Perinatal issues in sheep COPY COPY Flashcards
How much do of BW do animals usually eat
2.1%
Causes and treatment of ringwomb
Failure of cervix to dilate
- May be due to premature parturition, malpresentation, disruption, low Ca2+, cervical scarring, exogenous oestrogens
Treating: manual dilation
- May use prostaglandins or oestradiol benzoat
Treatment given in cases of uterine prolapse
Abs, NSAIDs, Ca2+ (because low Ca2+ can cause uterine inertia which contrbutes), oxytocin to aid involution
NB: not any more likely to recur
Purse string vs Buhner suture
Purse string is for anus
Buhner is for vagina
In twin lamb disease what two factors cause clinical signs
Hypoglycaemia
Hyperketonaemia
Traeting twin lamb disease
Palatable good, propylene glycol mixture, IV glucose if severe neurological signs…
Preventing twin lamb disease
Ensure sheep in good body condition at lambing i.e 3-3.5
> Should feed according to number of fetuses
Differences between ketosis in sheep and goats
In goats we see in lactation as well as late pregnancy
+ signs include subcut oedema
What is a low blood calcium level
<1.4mmol/L
Treating hypocalcaemia in sheep
Give 10-20ml of 40% calcium borogluconate; IV
+ calcium/magnesium/dextrose subcut solutino
Causes of hypocalcaemia
(high Ca2+ demand e.g in late pregnancy)
- See signs after stress, decreased food intake, change in diet, need enough Mg2+ for uptake which has no stores
When do we see issues of hypomagnesaemia in sheep
Early lactation
> No body stores and high demand
+ Uptake Na+ dependent so lower when lower Na:K ratio
Signs of hypomagnesaemia in sheep
Ataxia, incoordinatino, increased TPR< collapse, convulsion and death
Treating hypomagnesaemia in sheep
SUBCUTANEOUS injection of magnesium sulphate
Distinguishing hypocalcaemia, hypomagnesaemia and pregnancy toxaemia
Timing and speed of onset
Hypocalcaemia = fastest onset + pre-lambing
Pregnancy toxaemia = slow onset; >48hours; pre lambing
Hypomagnesaemia = medium onset BUT usually in lactation
Difference in presentation of acute vs chronic mastitis
Acute: sudden onset, lame appearance ewe, pyexic/inappatent, udder red/painful/swollen
Chronic: may just notice from poor lamb growth; lumps in udder but ewe systemically well
Difference in approach to acute vs chronic mastitis in ewes
Acute: systemic and antimammary Abs, NSAIDs, stripping BUT KEEP
Chronic: may lance abscesses but cull at weaning
Causative agents in mastitis
In 80% isolate M haemolytica (from lamb URT), S aureus (skin commensal)
Also get some cases with E coli, T pyogenes other staphs
Chronic cases often S aureus or T pyogenes