Neuropsychology Flashcards

1
Q

Nervous system composed of what?

A

Central Nervous System and Peripheral Nervous System

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2
Q

What makes up the CNS?

A

Brain and Spinal Cord

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3
Q

Brain composed of what 3 parts?

A
  1. primitive core - sleeping & breathing
  2. limbic system - basic drives (hunger/thirst) and emotions
    (rage/fear/pleasure)
  3. cerebral cortex or cerebrum - higher functions
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4
Q

paraplegia

A

lower limbs paralyzed

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5
Q

quadriplegia

A

four limbs paralyzed

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6
Q

hemiplegia

A

paralysis in an arm and leg on one side of body

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7
Q

paresis

A

slight or partial paralysis

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8
Q

paresthesia

A

abnormal sensations such as numbness, tingling or burning

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9
Q

hyperesthesia

A

abnormal sensitivity to sensation

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10
Q

Peripheral Nervous System

A

Somatic and Autonomic NS

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11
Q

Somatic Nervous System

A

controls skeletal muscles
voluntary movement
info from senses

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12
Q

Autonomic Nervous System

A

“auto” or involuntary activity i.e. BP, heartbeat.
Autonomic arousal correlated with changes in emotionality
Measured by EKG and GS (galvanic skin response)

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13
Q

Autonomic Nervous System divided into what?

A

Sympathectic - “fight or flight”

Parasympathetic - relaxation, conservation

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14
Q

Action potentials of any given neuron are governed by what?

A

All-or-None-Law
-action potential is not related to intensity of stimulation,
only minimum has to be reached to generate AP
-intensity can generate more APs to stimulate more neurons

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15
Q

Multiple Sclerosis caused by what?

A

Loss of myelin - that which increases the speed of
conduction.
Produces muscular weakness, poor coordination, and tremors

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16
Q

Acetylcholine’s effect on muscles

A
  • found in junction bt nerve & muscle fibers
  • causes muscles to contract
  • defects cause probs w/voluntary mvmt
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17
Q

Neurons that secrete Acetylcholine are called what?

A

cholinergic neurons

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18
Q

Acetylcholine’s effect on brain

A
  • learning and memory (new memories)
  • implicated in Alzheimers (loss of ACh)
  • mediates sexual behavior and REM sleep
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19
Q

Name the catecholamines

A

Norepinephrine, epinephrine and dopamine

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20
Q

Parkinson’s Disease

A
  • too little dopamine

* denigration of substantia nigra neurons in midbrain

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21
Q

Schizophrenia

A

too much dopamine,

also too much norepinephrine

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22
Q

depression

A

too little norepinephrine

too little serotonin

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23
Q

GABA

A

*major inhibitory - depresses activity in NS
*too little = anxiety
*too little in motor areas of brain = Huntington’s Chorea
(invol, jerky mvmts)

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24
Q

Glutamate

A

major excitatory in CNS

  • in hippocampus - memory
  • too much may be responsible for brain damage from stroke
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25
Q

Functions of the Medulla Oblonata

A
  • basic functions
  • breathing, HR, BP, digestion
  • damage is fatal
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26
Q

Functions of the Pons

A

*arousal states
*raphe nuclei (uses serotonin) - trigger and maintain slow
wave sleep

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27
Q

Functions of the Cerebellum

A

*balance, coordination, posture

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28
Q

ataxia

A

*slurred speech, severe tremors, loss balance
(T=tongue/tremors)
*damage to cerebellum produces this

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29
Q

Substantia Nigra

A
  • movement
  • part of extrapyramidal motor system
  • Parkinson’s disease
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30
Q

Reticular Formation

A
  • sleep and arousal
  • pain and touch
  • controls reflexes
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31
Q

Reticular Activitating System (RAS)

A
  • crucial for waking state, arousal and attention

* implicated in ADHD

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32
Q

Hypothalamus

A
  • maintains body homeostasis (i.e. metabolism, temp, fluids)
  • motivation beh (i.e. drinking, eating, sex, aggression)
  • strong feelings into physical responses
  • contains suprachiasmatic nucleus (SCN)
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33
Q

What is the suprachiasmatic nucleus (SCN)?

A

*controls circadian rhythms

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34
Q

Thalamus

A
  • “central relay station”

* relays sensory info to cortex except olfaction

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35
Q

Basal Ganglia

A
  • voluntary movement

* includes caudate nucleus, globus pallidus, and putamen

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36
Q

Extrapyramidal motor system

A

basal ganglia, substantia nigra, cerebellum

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37
Q

Limbic system

A

emotional component of behavior

*includes amygdala, septum and hippocampus

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38
Q

Amygdala

A
  • emotional significance to info
  • mediates aggressive behavior
  • damage produces lack of emotional response
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39
Q

Kluver-Bucy Syndrome

A
  • lesions in amygdala

* reduced fear and aggression, docile, hypersexual

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40
Q

Septum

A
  • part of limbic system
  • inhibits emotionality
  • damage produces hyper-emotionality and vicious behavior
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41
Q

Hyppocampus

A

*part of limbic system
*memory consolidation - from short to long term
*damage produces severe, permanent anterograde amnesia
(no new memories)

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42
Q

Damage to hippocampus, amygdala, and medial temporal

lobes produces what?

A

*severe, permanent anterograde amnesia (no new memories -
think memento)
*retain in short term but can’t recall later

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43
Q

Frontal Lobe

A
  • primary motor cortex
  • expressive lang
  • higher-order cognitive processes
  • orientation to person, place, time
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44
Q

apraxia

A

*inability to execute purposeful movements, despite normal
strength and coordination (AX)
*damage to frontal/parietal lobes

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45
Q

Broca’s aphasia

A

*frontal lobe, left hemisphere, speech production, expressive
lang
*speak w/great difficulty (“Broken” english)
*aware of deficits

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46
Q

Prefrontal cortex

A
  • executive functions
  • lesions disrupt cognition/planning
  • show decreased initiative, lack self-awareness, concrete
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47
Q

“Frontal Lobe Personality”

A
depressive syndrome (apathy) or psychopathic syndrome
(disinhibition - cussing, hypersexual, inappropriate social beh)
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48
Q

Temporal Lobe

A
  • receptive language (primary auditory cortex)

* memory and emotion

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49
Q

Wernicke’s aphasia

A
temporal lobe, left hemisphere, lang comprehension,
receptive lang
*receptive or fluent aphasia
*nonsense/jibberish speech
*dysnomia
*unaware of deficits
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50
Q

dysnomia

A

can’t name familiar objects

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51
Q

Temporal Lobe epilepsy

A

personality syndrome: intense emotions, religiosity, social

clinging, changes in sexual behavior

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52
Q

Parietal Lobe

A
  • primary somatosensory cortex

* touch-pressure, kinesthesia, pain and temp

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53
Q

Lesions in parietal lobe produce what?

A
  • insensitivity to touch on opp side of body
  • contralateral disruption of movement
  • tactile agnosia
  • impaired spatial orientation and facial recognition
  • apraxia
  • contralateral neglect
  • inability to recognize body parts
  • agraphia
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54
Q

Gertsmann’s syndrome

A
  • patterns of deficits caused by lesions to parietal lobe

* agrahia, acalculia, right-left confusion and finger agnosia

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55
Q

Occipital Lobe

A

*visual cortex
*destruction in either hemisphere results in contralateral
blindness
*less extensive damage causes various

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56
Q

left hemisphere (dominant) controls what functions?

A
  • verbal functions: written/spoken lang
  • rational/logical activities
  • damage results in clinical depression or intense anxiety
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57
Q

right hemisphere (non-dominant) controls what functions?

A

*visual-spatial activities
*artistic/musical abilities
*damage results in apathy and indifference, w/exaggerated
but short lived emotional responses

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58
Q

Corpus Callosum

A

bundle of fibers that enables communication between two

hemispheres

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59
Q

conduction aphasia

A

*caused by damage to arcuate fasciculus, fibers connecting
Broca’s and Wernicke’s area
*person’s speech makes sense but person cannot repeat what
they just heard

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60
Q

Freud’s belief about emotions

A
  • universal
  • not under conscious control
  • tied to early experiences
61
Q

James-Lange Theory of emotion

A

behavior first then emotion,

*we are afraid because we tremble, sorry because we cry

62
Q

Cannon-Bard Theory of emotion

A

*behavior (arousal) and emotion happen simultaneously, not
cause/effect
*all emotions identical in terms of arousal

63
Q

Cognitive-Arousal Theory of emotion

A
  • Schachter and Singer

* arousal and cognitive attributions for arousal

64
Q

Universal emotions: T or F?

A

True, agreement on 6 basic emotions: fear, anger, joy,

sadness, surprise and disgust,

65
Q

Which two brain regions are centers for hunger and satiety?

A

hypothalamus - lateral and ventromedial
LH - failure to eat/drink, “Little Hunger”
VMH - overeating, “Very Much Hunger”

66
Q

Research findings on external cues and obesity?

A

*obese are less responsive to internal, biological hunger cue
and more responsive to external cues
*stress eating

67
Q

What effect does lutenizing hormone (LH) have on body?

A
  • from pituitary & hypothalamus

* gonads (testes/ovaries) produce angdrogens and estrogen

68
Q

What effect does Follicle Stimulating hormone (FSH) have

on body?

A
  • from pituitary & hypothalamus

* causes production of sperm and release egg

69
Q

Androgens

A
  • primarily male but found in female
  • testosterone
  • cause dev of 2’ sex chara in males
  • sex interest in both males and females
70
Q

Estrogens

A
  • found in male and females, male function unknown
  • females: sex dev and reprod functioning
  • less of role in sex behavior - experience more of role
71
Q

Effect of spinal cord injury on sexual functioning

A
  • ejaculation affected, less on erection or interest

* less effect on females

72
Q

Stages of sleep

A
5 stages, 4 Non-REM, 5th REM
1-transitional, alpha into slower theta
2-theta, "sleep spindles", K-complexes (bursts faster/higher
waves)
3-delta appear
4-predominantely delta
(stages 3&4=delta/deep sleep)
5-REM, aroused nervous system w/low responsivity to
environ
73
Q

alpha waves

A

relaxed wakefulness

74
Q

beta waves

A

active, alert states - faster

75
Q

Sleep cycle

A
  • 100 minutes, 4-6x night
  • REM increases thru night
  • delta/deep sleep decreases thru night
76
Q

Memory: temporal lobe vs. hippocampus

A

temporal lobe - long-term memory

hippocampus - consolidation

77
Q

Korsakoff’s syndrome

A
  • memory disorder in alcoholics
  • lesions in mammillary bodies (hypothalamus) and thalamus
  • retro/antero amnesia, confabulation, apathy
78
Q

Long-term potentiation

A

*responsible for long-term memory
*changes in synaptic structrue following high stim of
neurons
*in hippocampal cells

79
Q

RNA

A
  • RiboNucleic acid
  • stimulates memory and learning
  • training/experiences increases RNA
80
Q

Endocrine system

A

*controlled by hypothalamus
*comprised of glands that secrete hormones to influence
organ functioning
*pituitary, adrenal, gonads, thyroid, pancreas

81
Q

Pituitary gland

A
  • “master gland”
  • secretes growth hormone/somatotropic hormone
  • and antidiuretic hormone
82
Q

Adrenal Cortex

A
  • secretes cortisol
  • stimulates liver to convert energy into glucose for fuel
  • cortisol released by adrenocorticotropic hormone (ACTH)
83
Q

Addison’s Disease

A

*under-secretion of ACTH in adrenal cortex
*fatigue, fainting, loss appetite, weight loss, depression,
apathy

84
Q

Cushing’s Disease

A

*over-secretion of ACTH in adrenal cortex
*obesity, memory loss, mood swings, depression, somatic
delusions

85
Q

Thyroid gland

A
  • hormone thyroxin

* controls metabolism

86
Q

Hypo-thyroidism

A

under-secretion of thyroxin
slows metab, weight gain, decreased sex drive, depression
w/cognitive impairment

87
Q

Hyper-thyroidism

A

over-secretion of thyroxin
*a.k.a. Grave’s Disease
*increase metab, appetite, weight loss, nervousness,
insomnia, fatigue

88
Q

Pancreas

A
  • releases insulin

* diabetes and hypoglycemia

89
Q

Anatomy of Eye

A
  1. Cornea - cover
  2. Pupil (Iris) - dilation
  3. Lens - focuses light waves on retina
  4. Retina - receptors of visual system
90
Q

Rods and Cones - compare/contrast

A

Rods - brightness, esp. at night, periphery of retina

Cones - color, center of retina

91
Q

Travel of visual signals

A
  1. optic tract
  2. lateral geniculate nucleus of thalamus
  3. visual cortex in occipital lobe
92
Q

describe variations of sound waves

A
  1. frequency (pitch) or Hertz
  2. amplitude (loudness) or decibels
  3. overtones (timbre)
93
Q

auditory localization

A
  • ability to orient toward the direction of a sound

* present at birth, declines, then re-emerges, full dev at 1 year

94
Q

Gate-control theory of pain

A

*activation of larger fibers “closes gate”
*activities include massage, heat/cold, distracting mental
activities

95
Q

what makes olfaction different from other senses?

A

*directly to limbic system, not relayed through thalamus or
cross hemispheric
*considered most primitive of senses or “lowest on
phylogenetic scale”

96
Q

absolute threshold

A

Fechner - psychophysics

  • weakest stim a person can detect
  • intensity at which stim is detected 50% of time
97
Q

difference threshold, “just noticeable difference”

A

Fechner - psychophysics
*smallest physical diff between two stim recognized as
difference
*vary depending on magnitude of original stim

98
Q

structural brain imaging techniques

A
  • info about structure of brain
  • CT scan (xrays, “slices”)
  • MRI (more precise, not xray)
99
Q

functional brain imaging techniques

A

*info both structure and function
*PET (inject radioactive material) - map NTs and dys due to
conditions
*SPECT and fMRI

100
Q

Dysarthria vs. Aphasia

A

*Dysarthria is problem in articulation, common in
Parkinson’s, Huntington’s and MS
*Aphasia is language disturbance

101
Q

Alexia vs Dyslexia

A
  • Alexia - word blindness caused by acquired brain lesion

* Dyslexia - learning disability (not lesion)

102
Q

Prosopagnosia

A

inability to recognize familiar faces (use “soap” to wash

face)

103
Q

Anosognosia

A

*inability or unwillingness to recognize one’s own functional
impairment, and sometimes other’s impairment
*often in stroke patients in right parietal cortex

104
Q

anosognosia vs. contralateral neglect

A

*contralateral neglect are inattentive to EVERYTHING in
oppositive field of vision vs. only functional defects
*contralateral neglect will acknowledge if attention directed
to it vs. anosognosia px continue to deny

105
Q

symptoms of brain tumor

A
  1. headaches
  2. seizures
  3. nausea/vomiting
  4. change vision/hearing
  5. focal neurological signs
106
Q

differences in locale of brain tumor, child vs. adult

A

child - brainstem and cerebellum

adult - cerebral cortex

107
Q

results of stroke

A

*50% die immed or w/i months
*only 10% fully recover
*greatest improvement occurs in 1st 6 months
*physical symptoms improve quicker than cognitive
symptoms.

108
Q

Most common cause of brain damage in people under 40?

A

Head trauma

109
Q

best predictor of degree of injury and recovery following

closed head trauma?

A
  • duration of anterograde amnesia

* most recovery occurs w/i 6-9 months

110
Q

postconcussional disorder - proposed DSM-IV for further

study

A

*presence of sig concussion following head trauma (LOC)
and
3+ symptoms for at least 3 mos following (i.e. fatigue,
headaches, irritability)

111
Q

Huntington’s chorea

A
*affective, personality, motor and cognitive
changes/deterioriation
*genetically trans to 1/2 offspring
*athetosis - slow writhing mvmts
*chorea - invol, jerky mvmts
*substantia nigra, basal ganglia, cortex
*glutamate, ACh, GABA, dopamine
112
Q

Parkinson’s disease

A
  • tremor at rest
  • muscle rigidity
  • invol mvmts
  • equilibrium disturbances
  • akinesia - slowness mvmt, blank exp, emotionless speech
  • 40% comorbid depression
  • substantia nigra
  • too little dopamine
113
Q

Name the 3 classes of antidepressants

A
  1. tricyclics
  2. SSRIs
  3. MAOIs
114
Q

MOA of tricyclics

A

block reuptake of norepi and serotonin

115
Q

tricyclics most effective at tx what?

A
  1. vegetative symptoms of depression

2. panic attacks, agoraphobia and obsessive states

116
Q

side effects of tricyclics

A
  • anticholinergic effects (dry up)
  • cardiovascular effects
  • gastrointestinal
117
Q

cautions of tricyclics

A
  • OD can be lethal (cardiac arrest)
  • combo w/MAOI causes severe convulsions
  • caution w/suicidal pt
118
Q

Examples of tricyclics

A

imipramine (Tofranil)
clomipramine (Anafranil) - OCD
amitriptyline (Elavil)

119
Q

MOA of SSRIs

A

inhibit reuptake of serotonin

120
Q

SSRIs prescribed for what?

A

depression
OCD
ED

121
Q

side effects if SSRIs

A

-gastrointestional
-akathesia
-initally worsen anxiety/sleep
(not cardiotoxic, no OD threat, no antichol, no cognitive imp,
faster acting)

122
Q

akathesia

A

motor restlessness

123
Q

Examples of SSRIs

A

Fluoxetine (Prozac)
Sertaline (Zoloft)
Paroxetine (Paxil)

124
Q

MOA of MAOIs

A

bloc enzymes that breakdown norepi and serotonin

125
Q

MAOIs recommended for what?

A

atypical depression w/anxiety

126
Q

side effects of MAOIs

A
  • fatal hypertensive crisis w/tyramine!!

- OD can be fatal (suicide risk)

127
Q

Examples of MAOIs

A

phenelzine (Nardil)

tranylcypromine (Parnate)

128
Q

Name mood stabilizers/anti-convulsant drugs

A

Lithium - tx if choice for bipolar
carbamazepine (Tegretol) - as effective as Lithium in mania
esp. dysphoric and rapid cycling mania, faster onset than
Lithium
valproic acid (Depakote) - fewer SE

129
Q

Side effects of lithium

A
  • gastric distress, weight gain, fatigue, mild cog imp
  • 35% tremor in fingers
  • lithium toxicity
130
Q

MOA of antipsychotics (neuroleptics)

A

block dopamine receptors

131
Q

Examples of antipsychotics (neuroleptics)

A

chlorpromazine (Thorazine)
thioridazine (Mellaril)
haloperidol (Haldol)
clozapine (Clozaril) - less SE, more NTs

132
Q

antipsychotics (neuroleptics) prescribed for?

A
  • schizophrenia: positive symptoms

- acute mania, psychotic symptoms

133
Q

dopamine hypothesis

A

schizophrenia relaed to overactivity of dopamine
(Parkinson’s too little), experts now believe more complex
and involve other NT like norepi and serotonin

134
Q

side effects of antipsychotics (neuroleptics)

A

anticholinergic
extrapyramidal: Parkinsonism and tardive dyskinesia
(abnormal invol mvmts esp in face)

135
Q

tardive dyskinesia

A
  • abnormal invol mvmts esp in face
  • delayed effect of antipsychotic use
  • relieved somewhat w/withdrawal
136
Q

What is special about clozapine (Clozaril)?

A
  • newer “atypical” antipsychotic
  • less SE (no tardive dys or exrapyram)
  • lowers activity of multiple NT
  • agranulocytosis (low white blood cells)
137
Q

MOA of benzodiazepines

A

increase GABA activity which inhibits CNS

138
Q

SE of benzos

A
  • drowsiness and sedation
  • anterorgade amnesia (esp w/IV)
  • elderly - confusion and disorient
  • addictive
  • fatal if mixed w/alcohol or other CNS depressant
139
Q

MOA and examples of barbiturates

A

interrupt impulses to RAS
thiopental (Pentothal)
amobarbital (Amytal)
secobarbital (Seconal)

140
Q

barbituates tx for what?

A
  • replaced by benzos for anxiety

- anesthetic, acute mgmt of agitated px

141
Q

SE of barbituates

A
  • addictive (fatal withdrawal)
  • drug-of-choice for suicide
  • suppress respiration
  • esp. lethal w/alcohol
142
Q

Beta-blockers as tx for what?

A

performance related anxiety

reduces sympathetic NS activity

143
Q

benzos vs. beta-blockers?

A

-beta blockers less effective in tx cognitive/psychic
experience of anxiety but more effective in physical
-beta blockers more likely cause memory impairment

144
Q

MOA of psychostimulants

A

mimic catecholamines (norepi and dopamine) in brain

145
Q

MOA of narcotic-analgesics

A

block transmission of neural impulses to CNS

146
Q

narcotic-analgesics tx for what?

A

pre-operative meds, acute pain, chronic pain w/cancer, detox

147
Q

Methadone used for what?

A

in detoxification programs as substitute for heroin, milder

withdrawal

148
Q

biofeedback to treat anxiety targets what part of nervous

system?

A

parasympathetic of autonomic NS - relaxation and

recuperation