NEW MD ENDO CARDS Flashcards

1
Q

what 3 types of cells are found in pulp?

A

Fibroblasts

Odontoblasts : primary (before root formation complete) and secondary dentin (after)

mesenchymal cells: tertiary dentin (secondary odontoblasts protect pulp from injury)

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2
Q

how does dentin affect pulp?

A

limits ability to expand

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3
Q

calcifcation of tubules in response to slowly advancing caries is what type of dentin ?

A

sclerotic dentin ( hard)

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4
Q

histologic zone of pulp

A

Inside -> out

predentin , odontoblastic layer, cell-free zone of weil, cell-rich zone, pulp core

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5
Q

this pulp fiber resonds to cold

A

A-delta fiber

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6
Q

structre of C vs alha-delta diber

A

alpha: marge myelated afference nerve

c-fiber: small unmyelinated afferent

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7
Q

which way do alpha and C fibers travel

A

alpha: coronally through pulp

C fiber: centrally om [ulp stroma

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8
Q

C fiber vs alpha fiber, which one is dull throbbing pain?

A

C fiber ( alpha is sharp transient)

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9
Q

a patient feels pain at a stimulus lower than they usually feel. what is this called

A

allodynia ( sun burn hurts when you touch skin, doesnt normally hurt)

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10
Q

an example of referred pain would be ?

A

preauricular pain from mandibular molars ( share V3 innervation)

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11
Q

EPT test is contraindicated in patients that have what>

A

cardiac pacemaker

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12
Q

a patient comes in no complaints of spontaneous pain. turns out symptom is coming from an irritant. what does he have?

A

reversible pulpitis

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13
Q

patient is asymtpmatic and no clinical signs. what do they have

A

asymtpomatic irreversible pulpitis

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14
Q

patient has a long term interruption of blood supply to the pulp. diagnosis ?

A

pulp necrosis

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15
Q

patient has no response to cold. what is it?

A

pulpal necrosis !!

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16
Q

patient has heightened and lingering response to pulp. what is it?

A

symptomatic irreversible pulpitis ( necrosis means no response)

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17
Q

patient has no sympts but PARL developing on radiograph. what do they have?

A

asymtpomatic apical periodontitis

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18
Q

straight line access should be to >

A

orifice and apex

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19
Q

what tooth has the highest rate of root occurrence?

A

maxillary canine 96% chance of 1 root

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20
Q

what tooth most likely to have 2 roots

A

max 1st pm

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21
Q

access shape of maxillary molars

A

blunted triangle/ rhomboidal

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22
Q

access prep shape of mandibular molars

A

trapezoid

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23
Q

file colors

A

why you run blu ? go Back

15 20 25 30 35 40

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24
Q

the SS hand files have what size taper?

A

.02

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25
the niTi rotary instruments have what size
.04 | .06
26
size 15 file is what at the tip? and what at 16 mm?
.15 mm at tip and .47 mm from tip to 16 mm from tip .15 + .02(16mm) .47 mm
27
what is the irrigant that dissolves ORGANIC materials?
NaOCl sodium hypochlorite
28
what is the lubricant that dissolves INORGANIC material
EDTA
29
what dissolves GP in retreatment of RC
chloroform
30
primary endodontic infection bacteria?
bacteroides
31
failed endo treatment bacteria?
enterococcus faecalis | EF
32
main ingredient for GP and sealer? **
zoe !!
33
when condensing GP which one is warm and which is cold vertical and lateral condensing?
Vertical: warm | Lateral : cold
34
what is surgical RCT?
cutting off apex tip and re instrumentation
35
best method for localized and fluctuant swelling?
incision and drainage
36
periapical microsurgery is what/
resectioning of 3 mm of diseased root tip
37
ways to avoid ledging in root canals
NiTI files use smaller instruments to bypass ledge instrument canal to full length pre bend file
38
what type of perforation is through pulpal floor?
furcal perforation
39
what is considered danger zone in a molar in endo?
D side of M root !!!
40
what is better. perforating more apical or more coronal?
more apical
41
what is sign of perforation?
immediate hemmorrhage | sudden pain
42
what material do you internally repair when perforating a tooth
MTA
43
trauma protocol for tooth
TRAVMA ``` tetanus booster ( avulsion only) radiograph antibiotic ( avulsion only) vitality test more appointments ```
44
when would you need tetanus booster for tooth trauma? what about antibiotics?
avulsion only | avulsions only
45
what are ellis classifcation
``` 1- enamel 2- enamel and dentin 3- e,d,p 4- traumatized tooth becomes non vital 5- luxation ( displacement of tooth) 6- avulsion ( completely out) ```
46
what is an uncomplicated fracture ?
fracture without pulp involvement
47
how to fix an euncomplicated fracture affected enamel only?
smooth edges
48
how to fix uncomplicated fracture that affects enamel and dentin?
restore
49
a patient fractures his tooth with pulp involvement. it has been less than 24 hrs. what should we do?
direct pulp cap
50
a patient fractures his tooth to the pulp and it has been 24 hours. what should clinician do?
CVEK ( partial pulpotomy
51
patient fractures his tooth with pulp involved and it has been 72 hours. what to do?
pulpotomy
52
patient has a horizontal root fracture on the coronal portion. what should treatment be>
RIGID splint 6-12 weeks ( WORST KIND)
53
patient has a horizontal root fracture on the midroot portion. what should treatment be>
FLEXIBLE splint for 3 weeks
54
patient has a horizontal root fracture on the APICAL portion. what should treatment be>
flexible splint for 2 weeks max to avoid ankylosis
55
if a tooth is concussed, what is the tx option?
let tooth rest
56
this is when a tooth does not displace, but mobilits is increased. PDL rips and bleeds what is tx? avulsion lateral luxation sub luxation extrusion
subluxation (tooth loose in socket) flexible spling 1-2 weeks
57
tooth is partially extruded from the socket tx? avulsion lateral luxation luxation extrusion
extrusion ( tooth out coronally) open apex: reposition, flexible splint, monitor closed: resp. flexible splint,RCT
58
``` displacement of tooth in any direction except axially. usually crown displaced palatally and root displaced labially tx? avulsion lateral luxation luxation extrusion ```
lateral luxation open: reposition, flexible splint, monitor closed: rep. flex sp. RCT if needed 80% necrosis closed apex
59
complete seperation of tooth from its alveolus tx? closed / open apex >60 and <60 ``` avulsion lateral luxation luxation intrusion extrusion ```
depends on EDT ( extra dry time) closed: <60 : reimplant, splint >60: reimplant, splint, RCT Open: <60: reimplant, splint , specification first sign of pulp infection >60: may or maynot implant, spling, rct, plan for implant
60
apical displacement of tooth tx? ``` avulsion lateral luxation luxation intrusion extrusion ```
intrusion: tooth gets pushed in to socket OPEN: ALLOW TO REERUP !!!! (BOARD ?_ closed: reposition, flex splint, RCT 96% necrosis closed apices
61
which one has 96% chance necrosis with closed apex? ``` avulsion lateral luxation intrusion luxation extrusion ```
intrusion
62
patient falls and tooth comes out. it has been only 45 minutes. what should tx be?
closed: <60 : reimplant, splint Open: <60: reimplant, splint , specification first sign of pulp infection
63
patient falls and tooth comes out. it has been 2 hours. what should tx be?
closed >60: reimplant, splint, RCT open >60: may or maynot implant, spling, rct, plan for implant
64
what is the best and worst store media for a tooth that comes out?
Hanks balanced salt solution -> milk _> saline -> water (hypotonic no balnce of ions)
65
external resorption initiates where and is due to damage of what?
periodonteium | cementoblastic layer
66
ankylosis is type of what ? cervical resorption replacement resorption inflammatory root resporption
replacement: repalces PDL with bone
67
a patient that had nonvital bleeching complains about sensitivity. radiograph shows r/l on cervix of tooth. what might this patient have?
cervical resorption
68
bacteria and byproducts from nectoric pulp travel through dentinal tubules to affect peridontiem called what?
inflammatory root resoprtion ( external resoprtion)
69
internal resoprtion initates where and damages what layer
root canal system | odontoblastic layer
70
true or false. external resoption easier to treat than internal
false | internal is easier
71
tmnt for internal resorption ?
RCT
72
calcific metamorphosis ( discolation of anterior teeth) is caused by what?
trauma that induces odontoblasts to rapidly crease a lot of dentin in pulp space
73
what is radiographic finding of calcific metamorphosis
canal obliteration b/c pulp canal shrink to point you cant see it
74
what does caoh do? | pH?
stimulates secondary odontoblasts (repair dentinal bridge formation) 12.5 ( kills bacteria)
75
what does MTA do? | what is it used for?
stimulates cementoblasts fxn: root repair, apex filler nonresorbable ( great sealer)
76
indirect pulp cap uses what materials
CAOH , RMGI
77
drilling and expose pulp that is 1 mm. what do you do? | material you add?
direct pulp cap, caoh
78
portion of pulp is diseases. what do you do?
remove small portion coronal diseased pulp
79
theres a traumatic exposure of pulp more than 24 hours. what do you do?
cvek pulpotomy (partial)
80
a traumatic exposure lasts more than 72 hours. what do you do? materials add?
pulpotomy zoe in crown formocresol in orifices
81
a primary tooth with traumatic exposure more than 72 hours that is VITAL and resotrable, what do you do?
pulpotomy
82
what are properties of formocresol? what is it made of? when do you use it?
20% formaldehyde bactericidal and fixative ( kills bacteria/ fixes pulp) USE PEDIATRIC PULPOTOMY OF VITAL TOOTH
83
doing a pulptomy on a kid, what do you use ?
formocresol
84
a patient has a nonvital, restorable primary tooth with pulp exposure that is ASYMPTOMATIC. what should they do>?
pulpectomy
85
what materials added for pulpectomy of primary tooth?
zoe in crown caoh in root ( caoh resorbed by underlying permanent teeth) pain relief on teeth with irreversible pulpitis Primary teeth: nonvital and reasonable with asymptomatic pulp exposure
86
a patient has symptomatic molar that nonrestorable and root resporption, what should we do?
extraction SYMPTOMATIC= extraction
87
what tooth is most susceptible to extraction
primary first molar= LOTS OF ACCESSORY CANALS
88
teenager walks in with vital pulp exposure, and is undergone pulp therapy ( CVEK, PPTy) etc. what happens now
apexogenesis ( development of apex in an IMMATURE PERMANENT TOOTH) happens afterpulp cap placed on healthy or diseased pulp ( CaOH and MTA)
89
difference between apexogenesis and apexification?
apexogenesis: - maintain pulp vitality - caOH or MTA place following IPC, DPC, CVEK, PPTY in IMMATURE permanent tooth specification: - disnfection of root canal - CaOH or MTA placed on base of canal after PCTY performed in IMMATURE PERMANENT TOOTJ
90
a pulpectomy is done on a teenager, what treatment should follow it?
apexification | caoh or mta placed at base of canal after pulp removed especially in IMMATURE PERMANENT TOOTH