Praxis Review Flashcards

1
Q

What does the superior vestibular branch of CNVIII innervates

A
  1. Superior (anterior) and Horizontal (lateral) semi-circular canals
  2. Utricle
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2
Q

What does the inferior vestibular branch of CNVIII innervates

A
  1. Posterior (inferior) SSCs
  2. Saccule
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3
Q

What is an expected condition of nystagmus for peripheral vestibular nystagmus?

A
  1. Horizontal
  2. Horizontal or combination of horizontal and torsional
  3. Follows Alexander’s Law
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4
Q

What does abnormal findings pertaining to caloric testing indicate?

A

Peripheral lesion involving the horizontal SCCs and/or superior nerve brance of the vestibular nerve on the side with the weaker response

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5
Q

List common causes of peripheral vestibulopathy

A
  1. Ototxicity
  2. BPPV
  3. Meniere’s Dx
  4. Vestibular Neuritis
  5. Labyrinthitis
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6
Q

Define peripheral vestibulopathy

A

Disorder or dysfunction that involves the inner ear structure responsible for balance and spatial orientation; specifically the vestibular apparatus and vestibular nerve

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7
Q

List of vestibular assessment testing that assess the horizontal SCCs and superior branch

A
  1. Calorics
  2. Rotational Chair Testing
  3. vHIT
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8
Q

What test assess the otolith organs?

A

VEMPs

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9
Q

What does the cVEMP pathway includes?

A
  1. Saccule
  2. Inferior Branch of CN VIII
  3. CN XI
  4. Descending vestibulo-spinal pathway to Sternocleidomastoid (SCM)
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10
Q

What kind of recorded response does cVEMP provide?

A

Inhibitory as the SCM nust be contracted to record the response

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11
Q

What kind of recorded response does oVEMP provide?

A

Excitory contralateral reflex, right ear stimulation is recorded from the left inferior oblique

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12
Q

What does the oVEMP pathway includes?

A
  1. Utricle
  2. Superior branch of CN VIII
  3. CN III
  4. Contralateral Inferior Oblique Muscle (ascending vestibulo-ocular reflex)
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13
Q

Between DPOAEs & TEOAEs state what type of hearing loss woule be expected

A
  1. DPOAEs: Can be recorded with up to moderate hearing loss
  2. TEOAEs: Typically absent with more than a mild hearing loss (20-30 dBHL)
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14
Q

For ABR testing, what is the waveform patterns for normal hearing?

A

Morphology: Good

Wave Latencies: All within normal limits

Wave Amplitude: Normal

Latency-Intensity Function: Falling within normal range

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15
Q

For ABR testing, what is the waveform patterns for CHL?

A

Morphology: Good

Wave Latencies: Interwave within normative limits; Waves I-V delayed

Wave Amplitude: Normal

Latency-Intensity Function: Outsie of normative range

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16
Q

For ABR testing, what is the waveform patterns for sensory HL?

A

Morphology: Poor

Wave Latencies: Interwave within normal limits; Waves I=V slightly delayed

Wave Amplitude: Waves I-III small to absent

Latency-Intensity Function: High-intensity responses within normal range; all others would be outside

17
Q

For ABR testing, what is the waveform patterns for neural HL?

A

Morphology: Poor

Wave Latencies: Interwave delayed; Waves III & V delayed

Wave Amplitude: Normal

Latency-Intensity Function: High-intensity responses within normal range; all others would be outside

18
Q

For neurodiagnositic ABR, what are the expected changes from a 21.1 to 89.1 repetition rate

A

Lower rate (e.g., 21.1):
Amplitude (Larger)
Wave V Latency (Shorter)
I-V Interpeak Latency (Normal)

Higher Rate (e.g., 89.1):
Amplitude (Shorter)
Wave V Latency (Longer)
I-V Interpeak Latency (Prolonged)

19
Q

For acute vestibular syndrome, what are the peripheral and central etiology

A

Peripheral Etiology = Vestibular Neuritis

Central Etiology = Brainstem, Cerebellar infarct

20
Q

For recurrent vertigo, what are the peripheral and central etiology

A

Peripheral Etiology = Meniere disease, superior semicircular canal dehiscence (SSCD)

Central Etiology = Vestibular migraine, transient ischemic attack (TIA)

21
Q

For positional vertigo, what are the peripheral and central etiology

A

Peripheral Etiology = BPPV

Central Etiology = Cerebellar disorder, vestibular migraine

22
Q

For chronic dizziness, unsteadiness, what are the peripheral and central etiology

A

Peripheral Etiology = Bilateral vestibulopathy, vestibular schwannoma

Central Etiology = Persistent postural-perceptual dizziness (PPPD)

23
Q

List what makes labyrinthitis different from vestibular neuritis

A
  • Similar to vestibular neuritis except patient reports concurrent sudden hearing loss and tinnitus
  • Hearing loss and vertigo may occur simultaneously or within a few days of sudden hearing loss and tinnitus
24
Q

What caues vestibular neuritis?

A

Infection/inflamation of the vestibular nerve, most often involving the superior branch

25
What are symptoms of vestibular neuritis?
* Acute onset of vertigo * Nausea/vomitting * Generalized imbalance * Acute phase can last for hours to days * Imbalance may linger for weeks after * No hearing loss or tinnitus associated
26
What causes labyrinthitis?
Due to inflammation of the inner ear and/or CN VIII
27
List all of the 3rd Window Disorders
1. Perilymph fistula 2. Superior Semicircular Canal Dehiscence (SSCD) 3. Enlarged Vestibular Adeduct Syndrome (EVAS)
28
What condition could Enlarged Vestibular Adeduct Syndrome (EVAS) also be seen in?
1. Pendred Syndrome 2. Brachino-oto-renal (BOR) Syndrome
29
What is the ANSI adpoted guidelines for classroom acoustics?
35 dBA or less and the time of 0.6 seconds or less
30