pathologies related to knee - exam 3 Flashcards

1
Q

what is DVT

A

partial or complete occlusion of a vein by a clot

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2
Q

causes of DVT:
conditions involving at least two of the following:

A
  1. venous stasis
  2. hypercoagulability
  3. damage to the venous wall
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3
Q

risk factors of DVT

A

prior DVT
hx of cancer, CHF, or lupus
major infection, surgery, or trauma
present chemo, immobility, pregnancy
use of oral contraceptives
genetic clotting disorder
> 60 years

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4
Q

3rd most common cardiovascular disease

A

DVT

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5
Q

where are DVTs most commonly found

A

LE deep veins
popliteal, femoral, tibial, peroneal

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6
Q

DVT is the most preventable cause of _____
DVT is the most common cause of ______

A

hospital related death
readmissions and death after THA/TKA

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7
Q

pathogenesis of DVT

A

greater exposure of platelets and clotting factors to damaged venous wall
fibrin, leukocytes, erythrocytes adhere and form thrombus

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8
Q

DVT prevention and treatment per MD direction

A

early and regular exercise
anticoagulants
compression stockings
intermittent pneumatic compression devices
avoid SAD
eliminate persistent smoking and drinking

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9
Q

true or false. ~50% DVTs asymptomatic in early stages

A

true

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10
Q

S&S DVT

A

gradual onset of dull ache, tightness, & P! in calf
edema, possibly pitting (70%)
increased calf girth
calf pain and tenderness (50%)
– Worsened with walking and calf down
possible warmth and redness

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11
Q

referral for DVT

A

urgent if </= 17% probability of DVT with </= 2 CDR
emergency if 75% probability of DVT with >/= 3 CDR

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12
Q

DVT may lead to:

A

pulmonary embolism

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13
Q

cause of pulmonary embolism

A

DVT that moves and lodges into smaller artery supplying lungs

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14
Q

risk factors for PE

A

same as DVT

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15
Q

what is known as “the great masqueraders”

A

PE

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16
Q

S&S of PE

A

respiratory S&S –> SOB, wheezing, rapid breathing
pleuritic chest P! - sudden, sharp and stabbing chest P! exacerbated by:
- deep inspiration
- coughing
- mechanical pain due to lung fascia attaching to ribs and thoracic vertebra (trunk motion, UE motion, thoracic & rib accessory motion testing)
blood with cough
painful breathing at rest
fainting
tachycardia and palpitations

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17
Q

referral for PE per CDR

A

urgent if < 2/6
emergency if ≥ 2/6

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18
Q

what is PAD

A

ischemia leading to symptoms in the most distal area from the blocked artery most often the calf

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19
Q

PAD most often found in the ____

A

LEs

20
Q

risk factors of PAD

A

≥ 45 years
family hx of MI or sudden death before 55
recent or current smoker
physical inactivity
metabolic syndrome

21
Q

pathogenesis of PAD

A

atherosclerosis or plaque build up in aa that also promotes vascular constriction thus further limiting circulation
symptoms begin once 50% of a. is narrowed

22
Q

S&S of PAD

A

intermittent claudication
- LE P! most often the calf, with a similar amount of activity and elevated positions
- bilateral or unilateral
- relieved with rest and dependent positions
- cramping, weakness, pressure or aching

23
Q

S&S distal to ischemic area of PAD

A

loss of pulses
TTP
muscle atrophy and weakness
loss of hair
cool and bluish skin
bruit on auscultation
possible necrosis/wound

24
Q

in the presence of severe ischemia, P! may also occur ____

A

at rest and create sleep interruptions

25
Q

what test should you perform with PAD to assess the post. tibial/brachial systolic BP in all 4 extremities
** invalid with HTN

A

ankle brachial index cuff test in supine

26
Q

what is an abnormal ABI result

A

< .9 (the lower the worse dz)
>/= 1.4 (poorly compressed veins due to hardened artery from atherosclerosis)

27
Q

how could you differentiate calf pain due to PAD vs stenosis

A

bicycle test (lean forward goes away = stenosis)

28
Q

how do you differentiate DVT vs PAD

A

DVT which is more painful in a dependent position and relieved with elevation

29
Q

referral of PAD

A

urgent to vascular MD

30
Q

what is osteochondritis dissecans

A

damage to subchondral bone

31
Q

incidence/prevalence of osteochondritis dissecans

A

rare
younger boys through early adulthood
males
medial femoral condyle and talus

32
Q

etiology of osteochondritis dissecans

A

mostly unknown
joint rotational or shearing trauma like a sprain

33
Q

pathogenesis of osteochondritis dissecans

A

ischemia (AVN) then separation of subchondral bone from convex and weightbearing end bones
overlying articular cartilage can remain visible

34
Q

S&S of osteochondritis dissecans

A

may be asymptomatic with incidental imaging
if symptomatic, persistent pain not progressing as expected
may progress into severe pain if fragment displaces with joint locking, catching and swelling

35
Q

S&S of osteochondritis dissecans: hypermobility of involved ligament but with persistent ARJC
- ROM:
- resisted:
- stress tests:
- palpation:

A
  • limited and painful, part. w ext
  • may be weak and painful, part. at end range ext
  • compression likely (+)
  • TTP over femoral condyle
36
Q

referral of osteochondritis dissecans

A

urgent to MD

37
Q

osteochondritis dissecans may take 2-3 years to revascularize and heal due to the following:

A

higher BMI
deficient passive restraints
muscle imbalances
impaired proprioception

38
Q

PT implications for osteochondritis dissecans

A

protection to avoid separation of subchondral bone and articular cartilage into the joint (joint mouse)
non-operative management for stable lesion
surgery for resurfacing or fixation needed for unstable lesion

39
Q

what is reactive arthritis aka Reiters syndrome

A

acute, infection at site remote from the primary infection (spreads unlike septic arthritis)

40
Q

risk factors of reactive arthritis

A

IV drug users
high sexual activity
infrequent pelvic examinations
weaker immune system

41
Q

incidence/prevalence of reactive arthritis

A

targets larger joints in the LE and primarily knee and ankle

42
Q

etiology of reactive arthritis

A

most commonly from respiratory infection
may occur from GI, genitourinary, and colon infections

43
Q

pathogenesis of reactive arthritis

A

bacteria stimulates antibody and protein factor production that creates inflammation and tissue damage leading to an arthritic joint

44
Q

S&S of reactive arthritis

A

begin 1-4 weeks after reccent infection
infection S&S
autoimmune S&S
may progress to incapacitating illness

45
Q

PT implications of reactive arthritis
- observation:
- vitals:
- scan/biomechanical exam:

A
  • redness, swelling
  • temp
  • like for ARJC
    TTP and warmth
    swollen and tender lymph nodes
46
Q

referral for reactive arthritis

A

urgent to MD