor sensory reflex and autunomic function

A

true!

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12
Q

when and how do we use a quad cough

A
  • use hands to push on their diaphragm when they breathe out

indication: secretions that cant be suctioned or reduced coughing ability

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13
Q

autonomic dysrefelxia occurs from sustained triggering stimuli at or below….

A

T6

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14
Q

stimuli that can cause autonomic dysreflexia =

A

◦ Full bladder / UTI
◦ Bowel distension, impaction, constipation
◦ Circumferential compression - thorax, abd, scrotum, or extremities
◦ Tight clothing
◦ Temp
◦ Pain / pressure

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15
Q

(7) sxs of autonomic dysreflexia:

A
  • SNS- Increase in systolic and diastolic BP (sudden drastic increase in BP– risk for stroke!)
  • Reflex Bradycardia - comes from increasing BP
  • Severe HA
  • Nasal congestion
  • Diaphoresis (above injury) cold or goose bumps below
  • Flush skin
  • anxiety
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16
Q

non pharm intervenrtions for SCI

A
  • spinal cord stabilization
  • traction
  • log roll
  • surgery
  • rehab
  • no BLT
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17
Q

meds for SCI

A
  • muscle relaxers
  • steroids
  • pain mangement
  • BP manafgemnt
  • stool softeners
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18
Q

special muscle relaxant we give for SCI?

A

‣ Intrathecal baclofen
‣ Given right at site of spinal cord injury

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19
Q

term, TBI:

  • Sudden and profound injury to the brain
    ◦ GSW
    ◦ Blow to the head
    ◦ Fall
  • Considered “complete” at the time of impact
A

direct

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20
Q

term, TBI:

  • Injury from force applied to another body part with rebound effect
  • Movement of the brain w/i the skull

Ex
◦ Whiplash
◦ Rear end MCV
◦ Skane baby syndrome

A

indirect

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21
Q

causes of secondary TBI (4)

A

◦ Hypotension - MAP < 65
◦ Hypoxia PaO2 80-100
◦ Inc intracranial pressure (aka intracranial hypertension)
◦ Cerebral edema

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22
Q

ICP > ____ = neurons die

A

22

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23
Q

term, TBI:

  • Skull fract or pierced
  • Brain and dura contaminated

Ex
◦ Foreign obj penetration
◦ Linear
◦ Depressed
◦ Comminuted
◦ Basilar*

A

open

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24
Q

term, tbi:
* Skull maintains integrity
* Compilations with ICP

Ex
◦ Contusion
◦ Cerebral lacerations

A

closed

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25
Q

findings of basilar skull fracture:

A
  • leaking CSF
  • blood in ear
  • ecchymosis behind ear (battle sign) or around eye
  • loss of smell/hearing
  • impaired facial nerve
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26
Q

mild vs moderate vs severe TBI: loss of consciousness

A

mild: disorient, loss of consc up to 3o min
moderate: loss of consc 30 min- 6 hour
severe: loss of cosc >6 hour

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27
Q

mild vs moderate vs severe TBI imaging

A

mild= no damage
moderate= focal or diffuse injury seen
severe = focal or diffuse in vessels or ventricles , can see injury early on

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28
Q

GCS moderate vs severe TBI

A

moderate = 9-12
severe = 3-8

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29
Q

who is going to ICU for ICP monitoring- mild, mod, or severe TBI?

A

severe

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30
Q

s/s of TBI

A

neuro changes, battle/raccoon eyes, muscle changes, visual changes, N/V, loc changes, halo sign

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31
Q

most common cause of death from TBI =

A

increase ICP –> herniation

32
Q

re tbi:
what is an uncal herniation

A

◦ Shift of temporal lobe (uncus)
◦ Pressure on oculomotor nerves
◦ Dilated nonreactive pupils

33
Q

re tbi: what is central herniation?

A

◦ Shift downward - towards brain stem
◦ Cheyne-strokes resp
◦ Pinpoint nonreactive pupils
◦ Hemodynamic instability

34
Q

way to reduce ICP (non pharm)

A

◦ Prevent coughing / bearing down
◦ HOB 30-45 degrees
◦ do not cluster care- will increase ICP

35
Q

TBI management general

A
  • decrease icp
  • bp management
  • o2
  • seizure precautions
  • mannitol
  • lasix
  • swall eval
  • nutrition management
36
Q

what kind of line do we need in order to give hypertonic saline?

A

central line –> vessicant

37
Q

precautions with craniectomy?

A

-where helmet when out of bed

38
Q

3 criteria for breath death/organ donation

A
  • irreversible unresponsive coma
  • absence of brain stem reflexes
  • apnea (no spont. breath over vent)
39
Q

term: type of cirrohsis:

caused by hep C or is drug induced

A

post necrotic cirrhosis

40
Q

term: type of cirrohsis:

caused by chronic alcoholism

A

Laennec’s

41
Q

term: type of cirrohsis:

caused by biliary obstruction or autoimmune disease

A

biliary cirrhosis

42
Q

cirrhosis patho real quick

A

Liver takes in blood from GI through the portal vein –> build up of blood in portal vein because it cannot move through due to scar tissue –> backs up into GI tract

43
Q

compensated vs decompensated cirrhosis

A

Compensated disease= Scarring is present but liver can still function
Decompensated disease= liver *failure *with symptoms
Decompensated and acute liver failure are the same thing

44
Q

complications from cirrhosis include

A
  • Portal Hypertension
  • Ascites
  • Esophageal Varices
  • Hepatorenal Syndrome- impact on kidneys, palliative care involvement at this stage
  • Hepatic Encephalopathy- build up of toxins
  • And coagulation problems, jaundice and peritonitis
45
Q

whats the big deal with portal hypertension? what does is allow for?

A

blood has resistance going through liver –> backs up into GI organs –> new veins form allowing liver bypass –> unfiltered blood enters circulation

46
Q

2 causes of ascites re: cirrhosis

A
  1. Increased hydrostatic pressure from portal hypertension pushes fluid out
    —>Plasma protein leaks out of vessels (less plasma protein in blood) –> not enough protein to pull fluid back into vasculature
  2. Liver can’t make enough albumin –> dependent edema in rest of the body/LE
47
Q

esophageal varices- why do they happen? what make it worse?

A

backing up of blood from portal hypertension
* Heavy lifting
* Exercise
* Dry, hard food
* Intubation/NG tube

—- can also just happen without aggrevating factors so you SOL

48
Q

hepatic enphalopathy is caused by thre backup of what chemical?

A

ammonia!

49
Q

signs of hepatic enceph.

A

-sleep/mood/mental/speech changes
progressing to altered LOC/neuromuscular/coma (stave IV)

50
Q

things that can lead to hepatic enceph.

A
  • GI bleed
  • High protein diet
  • Infection
  • Hypovolemia (third spacing)
  • Hypokalemia
  • Constipation

ANYTHING that INCREASES ammonia!

51
Q

patho of hepatorenal syndrome

A

Increased ascites > pressure on renal artery > decreased blood flow to kidney > renal failure > end of line

52
Q

hepatorenal urinary output is

A

<500 ml/ day

53
Q

relationship of NASH/ NAFLD/Cirrhosis

A

NAFLD –> NASH –> Cirrhosis

54
Q

cirrhosis assessmnt findings

A

-weight loss
-RUQ pain
- petechiae
- palmar erythema
- GI bleed
- jaundice
- ascites
- spider angioma
- peripheral edema
- asterixis
- fetor hepaticus (fruity musty breath)
- gynecomastia
- amenorrhea
- impotence
- caput medusae

55
Q

AST/ALT ratio for alcoholic liver disease =

A

> 2

56
Q

interventions for cirrhosis

A
  • -Na 1-2 g / day
  • -paracentesis
  • -vitamins
  • -diuretics
  • -bleeding precautions
  • -resp support
    • screening for varices
57
Q

cirrhosis patients are at risk for what kind of peritonitis ?

A

Spontaneous Bacterial Peritonitis
- lacks obvious source
- give ABX
- diagnosed with paracentesis

58
Q

drug to give for esophageal varices?

A

beta blocker - propranolol

Reduces Heart rate and hepatic venous pressure (reduces risk of bleeding)

59
Q

bleeding esophageal varices are chill or not chill? and what we gonna do>?

A

not chill- emergency!

lay on side, head down if hypovolemic , 2 large bore Ivs, get BP high enough that they can go down for procedure

60
Q

re surgical interventions for bleeding esoph. varices: a shunt that bypasses the liver?

A

TIPS

61
Q

re surgical interventions for bleeding esoph. varices: a shunt that bypasses the liver?

A

TIPS

62
Q

drugs for bleeding esoph. varices?

A
  • Vasopressors – levophed, epinephrine
  • IV Ocreotide- simatostatin  shunts blood away from GI
  • IV Protonix
  • Fluids
  • Blood products
  • Antibiotics –> GI bleeding is often caused by infection so treat as if
63
Q

hepatic encph interventions

A
  • moderate protein diet
  • lactulose (3-4 stools/day)
  • rifaximin - decreases rate of ammonia
64
Q

3 biggest patho developments of pancreatitis

A

-low calcium
-bleeding
-pus formation/necrosis

65
Q

assessment finding for pancreatitis

A
  • steatorrhea
  • boring mid-epigastric pain that worsens in supine
  • n/v
  • fever
  • tachy
  • hypotension
  • diaphoresis
  • abd guarding
  • dimished bowel sounds
  • cullens sign
  • grey turners sign
66
Q

pancreatitis complications

A
  • Infection/ SEPSIS
  • Hemorrhage/ Shock
  • Necrosis
  • ARDS/ ALI: Acute Respiratory Distress Syndrome and Acute Lung Injury
  • Renal Failure
  • Pneumonia
  • Paralytic ileus
  • Jaundice
  • Diabetes
67
Q

2 key labs for pancreatis

A

elevated amylase and lipase

68
Q

what kind of pain meds we giving for pancreatitis?

A

IV opioid

69
Q

best way to relieve pancreatisit pain?

A

NPO

70
Q

pancreatitis interventions

A
  • IVF
  • IV opioids
  • Monitor Ca
  • Antiemetics
  • Activity as tolerated
  • NG tube
  • NPO
  • skin integrity
  • education
  • monitor glucose
71
Q

where do we put feeding tube in for pancreatitis?

A

Tube feet into small intestine below ligament of treitz
–> bypasses pancreas area

72
Q

what is chronic pancreatitis?

A

-progressive
-remission/exacerbation
-fibrosis
-loss of exocrine function forever –> treat with enzymes

73
Q

pain meds for chronic panc?

A

non oipoid

74
Q

pancreatic enzyme admin

A

Do not crush
Take with food
In conjunction with antacids
Prevents pain with digesting food
Put in dobhoff if it is clogged

75
Q

what is a whipple done for? what happens?

A

pancreatic cancer
- Removal of proximal head of pancreas, duodenum, part of jejunem, part or all of stomach and gallbladder
Anastomosis of pancreatic duct, common bile duct, stomach and jejunem

76
Q

whipple complications

A
  • Diabetes
  • Bleeding
  • Infection
  • Bowel obstruction
  • Abscess from leaking
  • Pneumonia
  • Fistula- leaking that finds its own pathway
  • Peritonitis