Orthopedics For Kids Flashcards

1
Q

Legg-Calve-Perthes disease

A
●Idiopathic avascular necrosis of the epiphysis of the femoral head
●2-4 year progression and almost
  always heals itself
●Usually occurs b/w 3 & 12 yo
●Most common in boys 5-7 yo
●4 times more common in boys
●Girls often develop it later and more
  severe
●Bilateral 20% of cases
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2
Q

Legg Calve Perthes Disease cause

A

Unknown etiology

Trauma-> temporary blood loss to femoral head
Maternal smoking in utero

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3
Q

Legg Calve Perthes disease presentation & subjective complaint

A

●Present with mild pain at hip/thigh/knee with activity, but subsides with rest
●Progresses to limp
• Trendelenberg gait pattern

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4
Q

Treatment of Legg Calve Perthes Disease

A

-Gait training
• assistive device as needed
-Treatment controversial
•1° goal of treatment: avoid severe degenerative arthritis and encourage proper formation of femoral head.
*conservative: observation, ROM, w/c to decrease WBing, Petrie casts (best positioning of hip is abd/ER).

  • Bracing with Petrie casts (long leg casts with bar holding legs in ER)
  • Surgery: triple osteotomy
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5
Q

Slipped Capital Femoral Epiphysis

A
●Most commonly diagnosed in 10-15 yo
●Growth plate abnormality
●More common in African-American pop than Caucasians
●More common in males than females
●Surgery
•spica cast
•maintain ROM elsewhere
•Gait training
•WC
•Home assessment
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6
Q

Classification of SCFE

A
  • Grade I: displacement of epiphysis less than 30% of width of femoral neck
  • Grade II: slip between 30%-60%
  • Grade III: includes slips of greater than 60% the width of neck
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7
Q

How can you tell the difference between SCFE and LCP?

A
  • Similar in presentation
  • LCP usually younger
  • SCFE usually more pain
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8
Q

Osgood-Schlatter Syndrome

A
  • Traction apophysitis (inflammation of the tendon insertion) of the patellar tendon aggravated by activity
  • Osteochondrosis- degeneration followed by reossification of one or more ossification centers in children
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9
Q

Prevalence of Osgood-Schlatter’s Syndrome

A
  • Common in the 8-15 year range
  • More common in males
  • One of the most common causes of knee pain in adolescents
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10
Q

Treatment of Osgood-Schlatter’s Syndrome

A
●Rest, ice, decrease activity
●Modalities
•Estim to reduce inflammation
●Assess for:
•Patella tracking problems
•Patella mobility
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11
Q

Where can Apophysitis occur?

A

In addition to tibial tubercle apophysitis (Osgood-Schlatter’s disease), traction apophysitis may occur at the calcaneus, navicular and, rarely, the hip, most often as a result of repetitive trauma/overuse.

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12
Q

Prevalence of Developmental Dysplasia of the Hip (DDH)/ Congenital dislocation of the hip (CDH)/Infantile hip dislocation

A
  • More common in female
  • Most are unilateral
  • can be due to intrauterine positioning
  • can accompany torticollis
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13
Q

Developmental Dysplasia of the Hip (DDH)/ Congenital dislocation of the hip (CDH)/Infantile hip dislocation DIAGNOSIS AND TREATMENT

A

Barlow maneuver & Ortolani maneuver

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14
Q

Barlow maneuver

A

•Barlow maneuver (to determine if hip will dislocate):
●This is part of the standard infant exam performed until 8–12 weeks of age.
●Hip is adducted and gently pushed posteriorly.
●“Clunk” is heard (+)…the less pronounced the clunk, the more poorly formed the acetabulum.

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15
Q

Ortolani maneuver

A

•Ortolani maneuver (for posterior dislocation):
●passive flexion, keep flexion and abduct, then bring hip into neutral/extension.
●feel a click/pop (hip is reducing)

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16
Q

If you have a baby younger than 6 months and you want to immobilize their hips into abduction, what do you use?

A

Pavlik Harness

17
Q

If you have a baby older than 6 months and you want to immobilize their hips into abduction, what do you use?

A

Spica cast

18
Q

What is the number 1 cause of brachial plexus injuries in babies?

A

Ninety percent of brachial plexus injuries in children are caused by a traumatic stretching of the plexus during birth

19
Q

What percent of babies will recover without surgery after a brachial plexus injury?

A

80%!

20
Q

What is the incidence of brachial plexus injury?

A

1-2/1000 births

21
Q

Brachial Plexus Avulsion

A

Nerve is torn from the spine

Most severe

22
Q

Brachial Plexus rupture

A

Nerve is torn but not at the spinal attachment

23
Q

Brachial Plexus Neuroma

A

The nerve has tried to heal itself but scar tissue has grown around the injury, putting pressure on the injured nerve and preventing the nerve from conducting signals to the muscles

24
Q

Brachial Plexus Neuropraxia

A

Stretch
The nerve has been damaged but not torn
most common

25
Q

Erb’s Palsy

A

●Paralysis involving C5 & C6 and sometimes C7.
• the arm is turned towards the body
•the elbow does not bend and the hand is in a “waiters tip” (turning backwards) position.

26
Q

Klumpke’s palsy

A

●Paralysis involving C7, C8 & T1.
•the hand is limp
• fingers do not move
• there is often an associated Horner’s Syndrome (Horners Syndrome is when the eyelid droops, the cheek does not sweat and the pupil is smaller than the unaffected eye).

27
Q

Brachial Plexus Injury Treatment

A
•ROM (passive and active)
•Electrical stimulation
**Somewhat controversial on infants
•Parental education
•Sensory input
•Increase awareness of extremity
•Positioning
•Splinting
28
Q

Talipes Equinovarus/Club Foot

A
  • a congenital deformity seen in newborn children
  • the feet to point down and inward
  • does not cause pain in the newborn child
29
Q

What are some of the future concerns with Club Foot/ Talipes Equinovarus? What is the prognosis?

A

• can cause long-term abnormalities in gait
•may lead to complications such as chronic skin ulcers.
•If properly treated, the deformity can resolve in early childhood.
**however this will not correct for differences in foot and calf size

30
Q

How will an injury to the whole brachial plexus present?

A

Erb’s-Klumpke paralysis

31
Q

Torticollis prevalence

A

1/300 live births

32
Q

Torticollis cause

A

Intrauterine positioning
Extrauterine positioning
Trauma, structural (tumor)
Gastro esophageal reflux

33
Q

In torticollis, deformation of the infant’s skull due to:

A

normal brain growth combined with an asymmetric pre and postnatal resting position

34
Q

In torticollis, shortening of the SCM results in:

A

Lateral flexion of the cervical spine to one side
asymmetric pre and postnatal resting position
with cervical rotation toward the opposite side

35
Q

What are come of the things that may accompany torticollis depending on its severity and presentation?

A

○ Plagiocephaly (“oblique head”): describes
asymmetry of the head shape
○ Often, a flattening of the head on the
preferred side.
○ “Bald spot” may be present on the
latero-posterior aspect of the preferred side of rotation.
○ Facial asymmetry is often present (muscle bulk is usually smaller on the side of with shortened SCM. )
○ Palpable knot/cord may be present in the SCM

36
Q

What are some of the associated conditions that correlate with torticollis?

A
○ Changes in facial features 
○ Hip dislocation or dysplasia 
○ Equinovarus deformity (club
foot) 
○ Gastroesophageal reflux
37
Q

How does one even begin to treat torticollis?!

A

○ Stretching
● until you feel a pull or at baby’s discomfort
○ Massage
● SCM
○ Positioning
● Holding/Feeding (parents should switch sides)
● Presenting objects to the child during
interaction/play
● Environmental modifications
● Helmet therapy to reshape the infant’s skull
○ Typically started around 5-6 months, earlier in more severe cases
○ Surgery
○ Parent/Caregiver training