Nueromuscular/Nervous Systems Flashcards
If a pt with a R hemisphere stroke with pusher syndrome, which way would they push
To the left
What is dyspraxia?
incoordination, associated with developmental coordination disorder. Will have trouble maintaining balance in environments with changing surfaces and obstacles such as in a crowded hallway.
The Forebrain, midbrain, and hindbrain are also known as the:
Prosencephalon, mesencephalon, and rhombencephalon
The prosencephalon is divided into which 2 parts, which are further divided into which parts?
Telencephalon: Cerebrum, hippocampus, basal ganglia, amygdala.
Diencephalon: Thalamus, hypo, sub, epi
The mesencephalon is further divided into which 2 parts, which are further divided into:
Tectum: Superior and inferior colliculi
Tegmentum: Cerebral aqueduct, periaqueductal gray, retinacular formation, substantia nigra, red nucleus.
The Rhombencephalon is divided into what 2 parts, which are further divided into what parts?
Metencephalon: Cerebellum, pons
Myelencephalon: Medulla oblongata
Processing of olfaction occurs in which lobe?
Temporal
Inattention occurs if which lobe is damaged?
frontal
Agressive behaviors occur if which lobe is damaged?
Temporal
Dressing apraxia, constructional apraxia, anosognosia occurs if which lobe is damaged? Dominant or non-dominant side?
Parietal, non-dominant, typically R
Judgment of distance occurs in which lobe?
Occipital
Taste processing occurs in which lobe?
Parietal
Learning deficits occur if which lobe is damaged?
Temporal
Agraphia, alexia, agnosia occur if which lobe is damaged? dominant or non-dominant side?
Parietal, dominant, usually L
Facial recognition occurs in which lobe?
temporal
Which lobe provides meaning for objects?
Parietal
Which lobe enables humans to interpret other peoples’ emotions and reactions?
Temporal-rear
What does the calcarine sulcus separate?
Occipital lobe into superior and inferior halves
What does the sylvian fissure separate?
Anterior portion separates the temporal and frontal lobes, posterior separates the the temporal and parietal lobes
what does the sulcus of Rolondo separate?
Also called the central sulcus, separates the frontal and parietal lobes laterally
What gray matter masses compose the basal ganglia?
Caudate, putamen, globus pallidus, substantia nigra, subthalamic nuclei.
The thalamus receives information from all sensory pathways except which tract?
Olfactory
Damage to the thalamus can produce thalamic pain on which side of the body relative to the lesion?
Contralateral
what is the epithalamus involved in?
Contains the pineal gland, melatonis, internal clock, assoc. with limbic system and basal ganglia too.
Main function of the midbrain?
Connects forebrain and hindbrain so large relay area. Reflex center for visual, auditory, and tactile reflexes.
Damage to one side of the cerebellum with produce impairments on which side of the body?
Ipsilateral
Cranial nerves ___ through ___ originate from the pons
V through VIII
Which artery is the most common site of a CVA?
MCA
Occlusion to which cerebral artery results in loss of bowel/bladder?
ACA
Apraxia can occur with occlusion to which cerebral artery?
MCA (because it supplies the basal ganglia)
Hemiballismus occurs if which cerebral artery is occluded?
PCA-subthalamic nucleus
Memory impairment can occur if this cerebral artery is damaged:
PCA-inferior temporal lobe
Contralater sensory loss of face and UE and lesser LE can occur with which cerebral artery occlusion?
MCA
Locked in syndrome can result from an occlusion of which artery?
Vertebral-basilar
Alexia/dyslexia can occur from occlusion on which artery?
PCA
Neglect can occur with occlusion of which artery?
ACA
Thalamic pain syndrome can occur with occlusion to which artery?
PCA
The 2 parts of the brain the ACA supplies
Anterior frontal lobe
Medial surface of the parietal and frontal lobes (LE involvement)
6 parts of the brain the MCA supplies
1) Outer cerebrum
2) Basal ganglia
3) Post/ant internal capsule
4) Putamen
5) Pallidum
5) Lentiform nucleus
6 parts of the brain the PCA supplies
1) Part of the midbrain
2) Subthalamic nucleus
3) Basal nucleus
4) Thalamus
5) Inferior temporal lobe
6) Occipital and occiptoparietal cortices
The 2 most significant impairments with PCA occlusion are:
Thalamic pain syndrome and cortical blindness
Bilateral ACA occlusion will typically produce ___plegia, while bilateral MCA occlusion at the stem will typically produce ____plegia
1) paraplegia
2) hemiplegia
Brudzinki’s sign
Flexion of neck facilitates flexion of the hips and knees-for meningitis
Kernig’s sign
Pain with hip flexion combined with knee extension
What is sun-setting a sign of?
Hydrocephalus-downward deviation of the eyes
Fasciculus cuneatus
Ascending, sensory tract for trunk, neck, and UE proprioception, vibration, 2 point discrimination, and graphesthesia
Fasciculus gracilis
Ascending, sensory tract for trunk and LE proprio, vibration, 2 point discrimination, graphesthesia
Spinocerebellar tract, dorsal
Ascending, sensory, ipsilateral subconscious proprioception, tension in muscles, joint sense, posture of trunk and LE
Spinocerebellar tract, ventral
Ascending, sensory, some fibers cross then recross, ipsilateral proprio, muscle tension, joint sense, posture of the trunk, UE, LE
Spino-olivary tract
Ascending, from cutaneous and proprioceptive organs to the cerebellum
Spinoreticular tract
Afferent, for reticular formation, influences levels of consciousness
Spinotectal tract
Sensory, spinovisual reflexes, assists with eye movement toward stimulus
Spinothalamic tract, anterior
Light tough and pressure
Spinothalamic tract, lateral
pain, temp
Corticospinal tract, anterior
ipsilateral, voluntary, discrete, and skilled movements
Corticospinal tract, lateral
Contralateral, voluntary, movement
Reticulospinal tract
Extrapyramidal, motor, facilitation/inhibition of voluntary and reflex activity
Rubrospinal tract
extrapyramidal, motor, input of gross postural tone, facilitates activity of flexors, inhibits extensors
Tectospinal tract
Extrapyramidal motor, contralateral postural toen, associated with auditory and visual stimuli
Vestibulospinal tract
Extrapyramidal, ipsilateral gross postural adjustments, subsequent to head movements, facilitates extensor muscles, inhibits flexors. Damage results in significant paralysis, hypertonicity, and clasp-knife reaction.
Brown-Sequard’s Syndrome: which tract(s), contra or ipsi symptoms?
Corticospinal tract, spinothalamic tract, dorsal column, so ipsilateral paralysis and loss of vibratory sense, contra loss of pain and temp.
What is the difference between superficial and deep reflexes?
Superficial: A response to stimulation of receptors within the skin. The signal goes all the way to the brain, then has to descend motor tracts, making it a polysynaptic reflex, e.g. Babinski reflex
Deep: Reflex arc involving the spinal or brainstem segment that innervates the specific muscle.
Stereognosis vs Barognosis vs Graphesthesia
Barog: perceive the weight of different objects in hand.
Stereo: Identify an object without sight.
Graph: Identify a number or letter drawn on the skin without visual input.
The sensation of pain in response to a stimulus that would not normally produce pain?
Allodynia
Absence of pain while remaining conscious
Analgesia
Absence of touch sensation
Anesthesia
Constant, relentless, burning that develops after a peripheral nerve injury
Causalgia
Distortion of any of the senses, especially touch
Dysesthesia
Heightened sensation
Hyperesthesia
An extreme exaggerated response to pain
Hyperpathia
A diminished sensation of touch
Hypesthesia
Severe and multiple shock-like pains that radiate from a specific nerve distribution
Neuralgia
Loss of vibration sensation
Pallanesthesia
Abnormal sensations such as tingling, pins and needles or burning
paresthesia
Neurapraxia
No damage, typically a pressure injury, conduction block.
Axonotmesis
Reversible, injury to axon with preservation of endo and epineurium, as well Schwann cells.
neurotmesis
Axon, myelin, and connective tissue are damaged or transected. IRREVERSIBLE. If surgery, may recover, sensory before motor.
ALS affects what in the PNS?
Anterior horn cells
Myesthania gravis affects what in the PNS?
Neuromuscular junction
Fasiculations are present in UMN or LMN Diseases?
LMND
Chorea is typically secondary to damage of the ______
Caudate nucleus, “fidgeting”
Ballism is typically secondary to damage of the ______
subthalamic nucleus, it is a form of Chorea, “flailing”
Dystonia: type of movement, typical diseases, which muscles, etiologies
Sustained muscles contraction that cause abnormal/repetitive movements. CP, Parkinson’s, encephalitis. All muscles, agonists and/or antagonists, often accentuated during volitional movement. Etiologies: genetic, acquired, environmental, secondary to medications.
Athetosis
Slow, twisting/writhing movements, large in amplitude. When brief, they merge with chorea, when sustained, they merge with dystonia, and it is typically associated with spasticity. Common in CP secondary to basal ganglia pathology.
Akinesia, common in which disease(s)
Inability to initiate movement, PD
Asthenia, typically secondary to _____
Generalized weakness, secondary to cerebellar pathology.
Clasp-knife response
Resistance seen during ROM, greatest resistance at initiation of dance that lesson with movement through the range.
Dysmetria
Inability to control ROM and force
Dystonia
Related to athetosis, but more axial muscle involvement
Fasciculation
Muscular twitch that is Caused by random discharge of a LMN
Lead pipe rigidity
Consistent rigidity throughout the ROM
Modified Ashworth Scale: 0 through 4 scale
0: no increase in muscle tone
1: Slight, EROM
1+: Slight, at less than half of the ROM
2: Most of the ROM, but can still move the affected parts.
3: Passive movement difficult
4: Affected parts rigid in extension or flexion.
VOR vs VSR
VOR: Allows for head/eye movement coordination. Supports gaze stabilization when head moving.
VSR: Assits with body stability while the head is moving, as well as coordination of trunk during upright postures.
Peripheral vs Central Vertigo symptoms
Peripheral: Episodic, autonomic symptoms present, usually a precipitating factor.
Central: Autonomic less severe, loss of consciousness can occur, diplopia, hemianopsia, weans, numbness, ataxia, dysarthria
Peripher vs Central Vertigo Etiology (different diseases)
Peripheral: BPPV, Meniere’s, Infection, Trauma/tumor, DM, ETOH.
Central: Meningitis, Migraine, Trauma/tumor, MS
Gaze evoked nystagmus: Definition? Typically indicative of what type of pathology?
Occurs when eyes shift from a primary positions to an alternate position-pt can’t maintain stable gaze. Indicative of CNS pathology, e.g. TBI and MS
Central vs Peripheral Nystagmus: Direction
Central: Bi or unidirectional
Periph: Uni, with fast segment indicating the opposite direction of the lesion
Central vs Peripheral Nystagmus: Visual Fixation
Central: No inhibition with fixation
Periph: Will inhibit nyst. and vertigo
Central vs Peripheral Nystagmus: Vertigo
Central: Mild
Periph: Significant
Central vs Peripheral Nystagmus: Length of Symptoms
Central: May be chronic
Periph: Minutes, days, weeks, but finite and recurrent
Fugl-Meyer Sensorimotor Assessment of Balance Performance Battery: For whom, how many items, how many points, high or low is the best?
Hemiplegia, 0-2, 14 is best score, but still may not have normal balance