Psychopathology Flashcards

1
Q

Deviation from social norms

A

Can be seen as abnormal and undesirable, it looks at the impact of an individuals behaviour on other people, the behaviour is examined in terms of how desirable it is for the individual and the society as a whole. Each society has its own set of rules to govern behajour based on moral standards. Deviation from osical norms is behaviour which goes against unwritten expectations in a given society. Rules can be explicit snd to nreak them could mean breaking the law, other rules are seem as codes of conduct. Deviation from social norms can be used to help identify a person who might be suffering from a mental disorder, if a person is behaving strangely then we can be concerned enough to think they are suffering from a mental disorder.

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2
Q

Evaluation for deviation from social norms

A

-the definition doesn’t always indicate that a person has a psychological abnormality (they might just be odd/eccentric)
-context has to be taken into account (wearing no clothes in shops is abnormal but at a beach it is not)
-cultures have to be accounted for (African cultures believe a it is acceptable to hear God speaking to them but this is auditory hallucinations in western cultures)
+distinguishes between desirable and non desirable, protects members of the public from effects of abnormal behaviours and the damaging consequences of it (no clothes in shops is abnormal and can be damaging for anyone therefore we can minimise that behaviour)

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3
Q

Failure to function adequately

A

Person is unable to deal with everyday demands (jobs) because of personal distress or discomfort, behaviour can be maladaptive, irrational and dangerous. Global Assessment of Functioning Scale (GAF) to assess rates of social, occupational and psychological functioning, 7 criteria- higher the number more abnormal the person is. This model allows psychologists to think in terms of the degree to which a person is abnormal (SUMOVIV)

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4
Q

Evaluation of failure to function adequately

A

-abnormality is not always accompanied by dysfunction ( serial killer Ted Bundy)
-criteria is hard to define and can also be very hard to measure and analyse (how much suffering is enough suffering). Very subjective and lacks being scientific and objective, sometimes should be adhered to (suffering is normal when someone passes away)
+uses GAF scale- makes the model more objective e.g. if patient doesn’t seem to be coping very well in their social life then it could be concluded that is not functioning adequately and is therefore abnormal
+it recognises the patients perspective in terms of their experiences

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5
Q

Statistical infrequency

A

When an individual possesses a less common characteristic than most of the population ( iq above 130) behaviour displayed is statistically rare. (65% of population has iq 85-115, 95% population has average iq, 2.5% have above and below average IQ), the extreme ends of a normal distribution curve can indicate abnormality, statistical infrequency relies on using upto date statistics, can be displayed on a normal distribution curve.

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6
Q

Evaluation of statistical infrequency

A

+ statistical infrequency can be a good thing ( e.g. 130+ iq)
+ objective and scientific methodology to indicate abnormality, can indicate whether someone needs help or assistance
- self esteem and confidence of the person being labelled abnormal for having IQ below 70, due to negative manners from society
- subjective cut off point ( 71 is normal but 70 is not and why?)

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7
Q

Deviation from ideal mental health (Marie Jahoda)

A

Stems from the humanist approach (focusing on motivation and self development, uses mental health to judge abnormality and is related to the lack of contented existence, so someone who deviates from optimal mental health can be considered abnormal. Self actualisation- humans should strive to reach their full potential, she had developed 6 criteria that a person must have to have positive mental health and therefore normal. (APPIES)

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8
Q

Evaluation from deviation from ideal mental health

A

+ positive, productive and holistic
+ shows weak target areas of dysfunction so patient can work on and improve their life
-not very objective and scientific
-very few pple can achieve all criteria- so we can argue that it’s normal to be abnormal
-culture bias: autonomy is more applicable to western cultures whilst eastern ones focus more on whole community

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9
Q

Phobias

A

Actions- Avoidance, endurance, panic and disruption of functioning
Feelings- Fear, panic, anxiety, strong emotions
Thinking- Irrational, insight, cognitive distortions, selective attention

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10
Q

Classical conditioning

A

All behaviours (phobias) can be learnt and people who have an abnormality can learn negative behaviours. Classical conditioning: building up an association between two different stimuli so that learning can take place, e.g white rat (ns) is presented to a person, loud banging noise (us) is presented to make the person cry, then we repeatedly pair the two stimuli together, they learn to have an emotional response when the rat is present, rat becomes (cs) and the person has a emotional reaction (cr), learning has taken place via classical conditioning and association has been established

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11
Q

Evaluation of CC

A

-little Albert study is criticised (not reliable)
-some people do have traumatic experiences like car accident, however do not develop a phobia, some people r scared of dogs but haven’t had a single experience (dinardo et al)
-Menzies, pple with hydrophobia only 2% encountered before, 50% with dog phobia have never experienced anything bad
+king (1998) from reviewing cases has found out children develop phobias by having traumatic experiences with the phobic object e.g. children bitten by dogs go onto have phobias of dogs.

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12
Q

Social learning theory

A

Observational learning, whereby young children observe a reaction from parents or family to particular situations and the child copies this behaviour, Minneka found out that one monkey caged showed a fear response to snakes and then others started to copy it

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13
Q

operant conditioning

A

It explains how phobias are maintained, learning a new phobia that can result in reinforcement. Negative: if someone is scared of snakes they will try to avoid them to reduce the risk they fear.
Positive: avoiding snakes and not feeling fear, this is rewarding. Therefore the avoidance of snakes continues

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14
Q

Two process model evaluation

A

-some pple might have genetic vulnerability to development of phobias (genetic model is ignored)
-cognitive characteristics of phobias are ignored, this needs to be investigated more fully to gain a comprehensive view of how phobias might be learnt
-reductionist, over simplistic approach to explaining the phenomenon of learning phobias into two steps
+Bandar’s experiment- person acted to be in pain when buzzer sounded
+ two clear steps that highlight how phobias are learnt and how they are maintained

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15
Q

Systematic desensitisation

A

Behavioural therapy by Wolfe to diminish phobias using cc, a person with phobia experiences fear as a behavioural response to an object, sd replaces this irrational fear of the phobic object to calm and relaxed response instead, (hierarchy of fear, relaxation techniques, gradual exposure-leads to extinction)

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16
Q

Relaxation of techniques

A

Taught deep muscle relaxation techniques (deep breathing and progressive muscular relaxation)- tense a group of muscles so that they are tightly contracted and then relax muscles to their previous state. Sd revolves around the idea that it’s impossible to experience two opposite emotions at the same time (reciprocal inhibition) , counter conditioning- patients learn to remain relaxed in the presence of phobia

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17
Q

Evaluation of SD

A

+assessment of various treatments- 87% panic free after sd, 50% after medication, 36% with placebo, therefore SD is most effective
+less traumatic
- not very practical in real life instances
-time consuming when compared to flooding and other treatments
-addresses the symptoms not the actual underlying cause- symptom substitution may occur

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18
Q

Flooding

A

Direct exposure to phobic object, taught relaxation techniques beforehand, no gradual build up. It is done in vivo, immediate exposure, no option for avoidance, extinction occurs when the patient is too exhausted by their own fear response- it is ethical
Lasts for 2-3 hrs who’s is longer than SD sessions

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19
Q

Evaluation of flooding

A

+ cost effective
+ can be applied to every day life outside of the therapy situation- when facing their feared object in other situations
-less effective for social phobias: cognitive therapies might be more useful
-highly traumatic

20
Q

Depression

A

Action- Shift in energy, social impairment, weight changes, poor personal hygiene, sleep pattern disturbance, aggression and self harm
Thinking- Delusions, reduced concentration, thoughts of death. Poor memory, negative thinking, absolutist thinking
Feelings- Loss of enthusiasm, constant depressed mood, worthlessness, anger

21
Q

Cognitive approach to explaining depression

A

Individuals who suggest from depression often have distorted and negative thinking, people who think negatively or irrationally might be more prone to developing depression

22
Q

Cognitive triad

A

People become depressed due to negative outlook and develop negative schemas which dominate their thinking (developed in childhood due to harsh parenting) continue into adulthood and build into negative framework, (self blame and ineptness)
-self, world, future

23
Q

Evaluation of cognitive triad

A

+ influential in the last 30 yrs, since its objective and permits testing, distorted and negative thinking are common in people with depression, and they play a key role in the development of the illness
+Terry (2000) tested 65 pregnant women before and after birth for cognitive vulnerability
-behavioural approach argues otherwise
- cause and effect is unclear
-doesn’t explain how symptoms develop- hallucination anger and etc (Cotard syndrome) these symptoms aren’t accounted for

24
Q

Ellis’s ABC model

A

Activating event- has a negative affect on their mood and outlook, beliefs, consequences
Mustabatory thinking- causes irrational and negative thinking that can be emotionally damaging leading to depression, an individual with such beliefs is bound to get disappointed since these thoughts are too idealistic and the expectations are too high

25
Q

Evaluation for ABC model

A

+bates- depressed patients given negative statements become more and more depressed, if psychologists know what causes depression then effective treatment can be built
+based on scientific evidence to allow objective testing, improvement and greater understating for the causes of depression
+people who develop depression in adulthood tend to experience insecure attachments in childhood
-causes and effect unclear
-blames the client
-biological approach suggests genes and neurotransmitters cause depression, Zhang found that there is a gene linked to depression- 10x more likely to develop the illness

26
Q

Treatment of depression (cognitive)- CBT by Beck

A

Modify negative schemas so that they become positive and rational, can be done individually or in groups, gives the patient control over their thinking, focuses on present experiences, therapist should be highly trained, negative thinking is challenged, patients are encouraged to identify their negative thoughts (thought catching) in diaries, must test out their thoughts and acts as a scientist to generate a hypothesis to test validity of irrational thoughts. Reinforcements of positive thought is encouraged

27
Q

Evaluation of CBT

A

-drugs are more appropriate than CBT when severe depression, but it is fine for mild since it stops it from getting worse
-most popular treatment is anti depressant drugs, requires less effort
+ NHS supports it and widely uses it as a treatment for depression, quite economical compared to other treatments like psychodynamic
+uncovers the root of depression
+long term cure for depression

28
Q

REBT by Ellis

A

Same as CBT+ it is reinforcing the idea of positive thoughts from the ABC model, homework is given to participants to test irrational beliefs out in the real world and replacing it with more rational and positive beliefs, behavioural activation- encouragement depressed clients to become more active and engage in pleasurable activities. Uses logical and empirical disputing

29
Q

Evaluation of REBT

A
  • cause and effect is unclear
  • dependent on the patient to be articulate about their emotions
    -also depends heavily on the expertise of the therapist
30
Q

OCD

A

Anxiety disorder, characterised by the DSM-V as a disorder whereby the patient shows repetitive behaviour (compulsions) and obsessive thinking

31
Q

OCD

A

Behaviours- Compulsive behaviours, hinder everyday functioning, social impairment, repetitive, avoidance
Feelings- Anxiety, distress, accompanying depression, guilt, disgust
Thinking- Obsessions, recognised as self generated, insight of irrationality, selective attention

32
Q

Difference between compulsions and obsessions

A

Compulsions- are external components that can be seen by everyone whilst obsessions are intervals bevause they are thought that occur internally inside the mind

33
Q

Biological approach to explain OCD

A

Caused by genetics and neural explanations

34
Q

Genetic causes of OCD

A

Genes come in different alleles and OCD is an inherited condition, sufferers have genetic vulnerability to get the illness, often for studying this illness twins or family members are used- it is a polygenic illness ( upto 230 different genes)- candidate genes are responsible for causing ocd, different combination of candidate genes may account for different types of OCD which exist

35
Q

COMT/SERT gene

A

C-regulates the production of the neurotransmitter dopamine, high levels are associated with ocd it is responsible for motivation, drive and aggression, found to be more common in OCD patients than people who do not have the illness

S- Affects the transportation of serotonin which results in low levels of it (low mood and depressive symptoms)- present on chromosomes 17, mutation to this gene can cause OCD, ozaki found evidence to say that 6 out of 7 family members with ocd had a mutation to the SERT gene.

36
Q

Evaluation for genetic explaination

A

+ first degree relatives who had OCD meant that they were 5x more likely to get it aswell
+ meta analysis of 14 twin studies that OCD is twice more likely to be concordance in identical MZ twins than DZ twins
-concordance rate is not 100%, therefore can not be entirely genetic factors
-behaviour approach is ignored
-diathesis stress model that OCD can be caused by a combination of genes and trigger in the environment

37
Q

Beekman and Cath (2005)

A

Meta analysis of twin studies and OCD, MZ twins compared to DZ, OCD patients had been diagnosed with the older criteria (ICD), than also the DSM criteria, 10032 twin pairs were studied. The results found that it is inherited via genes and the genetic influence ranged from 27-47% (adults), 45-65% (adults), so the conclusion was that it is transmitted genetically and this was more apparent in children than adults

38
Q

Evaluation of Beekman and Cath

A
  • most of the twin studies were not done in controlled conditions
  • gene mapping was not considered ( DNA of the twins which had OCD vs DNA of those which didn’t), a comparison would make rhe results more robust and valid
    +large sample
39
Q

Neural explanation of OCD/ dopamine/ serotonin

A

Dopamine is a neurotransmitter that affects mood, the frontal lobes are linked to dopamine activity, patients have high levels of dopamine, research on animal has found that high doses of drugs which enhance dopamine levels can induce movement that resembles compulsions and repetition behaviours
Also linked to over activity in the basal ganglia area in the brain (motor function and learning)

Serotonin is a neurotransmitter which also affects mood, linked to the frontal lobes lobes, patients tend to have low levels of serotonin and this can cause depressive like symptoms and also obsessive thoughts.
Plays a key role in operating the caudate nucleus, and it seems that low levels cause it to malfunction

40
Q

Evaluation of neural explanations

A

+anti depressants raise serotonin levels, so low levels cause OCD
+risperidone drug helps to lower dopamine levels which alleviates the symptoms
-high levels of dopamine are not only related to OCD and other illnesses (schizophrenia and bipolar depression)
-cause and effect is not clear
-OCD comorbids (exists) alongside depression- so do low levels of serotonin cause OCD, depression or both? Needs to be investigated further

41
Q

SSRI to treat OCD (biological approach)

A

Corrects the imbalance of neurochemical, selective serotonin re uptake inhibitors (Prozac and fluoxetine), low levels of serotonin is associated to ocd and depression

42
Q

SSRI (how does it treat)

A

It prevents the reuptake of serotonin and prolongs its activity in the synapse, the person will feel less anxious because there is more serotonin available, also prevent the re absorption and breakdown of serotonin in the brain, low levels are implicated in the worry circuit, whereby damage to the caudete nucleus in the brain dials to suppress minor worry signals, sending a message to the orbital frontal cortex and the worrying becomes worse (prescribed for 3-4 months)

43
Q

Alternative for SSRI

A

SNRI- selective norepinephrine reuptake inhibitors- increases serotonin and norepinephrine levels, a neurotransmitter released from the sympathetic nervous system in response to stress if ain’t to mobilise the body and brain for action

44
Q

Evaluation for SSRI

A

+ soomro, reviewed 17 studies compared placebos to SSRI all showed the drug was more effective, especially when combined with CBT
+ really effective, 70% patients show a decline, whilst the other 30% had to combine with other methods such as psychological therapies
+cheap and cost effective
-doesn’t work for everyone
-side effects which stop patients from taking the medication (indigestion, blurred vision and loss of sex drive)
-drug therapies might relapse so psychotherapies should be tried first

45
Q

Benzodiazepine

A

Reduces anxiety and tries to control the action of the neurotransmitters (Xanax, Valium), it also reduces brain arousal, blood pressure and reduce heart rate, it aims to increase GABA (gamma amino butyric acid), this slows down the firing of neurons and makes the person less anxious and clad we, reduced physiological activity in the body
It also reduces serotonin levels

46
Q

BA and GABA

A

By binds to gaba receptor site of the post synaptic neurone, increasing the flow of the chloride ions into the post synaptic neurone, the chloride ions make it more difficult for the neuron to be stimulated by other neurotransmitters , thus slowing down its activity and making the person feel more relaxed

47
Q

Evaluation for Benzodiazepines

A

+ very effective- used by millions of
+immediate benefits of relief within short time of use unlike psychological treatments
+side effects are minimum
-longer term use has side effects (alcohol, depression and drowsiness)
-drug escalation if stopped using
-impairment of speed and processing of verbal learning