Neuro/Cardiac Section Flashcards

1
Q

Midline structures affected with stroke

A

Medial lemniscus
Motor pathway (corticospinal)
Motor aspects of CN
Medial longitudinal fasciculus

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2
Q

Lateral structures affected with stroke

A

Spinothalamic
Spinocerebellar
Sympathetic - Hoerner’s
Sensory sections of CN V

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3
Q

Signs and symptoms of uncal herniation

A

Dilation of pupil on side of hernitation (CN III paresis), hemiparesis, coma, homonymous hemianopsia

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4
Q

S/S of central hernitation

A

Decrebrate rigidity and coma

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5
Q

S/S of tonsilar herniation

A

Neck pain and stiffness, flaccidity, coma, alteration of vital signs

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6
Q

Branches of right coronary artery

A

Sinus node artery (right atrium)
Right marginal artery (right ventricle)
Posterior descending (inferior walls of ventricles)

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7
Q

Branches of left coronary artery

A
Circumflex artery (left atrium and ventricle)
Left anterior descending (IV septum)
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8
Q

ABI normal

A

1.0-1.3

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9
Q

ABI mild

A

0.99-0.80 (beginning of PAD)

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10
Q

ABI moderate

A

0.40-0.79 (intermittent claudication)

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11
Q

ABI severe

A

<0.40 (claudication at rest)

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12
Q

Bronchophony

A

Increased vocal resonance with greater clarity and loudness of spoken word

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13
Q

Egophony

A

Spoken long “E” changes to nasal “A”

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14
Q

Whispered pectoriloquy

A

Recognition of whispered word

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15
Q

Infant HR

A

100-130

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16
Q

Child HR

A

80-100

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17
Q

Adult HR

A

60-100

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18
Q

Newborn RR

A

33-45

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19
Q

1 yr RR

A

25-35

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20
Q

10 yr RR

A

15-20

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21
Q

Adult RR

A

12-20

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22
Q

PR interval

A

Time for atrial depolarization and conduction from SA node to AV node
0.12-.20

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23
Q

QRS compex

A

Ventricular depolarization and atrial repolarization

0.06-1.0

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24
Q

QT interval

A

Time for both ventricular depolarization and repolarization

0.20-0.40

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25
Submax training RPE
Initial 12-13 | Final 15-16
26
Mobitz I
General prolongation of PR interval until QRS is dropped
27
Mobitz II
Dropped QRS, PR interval remains unchanged
28
Hypermagenesemia
Calcium channel blocker so can lead to arrhythmias or cardiac arrest
29
Hypomagenesemia
Ventricular arrhythmias, coronary artery vasospasm, sudden death
30
Hyperkalemia
Decreases the rate and force of contraction, widened PR interval and QRS, tall T waves
31
Hypokalemia
Flattened T waves, prolonged PR and QT intervals, produces a U wave
32
Hypothermia effects on ECG
Elevates ST segment; slows rhythm
33
Digitalis effects on ECG
Depresses ST segment, flattens T wave, QT shortens
34
Quinidine effects on ECG
QT lengthens, T wave flattens, QRS lengthens
35
Beta blockers
Decrease HR, blunts HR response to exercise
36
Nitrates
Increase HR
37
Normal BP
120-129/80-84
38
High Normal BP
130-139/85-89
39
Grade I HTN
140-159/90-99
40
Grade II HTN
160-179/100-109
41
Grade III HTN
>180/>110
42
Levine's sign
Pt clenches fist over sternum
43
Inferior MI
Right coronary artery (upper conduction system)
44
Lateral MI
Circumflex artery
45
Anterior MI
L anterior descending (lower conduction system)
46
Tinetti high risk for falls
<18
47
Berg high risk for falls
<20
48
TUG high risk for falls
>30
49
Functional reach risk for falls
<10 in
50
DGI prediction of falls
<19/24
51
Berg moderate risk for falls
21-40
52
Tinetti moderate risk for falls
19-24
53
Normal adult TUG
<10
54
Normal elderly TUG
11-20
55
Mild TBI criteria
LOC <30 min, PTA <1 day, GCS 13-15
56
Moderate TBI criteria
LOC >30 min <1 day, PTA >1 day, <7 days, GCS 9-12
57
Severe TBI criteria
LOC >24 hrs, PTA > 7 days, GCS <9
58
MOA Alpha adrenergic agents
Cause vasodilation of arterioles and veins to decrease BP
59
MOA of ACE inhibitors
Suppress conversion of angiotensin I to angiotensin II to decrease BP and afterload
60
MOA Sodium channel blockers
Control cardiac excitation and conduction
61
MOA Beta blockers
Inhibit sympathetic activity which decreases HR and contractility
62
MOA of statins
Inhibit enzyme action in cholesterol synthesis, break down LDLs, decrease triglycerides, and increase HDL levels
63
MOA calcium channel blockers
Decrease entry of calcium into vascular smooth tissue resulting in diminished myocardial contraction, vasodilation, and decreased oxygen demand
64
MOA nitrates
Decrease ischemia through smooth muscle relaxation and dilation of peripheral vessels
65
MOA positive inotropic agents
Increase the force and velocity of myocardial contraction, slow HR, decrease conduction velocity thru AV node, and decrease activation of SNS