Neurological System Flashcards
frontal lobe is responsible for:
primary motor cortex:
personality
expressive speech (Broca’s area)
judgement & decision-making
abstract thought
concentration
temper
frontal lobe impairment causes:
contralateral weakness
inattention
apathy
poor inhibition
personality change
Broca’s aphasia (expressive deficits)
parietal lobe is responsible for:
gross sensation (touch, pressure)
fine sensation (texture, weight, size, shape)
sensory homonculus
language
spatial & visual perception
parietal lobe impairment causes:
L side: agraphia, alexia, agnosia (language comprehension)
R side: dressing & constructional apraxia, anosognosia (no insight)
contralateral sensory deficits: impaired language, taste & comprehension
temporal lobe is responsible for:
primary auditory processing & olfaction
behavior
language reception (Wernicke’s area)
understanding
Long term memory
interpret nonverbals of others
temporal lobe impairment causes:
learning deficit
antisocial/aggressive
Wernicke’s aphasia
difficulty w/ facial recognition, memory & categorizing objects
occipital lobe is responsible for:
vision & 3D
recognize size, shape & color
judging distance
occipital lobe impairment causes:
impaired eye muscle movement
visual deficits
decreased color recognition
read/write deficits
homonymous hemianopsia
cortical blindness w/ bilat. lobe involved
hippocampus location & function
(lower temporal lobe)
learning language
form/store new personal memories
declarative memory
sending memory to appropriate areas of cerebral hemisphere
basal ganglia function
voluntary movement
regulate autonomic movement, posture & mm tone
(includes caudate, putamen, globus, pallidus, substantia nigra, subthalamic nuclei)
diseases of dysfunctional basal ganglia
Parkinson’s
Tourette’s
Huntington’s
ADD
OCD
addictions
amygdala location & function
(in temporal lobe, next to hippocampus)
Emotional and social processing
Fear/pleasure response, arousal, memory processing, form emotional memories
hypothalamus function
receive/integrate info from autonomic NS
hormone regulation
controls hunger, thirst, sex, sleep
regulate body temp, adrenal glands, pituitary glands
subthalamus location & function
between thalamus & hypothalamus
regulates movement from skeletal muscle
epithalamus/pineal gland function
secretes melatonin for circadian rhythm
regulates some motor pathways & emotions
cerebellum function
balance, posture & complex muscle movements
integration, coordination & execution of multi-joint movements
regulates mm contractions (initiation, timing, sequencing, force)
pons function
helps regulate respiration rate
associated w/ head orientation to visual/auditory stimuli
includes CN 5-8 origins
medulla oblongata function & wht/gry matter distribution
influence autonomic activity (RR & HR)
reflex center for vomiting, coughing, sneezing
outer white matter, inner gray matter
What is responsible for exchanging nutrients between the CNS & vascular system?
blood/brain barrier
3 Meninges Layers & 3 spaces between
[skull]
-epidural space
dura mater (outer)
-subdural space
arachnoid mater (middle)
-subarachnoid space w/ CSF
pia mater (inner)
PNS: how many cranial nerves?
how many spinal nerves? (cervical, thoracic, lumbar, sacral, coccygeal)
12 CN
8 cervical
12 thoracic
5 lumbar
5 sacral
1 coccygeal
sympathetic NS: hormone, affect on BP, HR & blood flow
norepinephrine
inc. HR, BP & blood flow to skeletal mm
parasympathetic NS: hormone & affect on HR, BP & digestion; which CN affected (4 total)
acetylcholine
dec/normalized HR, BP & digestion
oculomotor (3), facial & glossopharangeal (7, 9, taste) & vagus
Name 3 early embryo layers
ectoderm: brain components
mesoderm: vertebrae/skull, dermis, skeletal mm
endoderm: organ development
upper motor neurons: cell body origin/termination
where do they cross?
where do they synapse w/ LMN?
origin: cerebral cortex
termination: brainstem/spinal cord
cross: medulla
LMN synapse: anterior horn of SC
UMN Lesions = which diseases?
MS
SCI
Parkinsons
CVA/stroke
TBI/head trauma
ALS
UMN lesion characteristics
spastic weakness
loss of autonomic control
clonus
paralysis
hyperreflexia
lower motor neurons: cell body origin of cranial nerves & spinal nerves
CN: brainstem
spinal nerves: anterior horn of S.C.
LMN lesions = what diseases?
muscular dystrophy
polio
myasthenia graves
ALS,
Guillan Barre
peripheral nerve injury
LMN lesion characteristics
hypotonia
hyporeflexia
paralysis
atrophy
loss of coordination
temporary nerve damage due to pressure
neuropraxia
prolonged nerve pressure causes muscle atrophy & axon degeneration (can be permanent)
axonotmesis
nerve axon/sheath damaged; can require surgery
neurotmesis
when peripheral nerve is cut & parts of axon distal to transaction point are disconnected from central neuron
Wallerian degeneration
PT tx: reduce return of primitive reflex patterns of movement (post CVA)
-stimulate return of normal tone/posture
Brunnstrom approach
PT tx: focusing on normal movement without compensations (often w/ UMN lesions)
neurodevelopmental therapy (NDT)/ Bobath technique
progressive degenerative organic brain syndrome that destroys neurons of cerebral cortex (frontal lobe first) w/ plaques
Alzheimer’s Disease