Orthopedic & Musculoskeletal Flashcards
Cervical spine ROM
Flexion 0-45
Extension 0-45
Lateral flexion 0-45
Rotation 0-60
Thoracic/lumbar ROM
Flexion 0-80
Extension 0-30
Lateral flexion 0-40
Rotation 0-45
Shoulder ROM
Flexion 0-180
Extension 0-60
AB 0-180
Horizontal AD 0-145
Horizontal AB 0-45
IR 0-70
ER 0-90
Wrist ROM
Flexion 0-80
Extension 0-70
Ulnar deviation 0-30
Radial deviation 0-20
Elbow/forearm ROM
Flexion 0-150
Supination/pronation 0-80
Hip ROM
Flexion 0-120
Extension 0-30
AB 0-40
IR/ER 0-45
Thumb ROM
MP flexion 0-50
IP flexion 0-80
AB 0-50
Finger ROM
MP flexion 0-90
MP hyperextension 0-45
PIP flexion 0-110
DIP flexion 0-80
AB 0-25
Knee ROM
Flexion 0-135
Foot/ankle ROM
Plantar flexion 0-50
Dorsiflexion 0-15
Inversion 0-35
Eversion 0-20
FWN
No limits or restrictions
WBAT
Pain to guide tolerance
PWB
30-50%
TDWB
10-15%
NWB
0%
Isometric contraction
No movement or change in angle (static holds, contract/relax)
Isotonic contraction
Movement over joint with change in angle
- Eccentric
- Concentric
Eccentric
Muscle lengthening (down)
- Elbow extension
- Stand to sit
Wheelchair consideration for bilateral LE amputation
Large rear facing wheels with anti-tippers for residual limb support/ counter balance missing limbs
Terminal device
UE: hand
LE: foot
Passive TD
realistic and non functional worn for cosmetic purposes
Active TD
Body-powered, externally powered through electrical signal or hybrid powered
Prosthetic wearing schedule
- Initially have patient wear for 15 to 30 minutes
- Remove to check for redness
- If no redness apparent post wear increase time by 15-30
min intervals until prosthetic is worn for entire day - If redness does not disappear after 20 minutes report to
prosthetist
Symptoms of fibromyalgia
Widespread pain, poor sleep, fatigue, inability to think clearly, joint swelling
Fibromyalgia diagnosis
of painful areas
Severity of fatigue
Presence of cognitive problems
Symptoms last for 3+ months
Symptoms not explained by other health issues
Common evaluation for fibromyalgia
- Daily activity log
- COPM
- Pain assessment
Treatment for fibromyalgia
Trigger management
Stress management
Myofascial release & trigger point massage
Daily stretching and light aerobic exercise
Progressive strengthening
Sleep hygiene
Energy conservation
OA
Noninflammatory condition resulting in breakdown of articular cartilage and reduced joint space causing painful bon-to-bone contact
Types of OA
Primary: localized or generalized joint involvement with no known cause
Secondary: related to trauma, infection or congenital abnormalities
Bouchard’s nodes
PIP bony overgrowths due to OA
Herberden’s nodes
DIP bony overgrowths due to OA
Surgical treatment options for OA
- Arthroscopic debridement to remove broken cartilage to reduce pain and improve movement
- Resection or perforation of subchondral bone to stimulate formation of cartilage
- Graft to replace damaged cartilage
- Joint fusion
- Arthroplasty (knee, hip replacement)
Kyphosis
Abnormal rounding outward of upper back in thoracic region
Lordosis
Abnormal rounding inwards of lower back in lumbar region
Kyphosis impact on ADL
Deficits in eating including manipulation and swallowing
Osteoprosis
Progressive low bone density that leads to bone fragility and frequent fx in weight bearing bones
Risk factors of osteoporosis
Osteopenia, inadequate calcium intake, estrogen deficiency, sedentary lifestyle, alcoholism, diabetes, RA medications, malnutrition, post menopausal, women
Treatment for osteoprosis
- Anti-resorptive medications to slow bone loss
- Hormone related therapy
- Calcium supplement with vitamin D to assist with absorption
- Exercise and reduced smoking, alcohol, caffeine intake
Presentation of RA
Polyarticular
RA
Systematic and inflammatory condition with resulting in progressive synovitis of diarthrodial joints due to excessive synovial build up and joint deformities due to uneven distribution of weakened tendons and ligaments
Age group for juvenile RA
1-6
Common RA deformities
Boutonniere
Swan neck
Mallet
Ulnar deviation
Subluxation
Joint fusion
Extensor tendon rupture
Trigger fingers
Mutilans deformity
Subcutaneous nodules
Claw toe
Hammer toe
Cock-up toe
Hallux valgus (bunion)
Boutonniere deformity
PIP flexion DIP hyper-extnesion
Swan-neck deformity
PIP hyper-extenion DIP flexion
Mallet figner
DIP flexion
Ulnar drift
MCP ulnar deviation, wrist radial deviation
Mutilans defomirty
Very floppy joints with shortened bones and redundant skin
Claw toe
Hyperextension of MTP and flexion of IP
Hammer toe
Hyperextension of MTP and PIP and DIP flexion
Early phase RA
No destructive change on x-ray
Possible osteoporosis
Moderate phase RA
Radiographic evidence of osteoporosis
Slight cartilage and bone destruction
No joint deformity
Possible limitations with ROM
Muscle atrophy
Severe phase RA
Cartilage and bone destruction
Joint deformity
Expensive atrophy
Possible soft-tissue lesions
Terminal phase RA
Fibrous or bony ankylosis in addition to stage 3
Neuroma
Ball of nerve tissue when axons attempt to regrow in distal limb that is painful with pressure on residual limb