neurological gait Flashcards

1
Q

kinematics vs. kinetics

A
  1. kinetics: forces producing movement

2. kinematics: description of movement; velocity or distance or stride length (nothing to do with force)

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2
Q

ankle/ foot necessities for gait (3)

A
  1. heel first initial contact
  2. smooth transition to foot flat
  3. heel off -> toe off (push off)
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3
Q

what is the main deviation at ankle/ foot?
3 names
why do we care?

A
main deviation is diminished clearance in swing...
1. foot slap
2. foot drop
 these both can lead to 
3. foot drag

these create a huge falls risk

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4
Q

2 things to remember while testing gait

A
  1. worsens on uneven terrain
  2. may not be present inside or on shorter tests

these should be provacative tests

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5
Q

what causes the ankle/foot deviations?

and what are these secondary to? (3)

A
  1. ankle plantarflexion contracture/ tightness in gastroc or heel cord
  2. plantarflexion spasticity = UMN
  3. dorsiflexor weakness = can be LMN
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6
Q

**what contributes most to stability during gait?

A
  1. push off in stance phase** = comes from hip extension
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7
Q

foot drag compensations (6)

A
  1. circumduction (swinging foot to side)
  2. contralateral vaulting (tip-toes of other foot)
  3. hip-hiking (QL)
  4. contralateral trunk lean
  5. hip ER (pivoting on toe of uninvolved side)
  6. trunk extension
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8
Q

treatments for foots drag
what we do (2)
meds (2)
last resort

A
  1. stretch PF** a lot** 30-60s/hr
  2. strengthen DF
    all with task specificity
    meds:
    if due to spasticity- baclofen or tizanidine

bracing as last resort because immobilizes foot

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9
Q

knee necessities during gait (2)

A
  1. knee flexion at heel off

2. smooth transition of knee flexion -> extension during swing

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10
Q

2 gait deviations at knee and what they are secondary to

A
  1. absent or inadequate knee flexion at heel off
    • secondary to spasticity at quads or weakness of hamstrings
  2. extension thrust (as you travel thru stance, knee snaps back into ext ** not genu recuvatum)
    • secondary to PF spasticity or contractures
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11
Q

some treatment options for knee gait deviations (2)

A
  1. stretch quads

2. activate hamstrings

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12
Q

two reasons we don’t like orthodics (AFOs)

A
  1. can make contracture worse

2. cause weakness secondary to immobility

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13
Q

critical components of hip during gait (4)

A
  1. flexion during swing
  2. extension during stance* most important thing for stability during gait
  3. neutral aB/aDduction during stance
  4. ER during swing/ IR during stance
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14
Q

usual compensation for lack of hip flexion during swing

concomitant with…

A
  1. trunk extension during swing

2. concomitant with foot drop

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15
Q
trendelenberg
 def and 2 muscles that cause this
 unlikely but occasional cause
 compensation
 treatment
A
  1. def- stance phase weakness of hip aBductors (glut med or TFL)
  2. rarely from spasticity of aDductors
  3. compensation is ipsilateral trunk lean
  4. treatment- strengthen glut med in closed chain position
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16
Q

scissoring gait
what it looks like
secondary to (2)

A
  1. swing phase hip aDduction -> flexion
    secondary to:
  2. spasticity or (2) weakness of aBductors therefore can be UMN or LMN
17
Q

glut max lurch
what it looks like
secondary to (2)
compensation

A
  1. lurching gait because of inadequate mid -> late stance hip extension

secondary to:

  1. hip extensor weakness
  2. hip flexor contracture

compensation = trunk extension

18
Q

gait deviation for pelvis (1)
secondary to (2)
what these are caused by
treatment

A
  1. unilateral pelvic retraction
  2. secondary to weakness or atrophy in hip protractors/ and trunk extensors
  3. usually part of larger hemiplegic pattern so common in stroke during cortical shock
  4. treatment- PNF
19
Q

2 gait deviations for trunk and what they are secondary to

A
  1. excessive trunk flexion- b/c of weakness in extensors or flexion contracture from prolonged positioning
  2. diminished absent rotation- parkinsonism
20
Q

scapula gait deviation (1)
causes if unilateral vs. bilateral
treatment

A

deviation = protraction

unilateral= stroke/ hemiplegia
bilateral = postural or PD

treatment- stretch protractors or strengthen retractors

21
Q

arm gait deviation (2)
unilat vs. bilat
and other disease for excessive arm swing

A
deviation=  absent or diminished arm swing
unilateral = stroke/ hemiplegia or antalgic
bilateral = parkinsonism

or
deviation = excessive arm swing from huntingtons

22
Q

forward head posture causes (3)

A
  1. prolonged positioning
  2. afraid of falling (but this increases falls risk)
  3. PD
23
Q

parkinsonian gait (8)

A
  1. flexed trunk
  2. cervical hyperextension
  3. shuffling (bilat toe first)
  4. festination - shorter and shorter and faster and faster => falls
  5. absent arm swing
  6. absent trunk rotation
  7. tremor
  8. freezing (at doorways or threshold)
24
Q

cerebellar gait (2)

A
  1. wide BOS

2. ataxic (disordered)

25
Q

hemiplegic gait (6)

A
  1. unilateral
  2. foot drag
  3. possible extension thrust
  4. possible trendelenberg
  5. pelvic retraction
  6. unequal step lengths and diminished time in stance on involved leg.
26
Q

MS gait

A
  1. anything is possible but worsens with fatigure- 6mwt- will slow down over time
27
Q

tabes dorsalis

A

a.k.a. syphilis

stamping feet to increase sensory feedback due to dorsal column damage

28
Q

3 biggest contributors to gait

A
  1. Push off in stance
  2. Heel strike
  3. Med/ lat stability