Muscle tone Flashcards

1
Q

define normal postural tone

A

a continuous state of mild contraction, or a state of preparedness of the muscle

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2
Q

what is normal postural tone characterized by?

A
  • effective co-activation (stabilization) at axial and proximal joints
  • ability to move against gravity and resistance (it is high enough to resist gravity and low enough to allow mobility)
  • ability to maintain the limb in a position
  • balanced tone between agonist and antagonistic muscles
  • ease of ability to shift from stability to mobility and reverse as needed
  • allows automatic postural adjustments to movement
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3
Q

what is muscle tone?

A

the degree of tension (contraction) in a skeletal muscle

- it is dependent on the integrity of the peripheral and CNS mechanisms and the properties of the muscle

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4
Q

what determine muscle tone?

A
  • elastic properties of connective tissue
  • visco-elastic properties of muscle fibers
  • motor unit activity (neural)
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5
Q

on what does normal muscle tone rely?

A
  • normal functioning of the cerebellum
  • motor cortex
  • basal ganglia
  • midbrain
  • vestibular system
  • spinal cord
  • neuromuscular system
  • on a normally functioning stretch reflex
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6
Q

characteristics of normal muscle tone

A
  1. effective coactivation at axial and proximal joints
  2. ability to move against gravity ad resistance
  3. ability to maintain the position of the limb if it is placed passively by the examiner and then released
  4. balanced tone between agonistic and antagonistic muscles
  5. ease of ability to shift from stability to mobility and reverse as needed
  6. ability to use muscles in groups or selectively with normal timing and coordination
  7. resilience or slight resistance in response to passive movement
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7
Q

explain the upper motor neuron control of muscles

A
  • motor cortex
  • descending motor cortex
  • anterior horn of spinal cord grey matter
  • additional brain areas
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8
Q

explain the lower motor neuron control of muscles

A

motor unit:

  • motor neuron
  • motor (efferent) nerve fibers
  • spinal nerves
  • muscle fibers it supplies
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9
Q

results of lesions of upper motor neuron

A
  • weakness of the specific movement (extension of upper limbs and flexion of lower limbs pyramidal weakness)
  • spasticity or high tone
  • overactive stretch reflex
  • hyperactivity of motor neurons
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10
Q

results of lesions of lower motor neuron

A
  • weakness (paresis)
  • wasting (atrophy)
  • flaccidity
  • low muscle tone
  • suppressed/ absent reflexes
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11
Q

causes of abnormal muscle tone

A
hypertonicity
- secondary to CNS pathology (stroke/ trauma)
- changes in the ascending tracks (efferent) inhibiting motor neurons
- range of postural tone
   \+ no anti-gravity control
   \+ no gradation of movement
   \+ = total reciprocal inhibition
- muscle tone
   \+ increased resistance to passive stretch and movement
  \+ hyperactive reflexes
  \+ changes in mechanical structures
  \+ types
      = spasticity
      = rigidity
      = dystonia
      = clonus
      = spasms 
hypotonicity
-cerebellum pathology
- abnormality in lower motor neuron
- neuromuscular junction pathway
- muscle pathology
- range of postural tone
   \+ no postural adjustments
   \+ movement blocked
   \+ = total co-contraction
- muscle tone
   \+ associated with:
      = weakness
      = atrophy
      = absent/ decreased reflex response
   \+ types
      = flaccidity
      = hypotony 
      = ataxi
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12
Q

what influences assessment of muscle tone?

A
  • speed
  • effort
  • emotional stress
  • temperature
  • fatigue
  • position of head
  • sensory stimulation
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13
Q

causes of hypotonia

A
  • usually occurs in newborn babies up to age of 6 weeks and is accompanied by phasic reflexes
  • most children with cerebral impairment start with hypotonia
  • muscle disease can cause hypotonia (muscular dystrophy or polio myositis
  • damage of motor fibers of peripheral nerves
  • damage of dorsal or ventral nerve roots usually occurs in isolation, but together with a spinal cord injury
  • spinal cord injuries like paraplegia
  • when cerebellum is affected, tone is lowered, although active movement can still occur, albeit in an abnormal manner
  • traumatic brain/spinal injury, flaccidity initially exists and could change to hypertonicity within a few weeks
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14
Q

what is flaccidity and its characteristics?

A
  • refers to absence of tone
  • deep tendon reflexes and active movement are absent
  • can result in LMN dysfunction
  • muscles feel soft and offer not resistance to passive movement
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15
Q

what can be observed in ataxia?

A
  • decomposition of movement
    + when client reaches for object, movement is jerky or broken up
  • dysmetry
    + when client reaches for object, they go beyond it or stop in front of it
  • disdiadokokinesia (DDK)
    + client cannot perform quick alternating movements like pronation and supination
  • intension tremor
    + tremor usually occurs at the end of a movement in distal muscles, causing rhythmic oscillation of hands when they near the object. this usually aggravates with complicated or difficult movement
  • nystagmus
    + rhythmic oscillation of both eyes
  • heel-knee test
    + when client is asked to move heel over tibia, they cannot do so in a coordinated movement and it is poorly coordinated
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16
Q

characteristics of spasticity

A
  • hyperactivity of muscle spindles phasic stretch reflex with hyperactive firing of the IA afferent nerve
  • velocity dependence, meaning stretch reflex is only elicited by the examiner’s rapid passive stretch
  • the ‘clasp-knife’ phenomenon. this means that when the examiner takes the extremity through quick passive stretch, a sudden catch or resistance is felt, followed by a release of the resistance. what actually happens is that the initial high resistance of the spasticity is inhibited suddenly.
  • client can usually not perform active movement
17
Q

characteristics of hypertonicity

A
  • more than normal resistance of a muscle against passive stretching or lengthening due to neurological and mechanical factors
  • increased tone is usually present in the anti-gravity muscles of the upper and lower limbs
  • degrees
    + mild
    + moderate
    + severe
18
Q

anti-gravity muscles of upper limb

A
shoulder joint
  - adductors
  - internal rotators
  - flexors
elbow joint
  - flexors
radio-ulnar joint
  - pronators
wrist joint
  - flexors
fingers
  - flexors
  - adductors
thumb
  - flexors
  - adductors
19
Q

anti-gravity muscles of lower limb

A
hip joint
  - extensors
  - adductors
  - internal rotators
knee joint
  - extensors
ankle joint
  - plantar flexors
  - invertors
toes
  - flexors
  - adductors
20
Q

what are the causes of hypertonicity

A
  • high tone, normally in babies aged 6-12 months
  • brain stem injuries
  • frontal lobe injuries (where the pyramidal tract is damaged)
  • injuries to internal capsule (spasticity can occur on one half of the body)
21
Q

what is rigidity

A

simultaneous increase in muscle tone of agonist and antagonist muscles

22
Q

types of rigidity

A
  • cog-wheel
  • lead pipe
  • decorticate
    + flexion rigidity in the UL and as extension rigidity in LL
  • decerebrate
    + rigid extension posturing of all limbs and the neck
23
Q

which precautions does one need to consider when assessing muscle tone?

A
  • immediately note whether there are fixated contractures
  • limb is often painful as a result of disuse. muscle tendons can be further damaged if limb is moved beyond pain threshold
  • client’s ability to adapt to changed environment is affected. position cannot automatically be adjusted, and take care that the patient is not injured or falls as a result of protective extension
24
Q

fluctuation muscle tone

A
  • muscle tone fluctuates between hyper-, hypotonicity, and normal tone
  • there is active movement but is abnormal and uncoordinated
  • causes
    + extrapyramidal damage
    = tremors
    = pill-rolling tremor
    = senile tremor
    = intention tremor
    = athetosis
    = myoclonus
    = ballismus
    = distonia
    + damage to spinal cord level and reflex arch
    + chronic muscle atrophy
    + psychogenic causes