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BDS4 > Paeds Anomalies > Flashcards

Paeds Anomalies Flashcards

1
Q

Least likely teeth to be missing as a result of hypodontia

A

6s
U1s

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2
Q

Conditions associated with hypodontia

A

Ectodermal dysplasia
Down Syndrome
Cleft palate
Hurler’s syndrome
Incontinentia pigmentii

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3
Q

What’s a problem that can arise when upper lateral incisors are missing

A

over-eruption of lower canines

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4
Q

Order of dental management of paeds hypodontia

A

Diagnosis
Removable pros
Ortho
Composite build ups
Porcelain veneers
Crowns and bridges
Preventative tx

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5
Q

How to treat abnormal shape/form of tooth due to hypodontia

A

overdenture

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6
Q

how to treat spacing due to hypodontia

A

partial denture

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7
Q

how to treat submergence due to hypodontia

A

composite

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8
Q

how to treat deep overbite due to hypodontia

A

porcelain veneers

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9
Q

how to treat reduced LFH due to hypodontia

A

fixed prostheses

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10
Q

risk factors of supernumary

A

Male 2:1
Japanese
maxilla
cleidocranial dysplasia

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11
Q

Root anomalies

A

short root
radiotherapy
dentine dysplasias
accessory roots

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12
Q

Types of amelogenesis imperfecta

A

hypoplastic
hypocalcified
hypomaturational
mixed with taurodontism

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13
Q

cause of environmental enamel hypoplasia

A

systemic
nutritional
metabolic e.g. Rhesus incompatibility, liver disease
infection e.g. measles

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14
Q

cause of localised enamel hypoplasia

A

trauma
infection of primary tooth

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15
Q

How to treat fluorosis

A

Microabrasion
Vital bleaching
veneers

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16
Q

prenatal causes of generalized enamel defects

A

congenital syphilis/cardiac and kidney disease
Fluoride
Thalidomide

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17
Q

neonatal causes of generalized environmental enamel defects

A

prematurity
meningitis

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18
Q

postnatal causes of generalized enamel defects

A

chickenpox
measles
otitis media
pneumonia

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19
Q

How to diagnose amelogensis imperfecta

A

FH
generally affects both dentitions/all teeth
tooth size, structure, colour
radiographs

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20
Q

Gene mutations in amelogenesis imperfecta

A

enamel extracellular matrix molecules amelogenin, enamelin, kallikrein 4

21
Q

what is hypoplastic amelogenesis imperfecta

A

enamel crystals do not grow to correct length

22
Q

what is hypomineralised/calcified AI

A

crystallites fail to grow in thickness and width

23
Q

What is hypomaturational AI

A

enamel crystals grow incompletely in thickness or width but to normal length with incomplete mineralisation

24
Q

Problems with AI

A

anterior open bite
caries
delayed eruption
poor aesthetic/OH
sensitivity

25
Q

AI Tx

A

composite veneers/comp wash
fissure sealants
metal onlays
ortho
preventative therapy
SS crowns

26
Q

3 types of dentinogenesis imperfecta

A

1- osteogenesis imperfecta
2- autosomal dominant
3- brandywine

27
Q

what is dentine dysplasia

A

normal crown morphology
amber radiolucency
pulpal obliteration
short constricted roots

28
Q

what is odontodysplasia

A

localised arrest in tooth development
thin layers of enamel and dentine
large pulp chambers
ghost teeth

29
Q

How to diagnose DI

A

appearance
associated osteogenesis imperfecta
both dentitions affected
enamel loss
FH
radiography: bulbous crown, pulp obliteration

30
Q

DI problems

A

aesthetics
caries
spontaneous abscess

31
Q

DI solutions

A

comp veneers
overdentures
prevention
removable pros
SS crowns

32
Q

hereditary dentine defects limited to dentine only

A

DI type 2
dentine dysplasia type 1+2
fibrous dysplasia of dentine

33
Q

hereditary dentine defects associated with general disorder

A

brachio-skeletal genital syndrome
ehlers-danlos syndrome
hypophosphatasia
osteogenesis imperfecta
rickets

34
Q

Overview of dental management of tooth defects

A

continuous dental care
management of growth and development
removable pros
crowns and bridges
interceptive ortho

35
Q

cementum anomalies: what is cleidocranial dysplasia

A

hypoplasia of cellular component of cementum

36
Q

cementum anomalies: what is hypophophatasia

A

hypoplasia or aplasia of cementum
early loss of primary teeth

37
Q

Eruption anomalies- premature eruption aetiology

A

High birth weight
Early puberty
Natal/neonatal teeth

38
Q

Eruption anomalies- delayed eruption aetiology

A

Low birth weight
Malnutrition
Downs syndrome
Gingival hyperplasia

39
Q

Eruption anonalies- premature exfoliation aetiology

A

Trauma
Following pulpotomy
Hypophosphatasia
Immune deficiency

40
Q

Eruption anomalies- delayed exfoliation aetiology

A

Infra-occlusion
Hypodontia
Ectopic successors
Post-trauma
Supernumaries
Early loss/retained primary
Crowding/dilacerated tooth

41
Q

MIH- pre-natal aetiology

A

Mother’s general health in 3rd trimester
Ask if pre-eclampsia/gestational diabetes

42
Q

MIH post-natal aetiology

A

Childhood infection-chickenpox,measles,rubella
Prolonged breast feeding/SES/medications

43
Q

MIH problems

A

Secondaries caries
Aesthetics/sensitivity
Toothwear
Discolouration

44
Q

MIH management of molars

A

Restorations
Consider XLA
SSC

45
Q

MIH- management of incisors

A

Microabrasion
Resin infiltration
Composite buildup
Composite veneers

46
Q

Why is MIH sensitive

A

Increased nerve and increased vascularity and increased immune cells

47
Q

What is MIH

A

Enamel hypomineralisation of systemic origin of first permanent molars, and frequently associated with incisors

48
Q

When can u extract 1st permanent molar

A

Pt between 8-10
Bifurcation of 7s
Calcification of 8s

BDS4 (50 decks)

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