Praxis Review Flashcards

1
Q

What does the superior vestibular branch of CNVIII innervates

A
  1. Superior (anterior) and Horizontal (lateral) semi-circular canals
  2. Utricle
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2
Q

What does the inferior vestibular branch of CNVIII innervates

A
  1. Posterior (inferior) SSCs
  2. Saccule
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3
Q

What is an expected condition of nystagmus for peripheral vestibular nystagmus?

A
  1. Horizontal
  2. Horizontal or combination of horizontal and torsional
  3. Follows Alexander’s Law
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4
Q

What does abnormal findings pertaining to caloric testing indicate?

A

Peripheral lesion involving the horizontal SCCs and/or superior nerve brance of the vestibular nerve on the side with the weaker response

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5
Q

List common causes of peripheral vestibulopathy

A
  1. Ototxicity
  2. BPPV
  3. Meniere’s Dx
  4. Vestibular Neuritis
  5. Labyrinthitis
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6
Q

Define peripheral vestibulopathy

A

Disorder or dysfunction that involves the inner ear structure responsible for balance and spatial orientation; specifically the vestibular apparatus and vestibular nerve

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7
Q

List of vestibular assessment testing that assess the horizontal SCCs and superior branch

A
  1. Calorics
  2. Rotational Chair Testing
  3. vHIT
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8
Q

What test assess the otolith organs?

A

VEMPs

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9
Q

What does the cVEMP pathway includes?

A
  1. Saccule
  2. Inferior Branch of CN VIII
  3. CN XI
  4. Descending vestibulo-spinal pathway to Sternocleidomastoid (SCM)
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10
Q

What kind of recorded response does cVEMP provide?

A

Inhibitory as the SCM nust be contracted to record the response

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11
Q

What kind of recorded response does oVEMP provide?

A

Excitory contralateral reflex, right ear stimulation is recorded from the left inferior oblique

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12
Q

What does the oVEMP pathway includes?

A
  1. Utricle
  2. Superior branch of CN VIII
  3. CN III
  4. Contralateral Inferior Oblique Muscle (ascending vestibulo-ocular reflex)
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13
Q

Between DPOAEs & TEOAEs state what type of hearing loss woule be expected

A
  1. DPOAEs: Can be recorded with up to moderate hearing loss
  2. TEOAEs: Typically absent with more than a mild hearing loss (20-30 dBHL)
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14
Q

For ABR testing, what is the waveform patterns for normal hearing?

A

Morphology: Good

Wave Latencies: All within normal limits

Wave Amplitude: Normal

Latency-Intensity Function: Falling within normal range

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15
Q

For ABR testing, what is the waveform patterns for CHL?

A

Morphology: Good

Wave Latencies: Interwave within normative limits; Waves I-V delayed

Wave Amplitude: Normal

Latency-Intensity Function: Outsie of normative range

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16
Q

For ABR testing, what is the waveform patterns for sensory HL?

A

Morphology: Poor

Wave Latencies: Interwave within normal limits; Waves I=V slightly delayed

Wave Amplitude: Waves I-III small to absent

Latency-Intensity Function: High-intensity responses within normal range; all others would be outside

17
Q

For ABR testing, what is the waveform patterns for neural HL?

A

Morphology: Poor

Wave Latencies: Interwave delayed; Waves III & V delayed

Wave Amplitude: Normal

Latency-Intensity Function: High-intensity responses within normal range; all others would be outside

18
Q

For neurodiagnositic ABR, what are the expected changes from a 21.1 to 89.1 repetition rate

A

Lower rate (e.g., 21.1):
Amplitude (Larger)
Wave V Latency (Shorter)
I-V Interpeak Latency (Normal)

Higher Rate (e.g., 89.1):
Amplitude (Shorter)
Wave V Latency (Longer)
I-V Interpeak Latency (Prolonged)

19
Q

For acute vestibular syndrome, what are the peripheral and central etiology

A

Peripheral Etiology = Vestibular Neuritis

Central Etiology = Brainstem, Cerebellar infarct

20
Q

For recurrent vertigo, what are the peripheral and central etiology

A

Peripheral Etiology = Meniere disease, superior semicircular canal dehiscence (SSCD)

Central Etiology = Vestibular migraine, transient ischemic attack (TIA)

21
Q

For positional vertigo, what are the peripheral and central etiology

A

Peripheral Etiology = BPPV

Central Etiology = Cerebellar disorder, vestibular migraine

22
Q

For chronic dizziness, unsteadiness, what are the peripheral and central etiology

A

Peripheral Etiology = Bilateral vestibulopathy, vestibular schwannoma

Central Etiology = Persistent postural-perceptual dizziness (PPPD)

23
Q

List what makes labyrinthitis different from vestibular neuritis

A
  • Similar to vestibular neuritis except patient reports concurrent sudden hearing loss and tinnitus
  • Hearing loss and vertigo may occur simultaneously or within a few days of sudden hearing loss and tinnitus
24
Q

What caues vestibular neuritis?

A

Infection/inflamation of the vestibular nerve, most often involving the superior branch

25
Q

What are symptoms of vestibular neuritis?

A
  • Acute onset of vertigo
  • Nausea/vomitting
  • Generalized imbalance
  • Acute phase can last for hours to days
  • Imbalance may linger for weeks after
  • No hearing loss or tinnitus associated
26
Q

What causes labyrinthitis?

A

Due to inflammation of the inner ear and/or CN VIII

27
Q

List all of the 3rd Window Disorders

A
  1. Perilymph fistula
  2. Superior Semicircular Canal Dehiscence (SSCD)
  3. Enlarged Vestibular Adeduct Syndrome (EVAS)
28
Q
A