Pulmonary Artery Catheters 3 Flashcards

0
Q

What causes decreased preload?

A
  • hypovolemia

- vasodilation

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1
Q

What causes increased preload?

A
  • hypervolemia

- vasoconstriction

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2
Q

What causes increased afterload?

A
  • hypovolemia

- vasoconstriction

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3
Q

What causes decreased afterload?

A
  • hypervolemia

- vasodilation

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4
Q

What pressure determines the preload in the right heart and what is the normal value?

A

CVP 2-6

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5
Q

What pressures determine afterload of the right heart and what are the normal values?

A

PAP 20-30/6-15

MAP 10-20

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6
Q

What resistances determine afterload of the right heart and what are the normal values?

A

PVR 50-150

PVRI 100-240

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7
Q

What pressure determines the preload of the left heart and what is the normal value?

A

PCWP 4-12

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8
Q

What pressures determine the afterload of the left heart and what are the normal values?

A

BP 120/80

MAP 80-100

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9
Q

What resistances determine the afterload of the left heart and what are the normal values?

A

SVR 900-1300

SVRI -100-500

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10
Q

What flows affect the contractility of the left heart and what are the normal values?

A

CO 4-8
CI 2.5-5
SV 60-130
SI 30-50

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11
Q

What affects the contractility/work of the right heart and what are the normal values?

A

LVSWI 50-60

RVSWI 8-10

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12
Q

What are the measured hemodynamic variables?

A
CVP (RA)
PAPS/D
PCWP
CO
HR
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13
Q

What are the derived hemodynamic variables?

A
CI
SV/SVI/SVR
PAM
PVR/PVRI
LVSWI 
RVSWI
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14
Q

What falls under category 1 in the table?

A

Normal or hyperdynamic state (septic shock)

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15
Q

What falls under category 2 in the table?

A

Fluid overload (normal contractility)

16
Q

What falls under category 3 in the table?

A

Hypovolemia (hemorrhage) or dehydration

17
Q

What falls under category 4 in the table?

A

Acute left heart failure (decreased contractility)

18
Q

How do you determine CMO and what is it affected by?

A

CO = HR x SV
CO = VO2/C(a-v)O2
Affected by circulating blood volume, contractility, valve function, pericardial function, metabolic rate, vascular resistance

19
Q

What causes an increased CMO (up to 5 times normal)?

A
Increased metabolism
Septic shock
Exercise
Stress
Pregnancy
Intracardiac shunt
Systemic vasodilation
20
Q

What causes a decreased CMO?

A
Shock
Hypovolemia
Left cardiac dysfunction
Decrease metabolism
Chronic hypotension
Vascular disease
Cardiomyopathies
21
Q

What are the three ways to measure CMO?

A

Thermodilution
Fick equation
Dilution method echo

22
Q

What causes a decreased preload?

A

Hypovolemia - sepsis

< 4 CVP and < 6 PCWP

23
Q

What are the treatments for decreased preload?

A
NSS
Albumin
Whole blood
Ringers lactate
Colloids
24
Q

What causes an increased preload?

A

Hypervolemia vs failure
> 10 CVP
> 20 PCWP

25
Q

What are the treatments for increased preload?

A

Diuretics for hypervolemia

Vasodilators, ionotropics, nitroglycerine for failure

26
Q

What are the treatments for decreased afterload?

A

Norepinephrine
Dopamine
Phenylephrine

27
Q

What are the treatments for increased afterload?

A

Vasodilators such as sodium nitroprusside

Alpha inhibitors such as clonidine

28
Q

What are the treatments for increased contractility?

A

Negative ionotropic agents
Propranolol
Diltiazem

29
Q

What are the treatments for decreased contractility?

A
Digoxin
Dopamine
Dobutamine
Epinephrine
Isoproterenol