orofacial 2, dental caries, perio Flashcards

1
Q

fascial space infections can come from

A
  • pericoronitis

- bacterial sialadenitis

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2
Q

two things that can result from fascial space infection

A
  • ludwig’s angina

- cavernous sinus thrombosis

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3
Q

dentoalveolar abscess can lead to

A
  • osteomyelitis
  • dry socket
  • fascial space infection
  • endocarditis
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4
Q

what can cause dentoalveolar abscess

A
  • periodontal abscess

- endodontic infection

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5
Q

periodontitis is associated with what systemic disease

A

endocarditis

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6
Q

dry socket usually occurs in which teeth

A

lower 2nd and 3rd molars

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7
Q

fascial space infections usually occur in what region

A

submandibular

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8
Q

pericoronitis

A

infected flap over tissue over 3rd molar (DO surface)

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9
Q

bacteria can spread along

A

muscle attachments

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10
Q

how do bacteria spread along muscle attachments

A

-produce hyaluronase and other matrix destructive enzymes

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11
Q

where do the bacteria target the muscles ad which teeth are effected

A

sublingual area

mandibular 2nd and 3rd molars due to

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12
Q

deep fascial space infections usually come from which teeth

A

lower 2nd and 3rd molars

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13
Q

symptoms of space infection

A

board like hardness

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14
Q

which bacteria cause space infections

A
  • normal oral flora (often the strep bacteria)

- anaerobic oral bacteria may be present

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15
Q

which Abx will you give for fascial space infection

A
  • penicillin (to kill the strep)

- clindamycin if they dont get better with pen (bc anaerobic) OR metronidazole +pen

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16
Q

dentoalveolar infections can lead to what (most commonly)

A

maxillary sinusitis

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17
Q

cavernous sinus thrombosis follows the spread of odontogenic infection along 2 pathways:

A
  • anterior facial vein

- pterygoid plexus of veins

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18
Q

symptoms of cavernous sinus thrombosis

A
  • swelling of eyelids
  • protrusion of eyeball
  • restricted movement of eyeball
  • bleeding in retina
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19
Q

ludwig’s angina description

A
  • swelling of head and neck spaces

- difficulty breathing

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20
Q

main cause of ludwig’s angina

A

cellulitis

*50-90% of cases were due to dental cource of infetion

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21
Q

teeth most commonly involved with ludwig’s angina

A

mandibular 2nd and 3rd molars

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22
Q

treatment for ludwig’s angina

A
  • parenteral abx
  • airway monitoring
  • i and d
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23
Q

non odontogenic infections of oral cavity

A
  • pericoronitis
  • bacterial sialadenitis
  • angular cheilitis
  • ulcerative mucositis
  • noma (gangrenous stomatitis)
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24
Q

pericoronitis

A
  • infection of flap of gum tissue
  • usually 3rd molars
  • pts usually older teens and early 20s
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25
Q

bacterial sialadenitis

A

stone in the stensons duct (parotid duct)

26
Q

angular cheilitis

A

C albicans and s aureus at edge of lips

27
Q

ulcerative mucositis

A
  • chemotherapy associated

- bacteria and yeasts

28
Q

noma (gangrnous stomatitis)

A
  • polymicrobial

- opportunistic in immunocompromised

29
Q

bacteria that cause pericoronitis

A
  • p intermedia
  • anaerobic strep
  • fusobacterium
  • A A
  • tannerella forsythia
30
Q

pericoronitis can lead to

A

spread of infection along facial plane (if drainage is interrupted)

31
Q

main cause of bacterial sialadenitis

A

s aureus (post surgery)

32
Q

abx for bacterial sialadenitis

A

pen
erythromycin
metro

33
Q

patients that usually get bacterial sialadenitis

A
  • pt with xerostomia
  • age
  • medication
  • parkinsons
34
Q

predisposing factors of angular cheilitis

A
  • low B vit
  • dry mouth
  • overclosure
  • habits or drooling
  • immunosuppression
  • poor fitted dentures
35
Q

ANUG

A
  • sudden onset painful infection of gums

- common in students around march

36
Q

cause of ANUG

A
  • fusobacterium and spirochetes like trep denticola

- stress and immunosuppression

37
Q

abx for ANUG

A

metro

38
Q

bacteria that cause actinomycosis

A
  • opportunistic

- cross tissue planes

39
Q

cervicofacial actinomycosis

A
  • Actinomyces (esp A israelii)
  • rock-hard induration
  • SPONTANOUS DRAINING OF SINUS TRACT
40
Q

once an enamel caries reaches dentin, it

A

fans out and lesion gets bigger

41
Q

what happens to biofilms when they cause disease

A

they become PLANKTONIC!

42
Q

types of study designs

A
  • cross sectional design

- longitudinal design

43
Q

cross sectional study design

A

-compare microflora in health and disease at single point in time

44
Q

longitudinal design

A

-compare in a single patient at different points in time

45
Q

no single microbe has satisfied

A

koch’s postulates for plaque-mediated disease

46
Q

specific plaque hypothesis

A

only few species involed in disease (eg RED organisms)

47
Q

non-specific plaque hypothesis

A

disease is outcome of overall activity of total plaque biome

48
Q

ecological plaque hypothesis

A
  • bacteria found in both healthy and disease

- disease is a shift in balance of resident bacteria due to change in environmental conditions

49
Q

bacteria that cause root surface caries

A
  • strep mutans
  • s sanguinis
  • a naeslundii
50
Q

specific model of enamel demin

A
  • s mutans only

- surface

51
Q

non specific model of enamel demin

A
  • s mutans AND Veillonella

- deep lesions

52
Q

two things that are lost in perio

A
  • collagen in gingiva

- bone

53
Q

dental abscess are __ while perio is ___

A

acute, chronic

54
Q

what happens to MMPs and TIMPs in perio

A
  • activate MMPs

- degrade TIMPs

55
Q

recognition of quorum sensing

A

plaque => biofilm

56
Q

red complex organisms

A

-p gingivalis
-t forsythia
t denticola
-AA

57
Q

gingivitis

A
  • precedes perio

- reversible

58
Q

periodontal disease

A
  • chronic
  • gingivitis but will loss of attachment between root surface, ging, and albeolar bone
  • bone loss
  • irreversible
59
Q

perio disease is a combo of what

A

biofilm (direct) + host immune response (indirect)

60
Q

advanced lesion

A
  • comes after established lesion (red gums)
  • conversion of ging to perio!
  • “leukocte wall” is unique
61
Q

main destruction in perio is due to

A

indirect mechanisms - immune response!