Opthalmology Flashcards
Signalment
Breed
Age - older dog, intraocular neoplasia, cat systemic hypertension
Gender (pyometra uveitis)
History for ophthalmic exam
Vaccination, worming, diet
Other pets
Travel
General health
Medications
Previous eye issues
Uni/bilateral
Duration/progression
Ocular pain, appearance change, vision changes
presenting ocular complaints
ocular pain - blepharospasm, increased tearing
change in appearance - redness, discharge, swelling
decreased vision - day vs night, changes in unfamiliar surroundings
things to examine - ophthalmic
behaviour - do they seem visual/painful etc
symmetry - colour, nostrils, swelling, palpebral fissure size, third eyelid position
external anatomy, palpation, under eyelids, anterior surface, retropulsion
schirmer tear test
ocular reflexes
ocular reflexes
menace - vision
palpebral (before menace to check blink)
dazzle
pupillary light - really bright light
vestibulo-ocular
corneal sensation (if suspect of blinking/trigeminal disorder)
anterior assessment
pen torch +/- magnification
examine outside to inside
eyelids/lashes/nasolacrimal puncta
third eyelid
conjunctiva, sclera, limbus
anterior chamber
iris and pupil
distant direct ophthalmoscopy
essential
0 dioptre at arms length
tapetal reflex to highlight visual axis
compare pupil sizes, opacities
close direct
0 dioptres 2-3cm from eye
dilate pupil with tropicamide
small highly magnified field of vision - hard to examine whole fundus
line up eye, light source and condensing lense, curved side towards you
fluorescein staining
orange dye - turns green in alkaline conditions
stroma turns it green
indicated in most opthalamic assessments but towards end of assessment
strips are preferable to drops
always flush afterwards
stain should appear at nose in 3-5 mins (jones test)
conjunctival swabs/scrapes
sterile swabs
if bacteria suspected - charcoal medium
ocular surface disease - chlamydophila felis, feline herpes virus PCR
tonometry
end of exam
essential for reduced vision
red/blue eyes or corneal oedema
measures IOP 10-25mmHh normal in dog/cat, 15-20mmHg normal in rabbit
raised - suspect glaucoma
lowered - suspect uveitis
difference >8mmHg abnormal
split lamp - specialist
judge depth and localise lesions
electroretinography - specialise
assessment of retinal function
gonioscopy - specialist
assessment of iridocorneal drainage angle
imaging - specialist
ultrasound, ct, mri
corneal pathology
oedema - breach/dysfunctional barrier
vascularisation - promotes healing but increases scarring
pigmentation - non-specific response to corneal insult
wound healing
corneal epithelium self renews - movement up/centrally
proliferation from basal epithelium at limbus
superficial, epithelial only ulcers
cover defects in hours-days with proliferation, migration and adhesion
stromal healing
healing starts after re-epithelialisation
fibroblasts migrate in and lay down new collagen
requires vascularisation and results in scar tissue/remodelling
common causes of corneal ulceration
trauma
tear film production - KCS
adnaxal conditions - eyelids/eyelashes
primary corneal disease (SCCEDs - spontaneous chronic corneal epithelial defect)
infection
neurological disease
types of ulcer
superficial
SCCED
stromal
desmetocoele
infected/melting
perforated
clinical signs of ulceration
pain
conjunctival hyperaemia
ocular discharge
corneal oedema
reflex uveitis
superficial ulcer
epithelial loss only
acute
painful
sharp distinct borders
minimal inflammatory response
+/- reflex uveitis
SCCED - spontaneous chronic corneal epithelial defect
non-healing, indolent ulcer
any breed but boxers and corgis are common
> 7 years old
epithelium loss only, usually unilateral
no stromal involvement
lip of loose epithelium - grows across but cannot adhere to stroma
stromal ulcer
epithelial and stromal loss
acute/chronic
fluorescein staining of walls and floor of ulcer
can be superficial or deep
anterior uveitis is common side effect
visible crater
desmetocoele
acute/chronic
complete stromal ulcer down to descemets membrane
walls of ulcer common bottom does not stain
revealed by flushing
melting corneal ulcers
keratomalacia
present with acute closed painful eye with ++ gloopy discharge
ill defined/ rounded/soft edges - look like melting away
anterior uveitis
can perforate within hours - ophthalmic emergency
pathogensis - enzymes digest stroma - caused by cornea itself or bacterial infection
steroids suppress and potentiate collagenase
treatment of simple superficial
chloramphenicol
nsaids
regular checks
SCCEDs treatment
debridement
keratotomy/keratectomy
chloramphenicol
nsaids
atropine
contact lens as bandage
keratotomy
grid - hash with needle, bevel up parallel to surface
diamond burr
complex corneal ulcer treatment
intensive treatment +/- infection
C+S - enrofloxacin
nsaids +/- opioids
atropine to effect
hospitalise/very close monitoring
melting treatment
apply serum 1-2 hourly for 24h then reduce
n-acetylcysteine (stromease) - new product
feline ulcers
infection/trauma/corneal sequestrum
non-healing - gentle cotton bud debridement, never grid on a cat
traumatic globe prolapse
pathophysiology - immediate oedema
traction on optic nerve can blind
desiccation of ocular surface - potential to ulcerate, dries quickly. rupture of ocular muscles
brachycephalic much more likely to occur but better prognosis, long nose, much less likely but bad prognosis, cats unlikely except RTA
replace or enucleate
treat immediately, dont refer. distressing, painful, prevent self trauma
GA for replacement
lateral canthotomy to make replacement easier
temporary tarsorrhaphy to prevent reprolapse and tamponade haematoma - needle cannot go full thickness as dont want sutures against eye
treat with antibiotics, antiinflammatories and buster collar
most eyes are blind
retrobulbar abscess
cs - acute onset, unilateral, exophthalmos, pain (worse on mouth opening) third eyelid protrusion/swelling, ocular discharge, pyrexia, lethargy
ultrasound for fluid filled cavity
treatment - drain abscess under GA, abscess via mouth and release pus, amoxy clav and nsaids
referral approrpiate
acute glaucoma
ocular pain (blepharospasm, increased lacrimation, photophobia), head shy, yelping, dull/quiet, vision loss, appearance change
emergency in purebreeds with hereditary, terrier breeds with acute lens luxation
diagnose with tonometry often >40mmHg can be 60-80mmHg
treatment - reduce IOP - prostaglandin analogue (latanoprost) is primary, carbonic anhydrase inhibitors (brinzolamide/dorzolamide). analgesia
seek referral advice/offer
anterior lens luxation
cs - acutely painful eye
glaucoma
focal corneal oedema
lens outline can be visible in anterior chamber
if terrier with acutely painful eye - assume luxation until proven otherwise
treatment - referral - couching, analgesia. ongoing management
chemical injury
acid/alkali - immediate loss of epithelium, alkali can be worse
acids - bleach, toilet cleaner, spirit base skin prep
alkali - caustic solution, lime burns, washing detergents
treatment - immediate irrigation - tap water and copious flushing
test pH
eye foreign body
urgent attention needed
most not true emergencies
refer with lots of pain relief
causes of sudden onset blindness
acute glaucoma
acute uveitis
intraocular haemorrhage
retinal detachment
optic neuritis
sudden acquired retinal degeneration
toxicity
intracranial lesion
vision diagnostics
menace
PLR
tracking
obstacle course
ophthalmoscopy