Pancreatitis Flashcards

1
Q

How is acute pancreatitis defined

A

Premature activation of excessive pancreatic enzymes that destroy pancreatic cells, resulting in autodigestion & fibrosis of pancreas.

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2
Q

acute pancreatitis can range from what

A

mild edema to severe hemorrhagic necrosis

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3
Q

what are the 2 most common causes of acute pancreatitis

A

Gallstones and ETOH

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4
Q

What is the best imaging for pancreatitis

A

CT scan

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5
Q

What characteristics does a CT scan show in pancreatitis

A

diameter,
calcifications,
pancreatic cysts
pseudocysts

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6
Q

2 complications of acute pancreatitis

A

Pseudocyst – cavity (filled w/ necrotic products) surrounding outside of pancreas (resolves spontaneously or perforates into peritoneum)

Abscess – large fluid-containing cavity within pancreas (results in extensive necrosis of pancreas); need prompt surgical drainage

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7
Q

Acute pancreatitis interventions to RELIEVE PAIN

A

IV morphine

Assume positions that flex the trunk (less stretch on peritoneum)

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8
Q

Acute pancreatitis interventions to maintain F&E balance

A

Monitor VS frequently (can be labile)

Monitor for fluid imbalances & electrolyte imbalances

Aggressive IV hydration

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9
Q

Acute pancreatitis interventions to rest/suppress pancreatic enzyme stimulation

A

NPO; meticulous oral care

NG to Low Wall Suction

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10
Q

What do we monitor the stool for in acute pancreatitis

A

steatorrhea which means the body has Impaired protein/fat metabolism. Stool will appear oily & float.

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11
Q

What health promotion do we want to encourage

A

stop ETOH intake

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12
Q

Diet teaching when patient is no longer NPO with acute pancreatitis

A

Diet teaching – CHO encouraged (less stimulating to pancreas); fat restriction

Need fluids!!!

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13
Q

Define Chronic Pancreatitis

A

Progressive, destructive – w/ remissions & flares; caused by inflammation & fibrosis of tissue

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14
Q

What are the Clinical Manifestations of Chronic Pancreatitis

A
Intense abdominal pain (tenderness less than when acute)
	Mass? – suspect pseudocyst or abscess
	Ascites
	Respiratory compromise
	Steatorrhea
	Dark urine
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15
Q

Do we use pain control in Chronic pancreatitis

A

Yes – opioids used;

however, to be used cautiously (some opioids might increase pain by causing spasms).

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16
Q

Describe Steatorrhea

A
oily
bulky
floats
greasy
malodorous
17
Q

What is Pancreatic-enzyme replacement therapy (PERT):

A

Standard of care to prevent malnutrition, malabsorption, & excessive weight loss.

18
Q

what is contained in pancrelipase

A

amylase
lipase
protease

19
Q

pancrelipase is given during what

A

What is Pancreatic-enzyme replacement therapy (PERT):

20
Q

What needs to be recorded during Pancreatic-enzyme replacement therapy (PERT):

A

Record number & consistency of stools per day to monitor effectiveness of enzyme therapy.

21
Q

What is the Goal of PERT

A

Goal = less frequent & less fatty stools

22
Q

What are the teaching points of PERT enzyme replacement

A

Take pancreatic enzymes before or with meals and snacks.
Sometimes ordered to administer with antacid or H2 blockers; (because a decreased pH inactivates drug).
Tell the patient to swallow the tablets without chewing to minimize oral irritation.
Avoid lip/skin contact with enzymes. (Wipe lips prn after ingesting.)
Mix the powder form in applesauce or fruit juice at patient’s request.
Do not mix enzyme preparations in protein-containing foods.
Do not crush enteric-coated preparations.

23
Q

What are some considerations with patient weight in chronic pancreatitis

A

Weight loss can be significant:

Sometimes a candidate for TPN

If taking PO, may need up to 4000 to 6000 calories/day to maintain weight.

24
Q

What do patients need to avoid to prevent exacerbation of chronic pancreatitis

A

Avoid things that make your symptoms worse, such as drinking caffeinated beverages.

Avoid alcohol ingestion; refer to self-help group for assistance.

Avoid nicotine.

25
How do patients need to manage nutrition to prevent exacerbation of chronic pancreatitis
Eat bland, low-fat, high-protein, high carbohydrate meals; avoid gastric stimulants, such as spices. Eat small meals and snacks high in calories.
26
what do patients need to consider with medications in Prevention of Exacerbation of Chronic Pancreatitis
Take the pancreatic enzymes that have been prescribed for you with meals.
27
Should patients exercise heavily to prevent exacerbation of chronic pancreatitis
No, they should Rest frequently; restrict your activity to one floor until you regain your strength. 
28
T/F Hyperglycemia should be monitored in acute pancreatitis
True, due to impact to the exocrine function
29
Why is there low serum calcium in acute pancreatitis
Happens with fat necrosis
30
why is there an increase in serum triglycerides in acute pancreatitis
due to fat necrosis
31
why is there an increase in amylase and lipase in acute pancreatitis
pancreatic cell injury
32
why do we see Hypovolemia/tachycardia in acute pancreatitis
Due to plasma vol being lost as inflammatory mediators released into circulation increase vascular permeability and dilate vessels
33
why do we see cullen's and turner's sign in acute pancreatitis
pancreatic enzyme leakage into cutaneous tissue
34
why do we see paralytic ileus in acute pancreatitis
related peritoneal irritaion causes intestinal motility to slow down/stop
35
why do we see jaundice in acute pancreatitis
hepatobiliary obstructive process (elevated bilirubin)
36
why do we see low-grade fever/ leukocytosis in acute pancreatitis
inflammatory process
37
why do we see N/V in acute pancreatitis
associated with any pain originating in viscera
38
why do we see pain in acute pancreatitis
due to distention of pancreas, peritoneal irritation and related inflammation
39
Clinical manifestations in acute pancreatitis
pain N/V low grade fever/ leukocytosis jaundice paralytic ileus cullen's & turner's sign hypovolemia/tachycardia increase serum amylase & lipase increase serum triglycerides decrease in serum calcium