Path Lab 2 Flashcards
Puppy about 4 weeks old
Was not doing well, lethargic
Small breed puppy
Rectovaginal fistula
Atresia Ani with a rectovaginal fistula - you can not see the anus here
The opening here of the vulva looks larger than normal
Absence of communication between the …?
Manual compression of distal colon leads to the elimination of fecal contents through vagina
Usually need to be euthanized after birth
Females can last for a little bit longer after birth
A probe inserted in the vagina demonstrates a communication between the vagina and the rectum. The rectum has been opened with scissors. This confirmed the diagnosis of Rectovaginal fistula
What is the species and tissue?
This is part of the small intestine
Horse
Piece of the ileum; relatively common condition in horses. The terminal ileum looks different (circle on left). The mucosa is much smaller –> intestinal stenosis (narrowing). The wall of the intestine looks hypertrophic (thickening) in both the internal and outer layer.
Dx: Ileal hypertrophy
Some horses develop no clinical signs, in others it can cause colic b/c this degree of stenosis –> ileal impaction –> mild/severe cases of colic.
Etiologic Dx: Idiopathic
Full Dx: Idiopathic Ileal Hypertrophy
Tissue from maxillary region of a horse.
What abnormalities are present?
Left side:
Right side: impacted feed (e.g. hay) at the root of the tooth, caseous, suppurative exudate (neutrophils present) in the sinuses –> chronic suppurative necrotizing sinusitis. This is a tooth root abscess –> destroys bone (tooth infection can lead to sinusitis).
Jejunum from a young goat with a history of diarrhea, AVC case.
What is the Morphologic and Etiologic diagnosis?
Chronic or Acute?
Inflammatory or Chronic ?
Morphologic Dx: Multifocal proliferative necrotizing enteritis Or proliferative enteritis Or Hyperplastic Enteritis
Etiologic Dx: Coccidiosis, Parasitic Enteritis
Coccidial infections in intestine usually produce these multifocal lesions. Areas where you have infestation of the epithelial cells of the intestine by the coccidial organisms –> significant proliferation as a compensatory measure.
Etiologic agent?
Etiologic Dx?
Name of the Disease?
Etiologic agent: Eimeria sp.
Etiologic Dx: Coccidial Enteritis OR Parasitic Enteritis OR Protozoal Enteritis
Name of the Disease: Coccidiosis
4 weeks old
Lethargy, diarrhea, anorexia
What is the etiologic dx?
Morphologic Dx?
Pale GI
Edematous; fluid in peritoneal cavity; Subcutis looks gelatinous; could also be hypoproteinemia; subcu fluids were administered to this puppy, so that could be the reason for why it looks “wet”
Parasitic Enteritis, Toxocara Canis aka Whipworm (bigger than hook and whip worm)
Microscopically you would see a lot of Eosinophils due to the parasite present.
Morphologic Dx: Catara = mucosuppurative + enteritis
Parasites migrating towards the stomach after death.
Anaprocephala Perfoliata
Found in the cecum or the colon
Most the time you do not see any clinical signs
31 year old horse.
cranial portion: scolex –> allows it to attach to the mucosa
The lesions that they produce, even if they are not significant, –> focal areas of erosion or ulceration.
Similar to gastrophilus
Horse presented to you with clinical signs of severe colic
Tried to treat pain, nothing was working.
Signs of toxemia according to clinicians –> septic shock
Morphologic Dx: Acute Necrohemorrhagic Colitis
Intusuception
Cecum on right
Cecum into colon –> Cecocolonic Intususception
Physiologic Process: Venous infarction
Diarrhea, mucosy
Key in parvo
Anemia
Pale occular mucous membranes
